Ian Marriott mainly investigates Immunology, Immune system, Receptor, Innate immune system and Inflammation. His work on Immunology is being expanded to include thematically relevant topics such as Sexual dimorphism. He has included themes like BCG vaccine and Osteoblast in his Immune system study.
He is interested in Immune receptor, which is a branch of Receptor. His work deals with themes such as Endocrinology and TLR4, which intersect with Cytokine. His Proinflammatory cytokine research is multidisciplinary, relying on both Tumor necrosis factor alpha, Neuroglia, Astrogliosis, Microglia and NOD2.
Ian Marriott focuses on Immunology, Immune system, Microglia, Cell biology and Inflammation. His Immunology research is multidisciplinary, incorporating elements of Receptor and Astrocyte. His Immune system research integrates issues from Interleukin, Microbiology and Osteoblast.
His Microglia study incorporates themes from Astrogliosis, Central nervous system, Neuroglia, Neuroscience and Neuroinflammation. His study on Intracellular is often connected to Downregulation and upregulation as part of broader study in Cell biology. Ian Marriott interconnects Gliosis and Antigen-presenting cell in the investigation of issues within Inflammation.
His primary scientific interests are in Neuroinflammation, Cell biology, Immune system, Microglia and Immunology. His Neuroinflammation research incorporates elements of RIG-I, Central nervous system, Astrocyte and Cytokine. Ian Marriott works mostly in the field of Immune system, limiting it down to topics relating to Proinflammatory cytokine and, in certain cases, TLR4 and TLR7, as a part of the same area of interest.
His Microglia research is multidisciplinary, incorporating perspectives in Tachykinin receptor 1, Neuroscience and Intracellular. His research integrates issues of Sex characteristics and Cell type in his study of Immunology. His Inflammation study frequently links to other fields, such as Receptor.
Ian Marriott spends much of his time researching Neuroinflammation, Microglia, Neuroscience, Immune system and Neurodegeneration. His Neuroinflammation study combines topics from a wide range of disciplines, such as RNA polymerase III activity, RNA polymerase, Virology and RIG-I-like receptor. His Microglia study integrates concerns from other disciplines, such as Pathogenesis, Intracellular, Human brain, Phosphorylation and Drug discovery.
His Immune system study is associated with Immunology. His work on BCG vaccine and Vaccination as part of general Immunology study is frequently linked to Yellow fever vaccine and Measles-Mumps-Rubella Vaccine, bridging the gap between disciplines. The study incorporates disciplines such as Tachykinin receptor 1, Interleukin 22 and Cytokine, Interleukin 10 in addition to Inflammation.
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Cultured astrocytes express toll‐like receptors for bacterial products
Christal C. Bowman;Amy Rasley;Susanne L. Tranguch;Ian Marriott.
Sex-based differences in immune function and responses to vaccination.
Sabra L. Klein;Ian Marriott;Eleanor N. Fish.
Transactions of The Royal Society of Tropical Medicine and Hygiene (2015)
A 3D human triculture system modeling neurodegeneration and neuroinflammation in Alzheimer's disease.
Joseph Park;Isaac Wetzel;Ian Marriott;Didier Dréau.
Nature Neuroscience (2018)
Testosterone Reduces Macrophage Expression in the Mouse of Toll-Like Receptor 4, a Trigger for Inflammation and Innate Immunity
Jennifer A. Rettew;Yvette M. Huet-Hudson;Ian Marriott.
Biology of Reproduction (2008)
Sexual dimorphism in innate immune responses to infectious organisms.
Ian Marriott;Yvette M. Huet-Hudson.
Immunologic Research (2006)
Sexual dimorphism in expression of receptors for bacterial lipopolysaccharides in murine macrophages: a possible mechanism for gender-based differences in endotoxic shock susceptibility.
Ian Marriott;Kenneth L. Bost;Yvette M. Huet-Hudson.
Journal of Reproductive Immunology (2006)
Estrogens augment cell surface TLR4 expression on murine macrophages and regulate sepsis susceptibility in vivo.
Jennifer A. Rettew;Yvette M. Huet;Ian Marriott.
Staphylococcus aureus Infection of Mouse or Human Osteoblasts Induces High Levels of Interleukin-6 and Interleukin-12 Production
Kenneth L. Bost;Warren K. Ramp;Natalie C. Nicholson;Jennifer L. Bento.
The Journal of Infectious Diseases (1999)
Salmonella efficiently enter and survive within cultured CD11c+ dendritic cells initiating cytokine expression.
Ian Marriott;Timothy G. Hammond;Elaine K. Thomas;Kenneth L. Bost.
European Journal of Immunology (1999)
Substance P activates NF-κB independent of elevations in intracellular calcium in murine macrophages and dendritic cells
Ian Marriott;Michael J Mason;Adam Elhofy;Kenneth L Bost.
Journal of Neuroimmunology (2000)
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