His primary areas of investigation include Virology, Molecular biology, Gene, Virus and Long terminal repeat. His Virology research includes themes of Nucleic acid sequence, Genome, Provirus and Genetics. His work carried out in the field of Molecular biology brings together such families of science as Jurkat cells, T cell, Gene expression, Transcription factor and Regulation of gene expression.
His Gene research is multidisciplinary, incorporating perspectives in Viral replication and Viral protein. As part of his studies on Virus, Mitsuaki Yoshida often connects relevant areas like Leukemia. His Long terminal repeat study also includes
The scientist’s investigation covers issues in Molecular biology, Virology, Virus, Gene and Leukemia. His study in Molecular biology is interdisciplinary in nature, drawing from both Jurkat cells, T cell, Enhancer, Transcription factor and Long terminal repeat. His Virology study integrates concerns from other disciplines, such as Nucleic acid sequence and Genome, Provirus.
Mitsuaki Yoshida works mostly in the field of Virus, limiting it down to topics relating to RNA and, in certain cases, Intron. His Gene study results in a more complete grasp of Genetics. His Virus type study in the realm of Leukemia connects with subjects such as Monoclonal.
His primary areas of study are Transcription factor, Molecular biology, Virology, Cell biology and Gene. The various areas that Mitsuaki Yoshida examines in his Molecular biology study include Jurkat cells, Lac repressor and In vivo. His Virology research includes elements of Human t cell leukemia and Asymptomatic carrier.
Mitsuaki Yoshida interconnects Cell cycle, Transcription and Molecular virology in the investigation of issues within Cell biology. Mitsuaki Yoshida has included themes like Cancer research and Leukemia in his Gene study. His T-cell leukemia research is multidisciplinary, incorporating elements of NFKB1, T cell and Tropical spastic paraparesis.
Mitsuaki Yoshida mainly focuses on Gene, Transcription factor, Genetics, Cell cycle and Cell biology. His Gene research incorporates elements of Molecular biology and Cancer research. His Molecular biology study combines topics from a wide range of disciplines, such as Jurkat cells, Interferon, Promoter, Repressor and Interferon regulatory factors.
In general Transcription factor study, his work on HMG-box often relates to the realm of SOX Transcription Factors, SOX gene family and SOXB2 Transcription Factors, thereby connecting several areas of interest. His biological study spans a wide range of topics, including Carcinogenesis, Tumor suppressor gene, Suppressor, DLG1 and PDZ domain. His research in Cell biology intersects with topics in Enhancer, P53 binding, Transcription, Two-hybrid screening and DNA.
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SR alpha promoter: an efficient and versatile mammalian cDNA expression system composed of the simian virus 40 early promoter and the R-U5 segment of human T-cell leukemia virus type 1 long terminal repeat.
Y. Takebe;M. Seiki;J.-I. Fujisawa;P. Hoy.
Molecular and Cellular Biology (1988)
Human adult T-cell leukemia virus: complete nucleotide sequence of the provirus genome integrated in leukemia cell DNA.
Motoharu Seiki;Seisuke Hattori;Yoko Hirayama;Mitsuaki Yoshida.
Proceedings of the National Academy of Sciences of the United States of America (1983)
Monoclonal integration of human T-cell leukemia provirus in all primary tumors of adult T-cell leukemia suggests causative role of human T-cell leukemia virus in the disease.
M Yoshida;M Seiki;K Yamaguchi;K Takatsuki.
Proceedings of the National Academy of Sciences of the United States of America (1984)
Multiple Viral Strategies of HTLV-1 for Dysregulation of Cell Growth Control
Annual Review of Immunology (2001)
Evidence for aberrant activation of the interleukin-2 autocrine loop by HTLV-1-encoded p40x and T3/Ti complex triggering.
Mitsuo Maruyama;Hiroshi Shibuya;Hisashi Harada;Masanori Hatakeyama.
Nonspecific integration of the HTLV provirus genome into adult T-cell leukaemia cells.
Motoharu Seiki;Roger Eddy;Thomas B. Shows;Mitsuaki Yoshida.
The trans-activator tax of human T-cell leukemia virus type 1 (HTLV-1) interacts with cAMP-responsive element (CRE) binding and CRE modulator proteins that bind to the 21-base-pair enhancer of HTLV-1.
Takeshi Suzuki;Jun-Ichi Fujisawa;Masami Toita;Mitsuaki Yoshida.
Proceedings of the National Academy of Sciences of the United States of America (1993)
Functional activation of the long terminal repeat of human T-cell leukemia virus type I by a trans-acting factor
Jun-Ichi Fujisawa;Motoharu Seiki;Tetsuyuki Kiyokawa;Mitsuaki Yoshida.
Proceedings of the National Academy of Sciences of the United States of America (1985)
Avian sarcoma virus Y73 genome sequence and structural similarity of its transforming gene product to that of Rous sarcoma virus
N Kitamura;A Kitamura;K Toyoshima;Y Hirayama.
A novel interferon regulatory factor family transcription factor, ICSAT/Pip/LSIRF, that negatively regulates the activity of interferon-regulated genes.
T Yamagata;J Nishida;S Tanaka;R Sakai.
Molecular and Cellular Biology (1996)
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