What is he best known for?
The fields of study he is best known for:
Fabio Martelli focuses on microRNA, Cell biology, Hypoxia, Bioinformatics and Regulation of gene expression.
His microRNA research is multidisciplinary, incorporating perspectives in Angiogenesis, Gene expression, Gene expression profiling, Cellular differentiation and Immunology.
His studies in Angiogenesis integrate themes in fields like Apoptosis and Downregulation and upregulation.
His Cell biology research includes elements of Oxidative stress, Molecular biology, Cell cycle and Cell migration.
His research investigates the link between Molecular biology and topics such as Myocyte that cross with problems in Anatomy.
The study incorporates disciplines such as Genetics, In situ hybridization, Transcription factor and Long non-coding RNA, HOTAIR in addition to Bioinformatics.
His most cited work include:
- MicroRNA-210 Modulates Endothelial Cell Response to Hypoxia and Inhibits the Receptor Tyrosine Kinase Ligand Ephrin-A3 (699 citations)
- Circulating microRNAs are new and sensitive biomarkers of myocardial infarction (582 citations)
- MicroRNA-210 as a Novel Therapy for Treatment of Ischemic Heart Disease (344 citations)
What are the main themes of his work throughout his whole career to date?
Fabio Martelli spends much of his time researching microRNA, Cell biology, Molecular biology, Cancer research and Bioinformatics.
His microRNA research incorporates elements of Hypoxia, Gene expression, Skeletal muscle, Regulation of gene expression and Myotonic dystrophy.
The various areas that Fabio Martelli examines in his Cell biology study include Endothelial stem cell, Oxidative stress, Downregulation and upregulation, Nitric oxide and Cell cycle.
His biological study spans a wide range of topics, including Protein phosphatase 2, Gene knockdown, Cellular differentiation, Histone deacetylase and Myocyte.
As a part of the same scientific study, Fabio Martelli usually deals with the Cancer research, concentrating on E2F1 and frequently concerns with E2F.
His research in Bioinformatics intersects with topics in Disease and Heart failure.
He most often published in these fields:
- microRNA (30.97%)
- Cell biology (29.03%)
- Molecular biology (17.42%)
What were the highlights of his more recent work (between 2017-2021)?
- RNA (13.55%)
- Disease (11.61%)
- microRNA (30.97%)
In recent papers he was focusing on the following fields of study:
RNA, Disease, microRNA, Bioinformatics and Epigenetics are his primary areas of study.
Fabio Martelli interconnects Gene expression, Genome, Cell biology, Regulation of gene expression and Computational biology in the investigation of issues within RNA.
Fabio Martelli connects Cell biology with Nitric Oxide Synthase Type III in his study.
His microRNA study combines topics in areas such as Competing endogenous RNA and Long non-coding RNA.
His Bioinformatics research includes themes of Autophagy, Cell, Senescence and Heart failure.
Fabio Martelli usually deals with Gene and limits it to topics linked to Myotonic dystrophy and Molecular biology and Peripheral blood mononuclear cell.
Between 2017 and 2021, his most popular works were:
- Regulatory RNAs in Heart Failure. (36 citations)
- Circular RNAs in Muscle Function and Disease (34 citations)
- Circular RNAs: Methodological challenges and perspectives in cardiovascular diseases. (34 citations)
In his most recent research, the most cited papers focused on:
The scientist’s investigation covers issues in RNA, Computational biology, Regulation of gene expression, microRNA and Epigenetics.
The concepts of his Regulation of gene expression study are interwoven with issues in RNA splicing and Cell biology.
His studies deal with areas such as Gene expression and Striated muscle tissue as well as Cell biology.
His microRNA study results in a more complete grasp of Gene.
Fabio Martelli has researched Myotonic dystrophy in several fields, including Molecular biology, RNA-induced silencing complex and Messenger RNA.
His Molecular biology research integrates issues from Alternative splicing and Exon.
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