His primary scientific interests are in Cell biology, Keratinocyte, Immunology, Cancer research and Cellular differentiation. He combines subjects such as Fibroblast, In vitro and Involucrin with his study of Cell biology. His Keratinocyte research is within the category of Cell culture.
His biological study spans a wide range of topics, including Cell growth, Phenotype, Molecular biology, Human skin and Transplantation. His work carried out in the field of Cancer research brings together such families of science as Tumor progression and Tumour stroma. His Cellular differentiation research integrates issues from Skin equivalent and JUNB.
Norbert E. Fusenig mostly deals with Cell biology, Keratinocyte, Cancer research, Pathology and HaCaT. Norbert E. Fusenig interconnects Involucrin and Keratin in the investigation of issues within Cell biology. The various areas that Norbert E. Fusenig examines in his Keratinocyte study include Fibroblast, Cellular differentiation, Tissue homeostasis and Immunology.
His Cancer research research is multidisciplinary, relying on both Autocrine signalling, Paracrine signalling, Carcinogenesis, Tumor progression and Human skin. The study incorporates disciplines such as Angiogenesis, Transplantation and Cell–cell interaction in addition to Pathology. As part of the same scientific family, he usually focuses on HaCaT, concentrating on Oncogene and intersecting with Malignant transformation.
His scientific interests lie mostly in Cell biology, Pathology, Cancer research, Angiogenesis and Stromal cell. Norbert E. Fusenig has researched Cell biology in several fields, including Cell culture, Immunology and Keratin. His study looks at the intersection of Pathology and topics like Cell–cell interaction with Tumor microenvironment, Cellular differentiation and Cell growth.
His Cancer research research includes elements of Tumor progression, Cancer stem cell, Hyaluronic acid and Transplantation. His Angiogenesis study integrates concerns from other disciplines, such as Human skin, Carcinoma, Vascular endothelial growth factor and Granulocyte macrophage colony-stimulating factor. His research on Human skin often connects related topics like Keratinocyte.
Immunology, Angiogenesis, Cancer research, Cell biology and Pathology are his primary areas of study. His Immunology research includes themes of Cancer cell and Cellular differentiation. His research in Cancer research intersects with topics in Tumor progression, Malignancy and Tumour stroma.
His Cell biology study combines topics from a wide range of disciplines, such as Cell culture and Organogenesis. His work deals with themes such as Magnetic resonance imaging and Kinase insert domain receptor, which intersect with Pathology. The Basement membrane study combines topics in areas such as Fibroblast, Dermis and Keratinocyte.
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Normal keratinization in a spontaneously immortalized aneuploid human keratinocyte cell line.
Petra Boukamp;R. T. Petrussevska;D. Breitkreutz;J. Hornung.
Journal of Cell Biology (1988)
Friends or foes - bipolar effects of the tumour stroma in cancer.
Margareta M. Mueller;Norbert E. Fusenig.
Nature Reviews Cancer (2004)
Absence of host plasminogen activator inhibitor 1 prevents cancer invasion and vascularization
Khalid Bajou;Agnès Noël;R. D. Gerard;Véronique Masson.
Nature Medicine (1998)
Halting angiogenesis suppresses carcinoma cell invasion.
M. Skobe;P. Rockwell;N. Goldstein;S. Vosseler.
Nature Medicine (1997)
Mutual induction of growth factor gene expression by epidermal-dermal cell interaction
H Smola;G Thiekötter;NE Fusenig.
Journal of Cell Biology (1993)
c-Jun and JunB Antagonistically Control Cytokine-Regulated Mesenchymal–Epidermal Interaction in Skin
Axel Szabowski;Nicole Maas-Szabowski;Sven Andrecht;Andrea Kolbus.
Cell (2000)
Molecular and cytogenetic analysis of immortalized human primary keratinocytes obtained after transfection with human papillomavirus type 16 DNA.
M. Durst;R. T. Dzarlieva-Petrusevska;P. Boukamp;N. E. Fusenig.
Oncogene (1987)
The Plasminogen Activator Inhibitor PAI-1 Controls In Vivo Tumor Vascularization by Interaction with Proteases, Not Vitronectin: Implications for Antiangiogenic Strategies
Khalid Bajou;Véronique Masson;Robert D. Gerard;Petra M. Schmitt.
Journal of Cell Biology (2001)
Epidermal organization and differentiation of HaCaT keratinocytes in organotypic coculture with human dermal fibroblasts.
Veronika M Schoop;Nicolae Mirancea;Norbert E Fusenig.
Journal of Investigative Dermatology (1999)
Multiple stages and genetic alterations in immortalization, malignant transformation, and tumor progression of human skin keratinocytes.
Norbert E. Fusenig;Petra Boukamp.
Molecular Carcinogenesis (1998)
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