D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 50 Citations 15,769 118 World Ranking 10073 National Ranking 4414

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • Amino acid

Michael F. Beers mainly focuses on Surfactant protein C, Molecular biology, Cell biology, Pathology and Lung injury. His Surfactant protein C study incorporates themes from Fibrosis, Idiopathic pulmonary fibrosis and Protein precursor. His work investigates the relationship between Idiopathic pulmonary fibrosis and topics such as Cancer research that intersect with problems in Immunology and Lung.

His work deals with themes such as Mutation, Lamellar granule, Mutant, Green fluorescent protein and Exon, which intersect with Molecular biology. His work carried out in the field of Cell biology brings together such families of science as Transcription factor and Induced pluripotent stem cell. His studies in Pathology integrate themes in fields like Internal medicine, Transplantation and Pulmonary surfactant.

His most cited work include:

  • Endoplasmic reticulum stress enhances fibrotic remodeling in the lungs (242 citations)
  • The three R’s of lung health and disease: repair, remodeling, and regeneration (219 citations)
  • A surfactant protein C precursor protein BRICHOS domain mutation causes endoplasmic reticulum stress, proteasome dysfunction, and caspase 3 activation. (193 citations)

What are the main themes of his work throughout his whole career to date?

His scientific interests lie mostly in Lung, Surfactant protein C, Cell biology, Molecular biology and Surfactant protein D. His Lung research includes themes of Immunology and Pathology. His Surfactant protein C research is multidisciplinary, relying on both Endoplasmic reticulum, Mutant and Protein precursor.

His Cell biology research integrates issues from Cell and Alveolar type. He has included themes like Wild type, In vitro and Green fluorescent protein in his Molecular biology study. He combines subjects such as Inflammation, Lung injury and Collectin with his study of Surfactant protein D.

He most often published in these fields:

  • Lung (29.12%)
  • Surfactant protein C (28.57%)
  • Cell biology (21.98%)

What were the highlights of his more recent work (between 2016-2021)?

  • Lung (29.12%)
  • Pulmonary fibrosis (10.99%)
  • Cancer research (9.89%)

In recent papers he was focusing on the following fields of study:

Michael F. Beers mainly investigates Lung, Pulmonary fibrosis, Cancer research, Pathology and Surfactant protein C. His Lung research is multidisciplinary, incorporating elements of Wnt signaling pathway, Phenotype, Immunology, ABCA3 and Disease. He interconnects Unfolded protein response, Proteostasis, Pathogenesis and Alveolar type in the investigation of issues within Pulmonary fibrosis.

The various areas that Michael F. Beers examines in his Pathogenesis study include Fibrosis and Idiopathic pulmonary fibrosis. His Surfactant protein C research incorporates themes from Monocyte, Molecular biology, Cell biology, Bronchoalveolar lavage and Lung injury. His work on Function and Regeneration is typically connected to Population as part of general Cell biology study, connecting several disciplines of science.

Between 2016 and 2021, his most popular works were:

  • Differentiation of Human Pluripotent Stem Cells into Functional Lung Alveolar Epithelial Cells. (168 citations)
  • Expression of mutant Sftpc in murine alveolar epithelia drives spontaneous lung fibrosis. (69 citations)
  • Expression of mutant Sftpc in murine alveolar epithelia drives spontaneous lung fibrosis. (69 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Amino acid

Michael F. Beers mostly deals with Lung, Pathogenesis, Lung injury, Bronchoalveolar lavage and Fibrosis. The concepts of his Lung study are interwoven with issues in Cancer research, Wnt signaling pathway, Disease and Immunology. His Lung injury research includes elements of Proinflammatory cytokine and Monocyte.

His study in Proinflammatory cytokine is interdisciplinary in nature, drawing from both Eosinophil, Pulmonary surfactant-associated protein C, Molecular biology, Idiopathic pulmonary fibrosis and Hyperplasia. His Bronchoalveolar lavage study frequently draws parallels with other fields, such as Surfactant protein C. His Pathology study combines topics in areas such as Embryonic stem cell and Induced pluripotent stem cell.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky;Amal Kamal Abdel-Aziz;Sara Abdelfatah;Mahmoud Abdellatif.
Autophagy (2021)

8964 Citations

Endoplasmic reticulum stress enhances fibrotic remodeling in the lungs

William E. Lawson;Dong-Sheng Cheng;Amber L. Degryse;Harikrishna Tanjore.
Proceedings of the National Academy of Sciences of the United States of America (2011)

363 Citations

The three R’s of lung health and disease: repair, remodeling, and regeneration

Michael F. Beers;Edward E. Morrisey.
Journal of Clinical Investigation (2011)

323 Citations

A surfactant protein C precursor protein BRICHOS domain mutation causes endoplasmic reticulum stress, proteasome dysfunction, and caspase 3 activation.

Surafel Mulugeta;Vu Nguyen;Scott J. Russo;Madesh Muniswamy.
American Journal of Respiratory Cell and Molecular Biology (2005)

251 Citations

SURFACTANT PROTEIN C BIOSYNTHESIS AND ITS EMERGING ROLE IN CONFORMATIONAL LUNG DISEASE

Michael F Beers;Surafel Mulugeta.
Annual Review of Physiology (2005)

240 Citations

Role of endoplasmic reticulum stress in epithelial-mesenchymal transition of alveolar epithelial cells: effects of misfolded surfactant protein.

Qian Zhong;Beiyun Zhou;David K. Ann;Parviz Minoo.
American Journal of Respiratory Cell and Molecular Biology (2011)

210 Citations

Differentiation of Human Pluripotent Stem Cells into Functional Lung Alveolar Epithelial Cells.

Anjali Jacob;Anjali Jacob;Michael Morley;Finn Hawkins;Finn Hawkins;Katherine B. McCauley;Katherine B. McCauley.
Cell Stem Cell (2017)

186 Citations

Partial Deficiency of Surfactant Protein B in an Infant With Chronic Lung Disease

Philip L. Ballard;Lawrence M. Nogee;Michael F. Beers;Roberta A. Ballard.
Pediatrics (1995)

181 Citations

Deletion of exon 4 from human surfactant protein C results in aggresome formation and generation of a dominant negative.

Wen-Jing Wang;Surafel Mulugeta;Scott J. Russo;Michael F. Beers.
Journal of Cell Science (2003)

166 Citations

Misfolded BRICHOS SP-C mutant proteins induce apoptosis via caspase-4- and cytochrome c-related mechanisms.

Surafel Mulugeta;Jean Ann Maguire;Jennifer L. Newitt;Scott J. Russo.
American Journal of Physiology-lung Cellular and Molecular Physiology (2007)

166 Citations

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