What is she best known for?
The fields of study she is best known for:
Konstanze F. Winklhofer mainly investigates Parkin, Mitochondrion, Cell biology, PINK1 and Neurodegeneration.
Her studies in Parkin integrate themes in fields like In vitro, Ubiquitin, Ubiquitin ligase, Cytosol and Folding.
Her Mitochondrion research is multidisciplinary, incorporating perspectives in Autophagy, Molecular biology and Programmed cell death.
Konstanze F. Winklhofer has included themes like Biochemistry, Mutant and Cellular pathology in her Cell biology study.
Her PINK1 research includes themes of mitochondrial fusion, DNAJA3, Phenotype and Parkinsonism.
Her Neurodegeneration research includes elements of Alzheimer's disease, Function and Protein folding.
Her most cited work include:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- Mitochondrial dysfunction in Parkinson's disease (529 citations)
- Loss-of-Function of Human PINK1 Results in Mitochondrial Pathology and Can Be Rescued by Parkin (444 citations)
What are the main themes of her work throughout her whole career to date?
Her main research concerns Cell biology, Mitochondrion, Parkin, Biochemistry and Neurodegeneration.
Her biological study spans a wide range of topics, including Ubiquitin and Cytosol.
Konstanze F. Winklhofer interconnects mitochondrial fusion, Molecular biology, PINK1, Phosphorylation and Programmed cell death in the investigation of issues within Mitochondrion.
The study incorporates disciplines such as Autophagy, Oxidative stress and Neuroinflammation in addition to Programmed cell death.
Her study looks at the intersection of Parkin and topics like Phenotype with Gene knockdown.
Her Neurodegeneration research is multidisciplinary, relying on both Mutant and Prion protein.
She most often published in these fields:
- Cell biology (60.50%)
- Mitochondrion (35.29%)
- Parkin (30.25%)
What were the highlights of her more recent work (between 2018-2021)?
- Cell biology (60.50%)
- Ubiquitin (12.61%)
- Signal transducing adaptor protein (5.04%)
In recent papers she was focusing on the following fields of study:
Konstanze F. Winklhofer mostly deals with Cell biology, Ubiquitin, Signal transducing adaptor protein, Mitochondrion and Programmed cell death.
Her work deals with themes such as Huntingtin and Cytosol, which intersect with Cell biology.
The Ubiquitin study combines topics in areas such as Parkin, Mitophagy and Protein folding.
Her studies deal with areas such as Neurodegeneration, Nucleus, Amyloid precursor protein, Transmembrane protein and Human brain as well as Signal transducing adaptor protein.
Her work is dedicated to discovering how Mitochondrion, PINK1 are connected with Receptor tyrosine kinase and other disciplines.
Her study in Programmed cell death is interdisciplinary in nature, drawing from both Immune signaling, Inflammasome and Cellular functions.
Between 2018 and 2021, her most popular works were:
- Propionic Acid Shapes the Multiple Sclerosis Disease Course by an Immunomodulatory Mechanism (74 citations)
- α-Synuclein in Parkinson's disease: causal or bystander? (28 citations)
- A protein quality control pathway regulated by linear ubiquitination. (23 citations)
In her most recent research, the most cited papers focused on:
Her scientific interests lie mostly in Atrophy, Disease, Neuroinflammation, Microbiome and Protein quaternary structure.
Her Atrophy study combines topics from a wide range of disciplines, such as Amyloid beta, Bioinformatics, Parkinson's disease, Gene mutation and Proteasome.
Much of her study explores Disease relationship to Missense mutation.
Konstanze F. Winklhofer combines subjects such as Innate immune system, Mitochondrion, Neuroscience and Programmed cell death with her study of Neuroinflammation.
Her Microbiome research incorporates themes from Immunotherapy, Gene, Pharmacology and Neuroregeneration.
Her Protein quaternary structure research incorporates elements of Biophysics, HeLa and Membrane.
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