D-Index & Metrics Best Publications

Michael Kinter

Oklahoma Medical Research Foundation
United States

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name Citations Publications World Ranking National Ranking

Biology and Biochemistry D-index 44 Citations 8,436 130 World Ranking 16172 National Ranking 6707

Overview

What is he best known for?

The fields of study Michael Kinter is best known for:

Gene
Enzyme
Metabolism

His work on Mitochondrion expands to the thematically related Biochemistry. He performs multidisciplinary studies into Gene and Proteomics in his work. His Internal medicine study frequently draws parallels with other fields, such as Disease. He regularly links together related areas like Lipoprotein in his Endocrinology studies. In his works, Michael Kinter undertakes multidisciplinary study on Lipoprotein and High-density lipoprotein. He incorporates High-density lipoprotein and Apolipoprotein B in his research. His study ties his expertise on Endocrinology together with the subject of Apolipoprotein B. His multidisciplinary approach integrates Cell biology and Molecular biology in his work. He integrates Molecular biology and Cell biology in his studies.

His most cited work include:

Apolipoprotein A-I is a selective target for myeloperoxidase-catalyzed oxidation and functional impairment in subjects with cardiovascular disease (553 citations)
Protein Sequencing and Identification Using Tandem Mass Spectrometry (333 citations)
Noncanonical Function of Glutamyl-Prolyl-tRNA Synthetase (219 citations)

What are the main themes of his work throughout his whole career to date

Michael Kinter carries out multidisciplinary research, doing studies in Biochemistry and Molecular biology. He performs integrative Internal medicine and Endocrinology research in his work. Endocrinology is often connected to Oxidative stress in his work. His Internal medicine research extends to Oxidative stress, which is thematically connected. His Cell biology study frequently involves adjacent topics like Mitochondrion. Many of his studies involve connections with topics such as Cell biology and Mitochondrion. His work blends Gene and Proteomics studies together. He combines Proteomics and Gene in his studies. In his articles, Michael Kinter combines various disciplines, including Chromatography and Mass spectrometry.

Michael Kinter most often published in these fields:

Biochemistry (79.07%)
Internal medicine (41.86%)
Cell biology (36.05%)

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Apolipoprotein A-I is a selective target for myeloperoxidase-catalyzed oxidation and functional impairment in subjects with cardiovascular disease

Lemin Zheng;Benedicta Nukuna;Marie Luise Brennan;Mingjiang Sun.
Journal of Clinical Investigation (2004)

965 Citations

The oxygen-rich postnatal environment induces cardiomyocyte cell-cycle arrest through DNA damage response.

Bao N. Puente;Wataru Kimura;Shalini A. Muralidhar;Jesung Moon.
Cell (2014)

631 Citations

Hypoxia induces heart regeneration in adult mice

Yuji Nakada;Diana C. Canseco;Suwannee Thet;Salim Abdisalaam.
Nature (2017)

504 Citations

Glycation of mitochondrial proteins from diabetic rat kidney is associated with excess superoxide formation

Mariana G. Rosca;Tiberiu G. Mustata;Michael T. Kinter;Aylin M. Ozdemir.
American Journal of Physiology-renal Physiology (2005)

419 Citations

Polycystin-1, STAT6, and P100 function in a pathway that transduces ciliary mechanosensation and is activated in polycystic kidney disease.

Seng Hui Low;Shivakumar Vasanth;Shivakumar Vasanth;Claire H. Larson;Sambuddho Mukherjee.
Developmental Cell (2006)

390 Citations

Localization of Nitration and Chlorination Sites on Apolipoprotein A-I Catalyzed by Myeloperoxidase in Human Atheroma and Associated Oxidative Impairment in ABCA1-dependent Cholesterol Efflux from Macrophages

Lemin Zheng;Megan Settle;Gregory Brubaker;Dave Schmitt.
Journal of Biological Chemistry (2005)

285 Citations

Noncanonical Function of Glutamyl-Prolyl-tRNA Synthetase: Gene-Specific Silencing of Translation

Prabha Sampath;Prabha Sampath;Barsanjit Mazumder;Barsanjit Mazumder;Vasudevan Seshadri;Carri A. Gerber.
Cell (2004)

263 Citations

Nitrotyrosine Proteome Survey in Asthma Identifies Oxidative Mechanism of Catalase Inactivation

Sudakshina Ghosh;Allison J. Janocha;Mark A. Aronica;Shadi Swaidani.
Journal of Immunology (2006)

242 Citations

Proteomic approach to coronary atherosclerosis shows ferritin light chain as a significant marker: evidence consistent with iron hypothesis in atherosclerosis

Sun Ah You;Stephen R. Archacki;Stephen R. Archacki;George Angheloiu;Christine S. Moravec.
Physiological Genomics (2003)

171 Citations

The histone deacetylase inhibitor butyrate improves metabolism and reduces muscle atrophy during aging

Michael E. Walsh;Arunabh Bhattacharya;Kavithalakshmi Sataranatarajan;Rizwan Qaisar.
Aging Cell (2015)

158 Citations

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