2023 - Research.com Biology and Biochemistry in Switzerland Leader Award
His work often combines Biochemistry and Computational biology studies. John Westwick performs multidisciplinary study on Computational biology and Biochemistry in his works. John Westwick connects Immunology with Lymphocyte in his research. Lymphocyte and Immunology are two areas of study in which John Westwick engages in interdisciplinary work. His research brings together the fields of Consensus conference and Internal medicine. Consensus conference is frequently linked to Internal medicine in his study. Endocrinology and Endothelium are frequently intertwined in his study. His Endocrinology research extends to the thematically linked field of Endothelium. Many of his studies on Gene apply to Western blot as well.
John Westwick links relevant study fields such as Prostacyclin and Platelet-activating factor in the subject of Endocrinology. His research combines Endocrinology and Prostacyclin. His Platelet studies intersect with other subjects such as Thromboxane, Thromboxane A2 and Thromboxane B2. John Westwick integrates many fields, such as Thromboxane A2 and Platelet, in his works. His Protein kinase C research covers fields of interest such as Signal transduction, Phorbol and Kinase. He merges Signal transduction with Receptor in his study. His Calcium in biology research extends to the thematically linked field of Receptor. He integrates several fields in his works, including Kinase and Protein kinase C. The research on T cell and Chemokine is part of his Immune system project.
Extracellular, Leukotriene B4 and Platelet-activating factor are fields of study that overlap with his Inflammation research. His study deals with a combination of Extracellular and Inflammation. He frequently studies issues relating to Leukotriene B4 and Internal medicine. His Platelet-activating factor research extends to Endocrinology, which is thematically connected. Many of his studies on Platelet apply to Platelet aggregation as well. He frequently studies issues relating to Platelet and Platelet aggregation. Many of his studies on Immunology apply to Pathogenesis as well. His research on Pathogenesis frequently links to adjacent areas such as Immunology. John Westwick combines Prostacyclin and Thromboxane B2 in his research.
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Cdc42 and Rac1 induce integrin-mediated cell motility and invasiveness through PI(3)K
Patricia J. Keely;John K. Westwick;Ian P. Whitehead;Channing J. Der.
Interleukin-8 gene expression by a pulmonary epithelial cell line. A model for cytokine networks in the lung.
T J Standiford;S L Kunkel;M A Basha;S W Chensue.
Journal of Clinical Investigation (1990)
Rac regulation of transformation, gene expression, and actin organization by multiple, PAK-independent pathways.
J K Westwick;Q T Lambert;G J Clark;M Symons.
Molecular and Cellular Biology (1997)
Chemokines and T lymphocytes: more than an attraction.
Stephen G Ward;Kevin Bacon;John Westwick.
IL-33–Dependent Type 2 Inflammation during Rhinovirus-induced Asthma Exacerbations In Vivo
David J. Jackson;David J. Jackson;Heidi Makrinioti;Batika M. J. Rana;Betty W. H. Shamji.
American Journal of Respiratory and Critical Care Medicine (2014)
Oncogenic Ha-Ras-induced Signaling Activates NF-κB Transcriptional Activity, Which Is Required for Cellular Transformation
Timothy S. Finco;John K. Westwick;Jacqueline L. Norris;Amer A. Beg.
Journal of Biological Chemistry (1997)
Ceramide Activates the Stress-activated Protein Kinases
John K. Westwick;Alicja E. Bielawska;Ghassan Dbaibo;Yusuf A. Hannun.
Journal of Biological Chemistry (1995)
Oncogenic Ras activation of Raf/mitogen-activated protein kinase-independent pathways is sufficient to cause tumorigenic transformation.
R Khosravi-Far;M A White;J K Westwick;P A Solski.
Molecular and Cellular Biology (1996)
The coupling of α6β4 integrin to Ras-MAP kinase pathways mediated by Shc controls keratinocyte proliferation
Fabrizio Mainiero;Chiara Murgia;Chiara Murgia;Kishore K. Wary;Kishore K. Wary;Anna Maria Curatola.
The EMBO Journal (1997)
Tumor necrosis factor alpha stimulates AP-1 activity through prolonged activation of the c-Jun kinase.
John K. Westwick;Christoff Weitzel;Audrey Minden;Michael Karin.
Journal of Biological Chemistry (1994)
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