Her main research concerns Neuroscience, Excitatory postsynaptic potential, Prefrontal cortex, Inhibitory postsynaptic potential and Electrophysiology. Her work on Neuroimmunology as part of general Neuroscience research is often related to Tau protein, thus linking different fields of science. The various areas that Jennifer I. Luebke examines in her Excitatory postsynaptic potential study include Glutamatergic and T-type calcium channel, Calcium channel, P-type calcium channel.
Jennifer I. Luebke integrates Prefrontal cortex and Cognitive decline in her studies. Her research in Inhibitory postsynaptic potential intersects with topics in Synapse and Symmetric synapse. Jennifer I. Luebke usually deals with Patch clamp and limits it to topics linked to Depolarization and Cell biology and Pathology.
Her scientific interests lie mostly in Neuroscience, Patch clamp, Excitatory postsynaptic potential, Prefrontal cortex and Electrophysiology. Her Dendritic spine, Membrane potential and Working memory study in the realm of Neuroscience connects with subjects such as Cognitive decline and Chemistry. Her Patch clamp study also includes
Jennifer I. Luebke has included themes like Neuropil, Glutamatergic and Medium spiny neuron in her Excitatory postsynaptic potential study. Jennifer I. Luebke interconnects AMPA receptor, Visual cortex and Synaptic signaling in the investigation of issues within Prefrontal cortex. In her study, Biological system is inextricably linked to Neuron, which falls within the broad field of Electrophysiology.
Her primary areas of study are Neuroscience, Cell biology, Patch clamp, Excitatory postsynaptic potential and Inhibitory postsynaptic potential. Her study in Neuroscience concentrates on Prefrontal cortex, Synapse, Spatial memory, Neural facilitation and Dorsolateral prefrontal cortex. Her research integrates issues of Dendritic spine, Innate immune system and Bursting in her study of Prefrontal cortex.
Her studies in Cell biology integrate themes in fields like Neuroinflammation and Microglia. Within one scientific family, Jennifer I. Luebke focuses on topics pertaining to Medium spiny neuron under Excitatory postsynaptic potential, and may sometimes address concerns connected to Biocytin. Her Inhibitory postsynaptic potential study integrates concerns from other disciplines, such as Stimulus and Working memory, Cognition.
The scientist’s investigation covers issues in Neuroscience, TIA1, Antigen, Intracellular and Stress granule. Her Prefrontal cortex and Excitatory postsynaptic potential investigations are all subjects of Neuroscience research. Jennifer I. Luebke has researched Prefrontal cortex in several fields, including Dendritic spine, Bursting and Microglia.
Her study on Bursting is mostly dedicated to connecting different topics, such as Innate immune system. Her Excitatory postsynaptic potential research includes themes of Medium spiny neuron, Glutamatergic and Indirect pathway of movement. Jennifer I. Luebke undertakes interdisciplinary study in the fields of TIA1 and In vivo through her works.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Exocytotic Ca2+ channels in mammalian central neurons.
Kathleen Dunlap;Jennifer I. Luebke;Timothy J. Turner.
Trends in Neurosciences (1995)
Depletion of microglia and inhibition of exosome synthesis halt tau propagation
Hirohide Asai;Seiko Ikezu;Satoshi Tsunoda;Maria Medalla.
Nature Neuroscience (2015)
Multiple calcium channel types control glutamatergic synaptic transmission in the hippocampus
Jennifer I. Luebke;Kathleen Dunlap;Timothy J. Turner.
Changes in the structural complexity of the aged brain
Dara L. Dickstein;Doron Kabaso;Anne B. Rocher;Jennifer I. Luebke.
Aging Cell (2007)
Serotonin hyperpolarizes cholinergic low-threshold burst neurons in the rat laterodorsal tegmental nucleus in vitro.
J I Luebke;R W Greene;K Semba;A Kamondi.
Proceedings of the National Academy of Sciences of the United States of America (1992)
Synapses are lost during aging in the primate prefrontal cortex
A. Peters;C. Sethares;J.I. Luebke.
Tau accumulation causes mitochondrial distribution deficits in neurons in a mouse model of tauopathy and in human Alzheimer's disease brain.
Katherine J. Kopeikina;Katherine J. Kopeikina;George A. Carlson;Rose Pitstick;Adam E. Ludvigson.
American Journal of Pathology (2011)
Dendritic spine changes associated with normal aging.
Dara L. Dickstein;Christina M. Weaver;Jennifer I. Luebke;Jennifer I. Luebke;Patrick R. Hof.
Structural and functional changes in tau mutant mice neurons are not linked to the presence of NFTs.
A.B. Rocher;J.L. Crimins;J.M. Amatrudo;M.S. Kinson.
Experimental Neurology (2010)
Normal aging results in decreased synaptic excitation and increased synaptic inhibition of layer 2/3 pyramidal cells in the monkey prefrontal cortex.
J.I Luebke;Y.-M Chang;T.L Moore;D.L Rosene;D.L Rosene.
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: