D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 60 Citations 12,608 122 World Ranking 7884 National Ranking 271

Overview

What is she best known for?

The fields of study she is best known for:

  • Gene
  • Cancer
  • DNA

Her main research concerns Hyaluronan-mediated motility receptor, Cell biology, Receptor, Molecular biology and Hyaluronic acid. In her study, which falls under the umbrella issue of Hyaluronan-mediated motility receptor, Kinase and Cancer research is strongly linked to Signal transduction. Her studies deal with areas such as Cell surface receptor, Morphogenesis and TSG-6 as well as Cell biology.

Eva A. Turley combines subjects such as Transcriptome, CD44 and BARD1 with her study of Receptor. Her studies in Molecular biology integrate themes in fields like Amino acid, Binding protein, Complementary DNA, Peptide sequence and Fusion protein. Her Hyaluronic acid research focuses on subjects like Motility, which are linked to Autocrine signalling and Cell.

Her most cited work include:

  • Signaling properties of hyaluronan receptors. (845 citations)
  • Hyaluronan and homeostasis : a balancing act (402 citations)
  • HA receptors: regulators of signalling to the cytoskeleton (397 citations)

What are the main themes of her work throughout her whole career to date?

Her primary areas of investigation include Cell biology, Hyaluronan-mediated motility receptor, Receptor, Molecular biology and Cancer research. Her research investigates the connection with Cell biology and areas like Wound healing which intersect with concerns in Cell migration. Her Hyaluronan-mediated motility receptor study incorporates themes from Tyrosine phosphorylation, Phosphorylation, Focal adhesion and Hyaluronic acid.

As a part of the same scientific study, Eva A. Turley usually deals with the Receptor, concentrating on CD44 and frequently concerns with Inflammation, Knockout mouse and Arthritis. As a part of the same scientific family, Eva A. Turley mostly works in the field of Molecular biology, focusing on Fusion protein and, on occasion, Amino acid and Binding site. The various areas that Eva A. Turley examines in her Cancer research study include Tumor microenvironment, Cancer, Metastasis, Tumor progression and Pathology.

She most often published in these fields:

  • Cell biology (44.70%)
  • Hyaluronan-mediated motility receptor (30.30%)
  • Receptor (31.82%)

What were the highlights of her more recent work (between 2015-2020)?

  • Cell biology (44.70%)
  • Receptor (31.82%)
  • CD44 (20.45%)

In recent papers she was focusing on the following fields of study:

Eva A. Turley mostly deals with Cell biology, Receptor, CD44, Cancer research and Tumor initiation. Her Cell biology study integrates concerns from other disciplines, such as Wound healing, Inflammation and RHAMM protein. Receptor is a subfield of Biochemistry that she studies.

The study incorporates disciplines such as Molecular biology, Morphogenesis, Growth factor, Mammary gland morphogenesis and Extracellular matrix in addition to CD44. Eva A. Turley works mostly in the field of Cancer research, limiting it down to concerns involving Cell and, occasionally, Glioma. Her Oncology research incorporates themes from Internal medicine and Hyaluronan-mediated motility receptor.

Between 2015 and 2020, her most popular works were:

  • Hyaluronan, Cancer-Associated Fibroblasts and the Tumor Microenvironment in Malignant Progression. (51 citations)
  • Biphasic Dependence of Glioma Survival and Cell Migration on CD44 Expression Level (47 citations)
  • Carcinoma cell hyaluronan as a "portable" cancerized prometastatic microenvironment (46 citations)

In her most recent research, the most cited papers focused on:

  • Gene
  • Cancer
  • DNA

Eva A. Turley spends much of her time researching Cell biology, Tumor initiation, Tumor microenvironment, CD44 and Receptor. Her study in the fields of Signal transduction under the domain of Cell biology overlaps with other disciplines such as Beta-Arrestins. Her biological study spans a wide range of topics, including Stroma and Circulating tumor cell.

Her Circulating tumor cell study also includes

  • Epithelial–mesenchymal transition which intersects with area such as Cancer research,
  • Parenchyma which intersects with area such as Metastasis. Her study looks at the relationship between Cancer research and fields such as Cell, as well as how they intersect with chemical problems. Her research in Tumor microenvironment intersects with topics in Wound healing, Epithelial cell migration and Cell growth.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Signaling properties of hyaluronan receptors.

Eva A. Turley;Paul W. Noble;Lilly Y.W. Bourguignon.
Journal of Biological Chemistry (2002)

1213 Citations

HA receptors: regulators of signalling to the cytoskeleton

Joycelyn Entwistle;Christine L. Hall;Eva A. Turley.
Journal of Cellular Biochemistry (1996)

579 Citations

Hyaluronan and homeostasis : a balancing act

Markku I. Tammi;Anthony J. Day;Eva A. Turley.
Journal of Biological Chemistry (2002)

537 Citations

Identification of a common hyaluronan binding motif in the hyaluronan binding proteins RHAMM, CD44 and link protein.

Baihua Yang;Bing Luo Yang;Rashmin C. Savani;Eva Ann Turley.
The EMBO Journal (1994)

461 Citations

Molecular cloning of a novel hyaluronan receptor that mediates tumor cell motility.

C Hardwick;K Hoare;R Owens;HP Hohn.
Journal of Cell Biology (1992)

419 Citations

Overexpression of the hyaluronan receptor RHAMM is transforming and is also required for H-ras transformation

Christine L. Hall;Baihua Yang;Xuiwei Yang;Shiwen Zhang.
Cell (1995)

373 Citations

Phenotypic reversion or death of cancer cells by altering signaling pathways in three-dimensional contexts.

Fei Wang;Rhonda K. Hansen;Derek Radisky;Toshiyuki Yoneda.
Journal of the National Cancer Institute (2002)

320 Citations

Mechanisms of disease: epithelial-mesenchymal transition--does cellular plasticity fuel neoplastic progression?

Eva A Turley;Mandana Veiseh;Derek C Radisky;Mina J Bissell.
Nature Reviews Clinical Oncology (2008)

276 Citations

Hyaluronan and the hyaluronan receptor RHAMM promote focal adhesion turnover and transient tyrosine kinase activity.

C. L. Hall;Chao Wang;L. A. Lange;E. A. Turley.
Journal of Cell Biology (1994)

264 Citations

The Hyaluronan Receptor RHAMM Regulates Extracellular-regulated Kinase

Shiwen Zhang;Michael C.Y. Chang;Danuta Zylka;Stefanie Turley.
Journal of Biological Chemistry (1998)

263 Citations

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