Michael F. Beers mainly focuses on Surfactant protein C, Molecular biology, Cell biology, Pathology and Lung injury. His Surfactant protein C study incorporates themes from Fibrosis, Idiopathic pulmonary fibrosis and Protein precursor. His work investigates the relationship between Idiopathic pulmonary fibrosis and topics such as Cancer research that intersect with problems in Immunology and Lung.
His work deals with themes such as Mutation, Lamellar granule, Mutant, Green fluorescent protein and Exon, which intersect with Molecular biology. His work carried out in the field of Cell biology brings together such families of science as Transcription factor and Induced pluripotent stem cell. His studies in Pathology integrate themes in fields like Internal medicine, Transplantation and Pulmonary surfactant.
His scientific interests lie mostly in Lung, Surfactant protein C, Cell biology, Molecular biology and Surfactant protein D. His Lung research includes themes of Immunology and Pathology. His Surfactant protein C research is multidisciplinary, relying on both Endoplasmic reticulum, Mutant and Protein precursor.
His Cell biology research integrates issues from Cell and Alveolar type. He has included themes like Wild type, In vitro and Green fluorescent protein in his Molecular biology study. He combines subjects such as Inflammation, Lung injury and Collectin with his study of Surfactant protein D.
Michael F. Beers mainly investigates Lung, Pulmonary fibrosis, Cancer research, Pathology and Surfactant protein C. His Lung research is multidisciplinary, incorporating elements of Wnt signaling pathway, Phenotype, Immunology, ABCA3 and Disease. He interconnects Unfolded protein response, Proteostasis, Pathogenesis and Alveolar type in the investigation of issues within Pulmonary fibrosis.
The various areas that Michael F. Beers examines in his Pathogenesis study include Fibrosis and Idiopathic pulmonary fibrosis. His Surfactant protein C research incorporates themes from Monocyte, Molecular biology, Cell biology, Bronchoalveolar lavage and Lung injury. His work on Function and Regeneration is typically connected to Population as part of general Cell biology study, connecting several disciplines of science.
Michael F. Beers mostly deals with Lung, Pathogenesis, Lung injury, Bronchoalveolar lavage and Fibrosis. The concepts of his Lung study are interwoven with issues in Cancer research, Wnt signaling pathway, Disease and Immunology. His Lung injury research includes elements of Proinflammatory cytokine and Monocyte.
His study in Proinflammatory cytokine is interdisciplinary in nature, drawing from both Eosinophil, Pulmonary surfactant-associated protein C, Molecular biology, Idiopathic pulmonary fibrosis and Hyperplasia. His Bronchoalveolar lavage study frequently draws parallels with other fields, such as Surfactant protein C. His Pathology study combines topics in areas such as Embryonic stem cell and Induced pluripotent stem cell.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)
Daniel J. Klionsky;Amal Kamal Abdel-Aziz;Sara Abdelfatah;Mahmoud Abdellatif.
Endoplasmic reticulum stress enhances fibrotic remodeling in the lungs
William E. Lawson;Dong-Sheng Cheng;Amber L. Degryse;Harikrishna Tanjore.
Proceedings of the National Academy of Sciences of the United States of America (2011)
The three R’s of lung health and disease: repair, remodeling, and regeneration
Michael F. Beers;Edward E. Morrisey.
Journal of Clinical Investigation (2011)
A surfactant protein C precursor protein BRICHOS domain mutation causes endoplasmic reticulum stress, proteasome dysfunction, and caspase 3 activation.
Surafel Mulugeta;Vu Nguyen;Scott J. Russo;Madesh Muniswamy.
American Journal of Respiratory Cell and Molecular Biology (2005)
SURFACTANT PROTEIN C BIOSYNTHESIS AND ITS EMERGING ROLE IN CONFORMATIONAL LUNG DISEASE
Michael F Beers;Surafel Mulugeta.
Annual Review of Physiology (2005)
Role of endoplasmic reticulum stress in epithelial-mesenchymal transition of alveolar epithelial cells: effects of misfolded surfactant protein.
Qian Zhong;Beiyun Zhou;David K. Ann;Parviz Minoo.
American Journal of Respiratory Cell and Molecular Biology (2011)
Differentiation of Human Pluripotent Stem Cells into Functional Lung Alveolar Epithelial Cells.
Anjali Jacob;Anjali Jacob;Michael Morley;Finn Hawkins;Finn Hawkins;Katherine B. McCauley;Katherine B. McCauley.
Cell Stem Cell (2017)
Partial Deficiency of Surfactant Protein B in an Infant With Chronic Lung Disease
Philip L. Ballard;Lawrence M. Nogee;Michael F. Beers;Roberta A. Ballard.
Deletion of exon 4 from human surfactant protein C results in aggresome formation and generation of a dominant negative.
Wen-Jing Wang;Surafel Mulugeta;Scott J. Russo;Michael F. Beers.
Journal of Cell Science (2003)
Misfolded BRICHOS SP-C mutant proteins induce apoptosis via caspase-4- and cytochrome c-related mechanisms.
Surafel Mulugeta;Jean Ann Maguire;Jennifer L. Newitt;Scott J. Russo.
American Journal of Physiology-lung Cellular and Molecular Physiology (2007)
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