Fabio Martelli focuses on microRNA, Cell biology, Hypoxia, Bioinformatics and Regulation of gene expression. His microRNA research is multidisciplinary, incorporating perspectives in Angiogenesis, Gene expression, Gene expression profiling, Cellular differentiation and Immunology. His studies in Angiogenesis integrate themes in fields like Apoptosis and Downregulation and upregulation.
His Cell biology research includes elements of Oxidative stress, Molecular biology, Cell cycle and Cell migration. His research investigates the link between Molecular biology and topics such as Myocyte that cross with problems in Anatomy. The study incorporates disciplines such as Genetics, In situ hybridization, Transcription factor and Long non-coding RNA, HOTAIR in addition to Bioinformatics.
Fabio Martelli spends much of his time researching microRNA, Cell biology, Molecular biology, Cancer research and Bioinformatics. His microRNA research incorporates elements of Hypoxia, Gene expression, Skeletal muscle, Regulation of gene expression and Myotonic dystrophy. The various areas that Fabio Martelli examines in his Cell biology study include Endothelial stem cell, Oxidative stress, Downregulation and upregulation, Nitric oxide and Cell cycle.
His biological study spans a wide range of topics, including Protein phosphatase 2, Gene knockdown, Cellular differentiation, Histone deacetylase and Myocyte. As a part of the same scientific study, Fabio Martelli usually deals with the Cancer research, concentrating on E2F1 and frequently concerns with E2F. His research in Bioinformatics intersects with topics in Disease and Heart failure.
RNA, Disease, microRNA, Bioinformatics and Epigenetics are his primary areas of study. Fabio Martelli interconnects Gene expression, Genome, Cell biology, Regulation of gene expression and Computational biology in the investigation of issues within RNA. Fabio Martelli connects Cell biology with Nitric Oxide Synthase Type III in his study.
His microRNA study combines topics in areas such as Competing endogenous RNA and Long non-coding RNA. His Bioinformatics research includes themes of Autophagy, Cell, Senescence and Heart failure. Fabio Martelli usually deals with Gene and limits it to topics linked to Myotonic dystrophy and Molecular biology and Peripheral blood mononuclear cell.
The scientist’s investigation covers issues in RNA, Computational biology, Regulation of gene expression, microRNA and Epigenetics. The concepts of his Regulation of gene expression study are interwoven with issues in RNA splicing and Cell biology. His studies deal with areas such as Gene expression and Striated muscle tissue as well as Cell biology.
His microRNA study results in a more complete grasp of Gene. Fabio Martelli has researched Myotonic dystrophy in several fields, including Molecular biology, RNA-induced silencing complex and Messenger RNA. His Molecular biology research integrates issues from Alternative splicing and Exon.
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MicroRNA-210 Modulates Endothelial Cell Response to Hypoxia and Inhibits the Receptor Tyrosine Kinase Ligand Ephrin-A3
Pasquale Fasanaro;Yuri D'Alessandra;Valeria Di Stefano;Roberta Melchionna.
Journal of Biological Chemistry (2008)
Circulating microRNAs are new and sensitive biomarkers of myocardial infarction
Yuri D'Alessandra;Paolo Devanna;Federica Limana;Stefania Straino.
European Heart Journal (2010)
MicroRNA-210 as a Novel Therapy for Treatment of Ischemic Heart Disease
Shijun Hu;Mei Huang;Zongjin Li;Fangjun Jia.
Deregulation of microRNA-503 Contributes to Diabetes Mellitus–Induced Impairment of Endothelial Function and Reparative Angiogenesis After Limb Ischemia
Andrea Caporali;Marco Meloni;Christine Vollenkle;Desireee Bonci.
miR-200c is upregulated by oxidative stress and induces endothelial cell apoptosis and senescence via ZEB1 inhibition
A Magenta;C Cencioni;P Fasanaro;G Zaccagnini.
Cell Death & Differentiation (2011)
An Integrated Approach for Experimental Target Identification of Hypoxia-induced miR-210
Pasquale Fasanaro;Simona Greco;Maria Lorenzi;Mario Pescatori.
Journal of Biological Chemistry (2009)
Common micro-RNA signature in skeletal muscle damage and regeneration induced by Duchenne muscular dystrophy and acute ischemia
Simona Greco;Marco De Simone;Claudia Colussi;Germana Zaccagnini.
The FASEB Journal (2009)
The retinoblastoma gene product protects E2F-1 from degradation by the ubiquitin-proteasome pathway.
Francesco Hofmann;Fabio Martelli;David M. Livingston;Zhiyan Wang.
Genes & Development (1996)
miR-210: More than a Silent Player in Hypoxia
Cecilia Devlin;Simona Greco;Fabio Martelli;Mircea Ivan.
Iubmb Life (2011)
HDAC2 blockade by nitric oxide and histone deacetylase inhibitors reveals a common target in Duchenne muscular dystrophy treatment
Claudia Colussi;Chiara Mozzetta;Aymone Gurtner;Barbara Illi.
Proceedings of the National Academy of Sciences of the United States of America (2008)
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