The scientist’s investigation covers issues in Cancer research, Endocrinology, Internal medicine, In vivo and Carcinogenesis. His Cancer research research is multidisciplinary, relying on both Prostaglandin, Tumor promotion, Proinflammatory cytokine, Phorbol and Hyperplasia. As a member of one scientific family, Gerhard Fürstenberger mostly works in the field of Tumor promotion, focusing on Immunology and, on occasion, Mezerein.
As part of one scientific family, Gerhard Fürstenberger deals mainly with the area of Proinflammatory cytokine, narrowing it down to issues related to the Biochemistry, and often Molecular biology. His biological study spans a wide range of topics, including Receptor and Baicalein. His studies deal with areas such as Prostaglandin E, 12-O-Tetradecanoylphorbol-13-acetate and Lipoxygenase as well as In vivo.
Biochemistry, Molecular biology, Cancer research, Carcinogenesis and Arachidonic acid are his primary areas of study. His Biochemistry study incorporates themes from Cell culture and Epidermis. The concepts of his Epidermis study are interwoven with issues in Endocrinology, Internal medicine, In vivo and Keratinocyte.
His Molecular biology study also includes fields such as
His primary scientific interests are in Cancer research, Cell biology, Carcinogenesis, Pathology and Molecular biology. His studies deal with areas such as Cell, Cancer and Immunology as well as Cancer research. His work in Immunology addresses subjects such as Tumor promotion, which are connected to disciplines such as S100A9, Immune system and Bone marrow.
Within one scientific family, Gerhard Fürstenberger focuses on topics pertaining to Lipid metabolism under Cell biology, and may sometimes address concerns connected to Ceramide and Epidermis. He focuses mostly in the field of Carcinogenesis, narrowing it down to matters related to Skin cancer and, in some cases, c-Fos. His Molecular biology research is multidisciplinary, incorporating perspectives in Adduct, DNA adduct, Arachidonic acid and Toxicology.
Gerhard Fürstenberger mainly focuses on Carcinogenesis, Cancer research, Congenital ichthyosis, ALOX12B and ALOXE3. His primary area of study in Carcinogenesis is in the field of Tumor promotion. His studies examine the connections between Cancer research and genetics, as well as such issues in Hyperplasia, with regards to Angiogenesis, Keratin 5 and Cell growth.
Gerhard Fürstenberger works mostly in the field of Congenital ichthyosis, limiting it down to topics relating to Barrier function and, in certain cases, Ceramide, Lipid metabolism, Cell type and Biochemistry. His work deals with themes such as Prostaglandin E synthase, Endocrinology, Prostaglandin and Human skin, which intersect with Skin cancer. He studied Epidermis and Protein kinase A that intersect with Keratin and Pathology.
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RAGE signaling sustains inflammation and promotes tumor development
Christoffer Gebhardt;Astrid Riehl;Moritz Durchdewald;Julia Németh.
Journal of Experimental Medicine (2008)
Transgenic cyclooxygenase-2 overexpression sensitizes mouse skin for carcinogenesis
Karin Müller-Decker;Gitta Neufang;Irina Berger;Melanie Neumann.
Proceedings of the National Academy of Sciences of the United States of America (2002)
The mRNA coding for the secreted protease transin is expressed more abundantly in malignant than in benign tumors.
L M Matrisian;G T Bowden;P Krieg;G Fürstenberger.
Proceedings of the National Academy of Sciences of the United States of America (1986)
Abnormal differentiation of epidermis in transgenic mice constitutively expressing cyclooxygenase-2 in skin
Gitta Neufang;Gerhard Fürstenberger;Markus Heidt;Friedrich Marks.
Proceedings of the National Academy of Sciences of the United States of America (2001)
Skin tumor promotion by phorbol esters is a two-stage process.
G Fürstenberger;D L Berry;B Sorg;F Marks.
Proceedings of the National Academy of Sciences of the United States of America (1981)
Sequential expression of mRNA-encoded keratin sets in neonatal mouse epidermis: Basal cells with properties of terminally differentiating cells
Jürgen Schweizer;Mitsuru Kinjo;Gerhard Fürstenberger;Hermelita Winter.
12R-lipoxygenase deficiency disrupts epidermal barrier function
Nikolas Epp;Gerhard Fürstenberger;Karsten Müller;Silvia de Juanes.
Journal of Cell Biology (2007)
DIFFERENTIAL EXPRESSION OF PROSTAGLANDIN H SYNTHASE ISOZYMES DURING MULTISTAGE CARCINOGENESIS IN MOUSE EPIDERMIS
Karin Müller‐Decker;Kirsten Scholz;Friedrich Marks;Gerhard Fürstenberger.
Molecular Carcinogenesis (1995)
Early prostaglandin E synthesis is an obligatory event in the induction of cell proliferation in mouse epidermis in vivo by the phorbol ester TPA.
Gerhard Fürstenberger;Friedrich Marks.
Biochemical and Biophysical Research Communications (1980)
Adipocyte differentiation of 3T3-L1 preadipocytes is dependent on lipoxygenase activity during the initial stages of the differentiation process
Lise Madsen;Rasmus K Petersen;Morten B Sørensen;Claus Jørgensen.
Biochemical Journal (2003)
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