What is he best known for?
The fields of study he is best known for:
- Gene
- Internal medicine
- Enzyme
Kjell Hultenby focuses on Cell biology, Immunology, Internal medicine, Endocrinology and Biochemistry.
His Cell biology research integrates issues from Protein biosynthesis, Osteopontin and Pathology.
His biological study spans a wide range of topics, including Golgi apparatus, Osteoclast, Bone cell and Bone resorption.
His work deals with themes such as Vascular obstruction and Microbiology, which intersect with Immunology.
His Endocrinology research focuses on Extracellular matrix and how it relates to Hypoxia, Lung and Matrix metalloproteinase.
Kjell Hultenby studied Tight junction and Gut flora that intersect with Central nervous system and Blood–brain barrier.
His most cited work include:
- The gut microbiota influences blood-brain barrier permeability in mice (781 citations)
- Osteopontin--a possible anchor of osteoclasts to bone. (708 citations)
- Mitochondrial transcription factor A regulates mtDNA copy number in mammals (612 citations)
What are the main themes of his work throughout his whole career to date?
His main research concerns Cell biology, Internal medicine, Pathology, Endocrinology and Immunology.
His Cell biology study combines topics in areas such as Osteoclast, Osteopontin, Biochemistry and Podocyte.
His research investigates the connection between Podocyte and topics such as Zebrafish that intersect with issues in Gene knockdown.
Kjell Hultenby has researched Pathology in several fields, including Cartilage and Anatomy.
He has included themes like Liver X receptor and Immunohistochemistry in his Endocrinology study.
His work in Immunology addresses subjects such as Microbiology, which are connected to disciplines such as Bacteria and Virulence.
He most often published in these fields:
- Cell biology (28.22%)
- Internal medicine (20.75%)
- Pathology (19.92%)
What were the highlights of his more recent work (between 2014-2021)?
- Cell biology (28.22%)
- Pathology (19.92%)
- Internal medicine (20.75%)
In recent papers he was focusing on the following fields of study:
The scientist’s investigation covers issues in Cell biology, Pathology, Internal medicine, Mitochondrion and Endocrinology.
Kjell Hultenby studies Cell biology, namely Function.
His Pathology study integrates concerns from other disciplines, such as Cellular homeostasis, ATG16L1 and Tryptase.
His Mitochondrion study combines topics from a wide range of disciplines, such as mitochondrial fusion, Mitochondrial DNA and DNAJA3.
His study in Endocrinology is interdisciplinary in nature, drawing from both Bleeding diathesis, Dense granule, Collagen receptor, Fibrinogen binding and Gray platelet syndrome.
Kjell Hultenby works mostly in the field of Pathogen, limiting it down to concerns involving Periodontal pathogen and, occasionally, Immunology.
Between 2014 and 2021, his most popular works were:
- Mitofusin 2 is required to maintain mitochondrial coenzyme Q levels (117 citations)
- Molecular profiling of prostate cancer derived exosomes may reveal a predictive signature for response to docetaxel. (76 citations)
- mTor mediates tau localization and secretion: Implication for Alzheimer's disease. (65 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Internal medicine
- Enzyme
Kjell Hultenby mostly deals with Cell biology, Internal medicine, Genetics, Biophysics and Mitochondrion.
His Cell biology research integrates issues from Autophagy, Podocyte foot, Glomerular basement membrane, Glomerular Filtration Barrier and Actin cytoskeleton.
His Etiology, Albuminuria and Proinflammatory cytokine study in the realm of Internal medicine interacts with subjects such as Inclusion and exclusion criteria.
His study in the field of DNA Mutational Analysis, Common variable immunodeficiency, Mutation and Genotype also crosses realms of Text mining.
The Biophysics study combines topics in areas such as Biocompatibility, Plasma protein binding and Proteomics.
His Mitochondrion research incorporates themes from mitochondrial fusion, Mitofusin-2 and Mitochondrial DNA.
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