Her main research concerns Hyaluronan-mediated motility receptor, Cell biology, Receptor, Molecular biology and Hyaluronic acid. In her study, which falls under the umbrella issue of Hyaluronan-mediated motility receptor, Kinase and Cancer research is strongly linked to Signal transduction. Her studies deal with areas such as Cell surface receptor, Morphogenesis and TSG-6 as well as Cell biology.
Eva A. Turley combines subjects such as Transcriptome, CD44 and BARD1 with her study of Receptor. Her studies in Molecular biology integrate themes in fields like Amino acid, Binding protein, Complementary DNA, Peptide sequence and Fusion protein. Her Hyaluronic acid research focuses on subjects like Motility, which are linked to Autocrine signalling and Cell.
Her primary areas of investigation include Cell biology, Hyaluronan-mediated motility receptor, Receptor, Molecular biology and Cancer research. Her research investigates the connection with Cell biology and areas like Wound healing which intersect with concerns in Cell migration. Her Hyaluronan-mediated motility receptor study incorporates themes from Tyrosine phosphorylation, Phosphorylation, Focal adhesion and Hyaluronic acid.
As a part of the same scientific study, Eva A. Turley usually deals with the Receptor, concentrating on CD44 and frequently concerns with Inflammation, Knockout mouse and Arthritis. As a part of the same scientific family, Eva A. Turley mostly works in the field of Molecular biology, focusing on Fusion protein and, on occasion, Amino acid and Binding site. The various areas that Eva A. Turley examines in her Cancer research study include Tumor microenvironment, Cancer, Metastasis, Tumor progression and Pathology.
Eva A. Turley mostly deals with Cell biology, Receptor, CD44, Cancer research and Tumor initiation. Her Cell biology study integrates concerns from other disciplines, such as Wound healing, Inflammation and RHAMM protein. Receptor is a subfield of Biochemistry that she studies.
The study incorporates disciplines such as Molecular biology, Morphogenesis, Growth factor, Mammary gland morphogenesis and Extracellular matrix in addition to CD44. Eva A. Turley works mostly in the field of Cancer research, limiting it down to concerns involving Cell and, occasionally, Glioma. Her Oncology research incorporates themes from Internal medicine and Hyaluronan-mediated motility receptor.
Eva A. Turley spends much of her time researching Cell biology, Tumor initiation, Tumor microenvironment, CD44 and Receptor. Her study in the fields of Signal transduction under the domain of Cell biology overlaps with other disciplines such as Beta-Arrestins. Her biological study spans a wide range of topics, including Stroma and Circulating tumor cell.
Her Circulating tumor cell study also includes
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Signaling properties of hyaluronan receptors.
Eva A. Turley;Paul W. Noble;Lilly Y.W. Bourguignon.
Journal of Biological Chemistry (2002)
HA receptors: regulators of signalling to the cytoskeleton
Joycelyn Entwistle;Christine L. Hall;Eva A. Turley.
Journal of Cellular Biochemistry (1996)
Hyaluronan and homeostasis : a balancing act
Markku I. Tammi;Anthony J. Day;Eva A. Turley.
Journal of Biological Chemistry (2002)
Identification of a common hyaluronan binding motif in the hyaluronan binding proteins RHAMM, CD44 and link protein.
Baihua Yang;Bing Luo Yang;Rashmin C. Savani;Eva Ann Turley.
The EMBO Journal (1994)
Molecular cloning of a novel hyaluronan receptor that mediates tumor cell motility.
C Hardwick;K Hoare;R Owens;HP Hohn.
Journal of Cell Biology (1992)
Overexpression of the hyaluronan receptor RHAMM is transforming and is also required for H-ras transformation
Christine L. Hall;Baihua Yang;Xuiwei Yang;Shiwen Zhang.
Phenotypic reversion or death of cancer cells by altering signaling pathways in three-dimensional contexts.
Fei Wang;Rhonda K. Hansen;Derek Radisky;Toshiyuki Yoneda.
Journal of the National Cancer Institute (2002)
Mechanisms of disease: epithelial-mesenchymal transition--does cellular plasticity fuel neoplastic progression?
Eva A Turley;Mandana Veiseh;Derek C Radisky;Mina J Bissell.
Nature Reviews Clinical Oncology (2008)
Hyaluronan and the hyaluronan receptor RHAMM promote focal adhesion turnover and transient tyrosine kinase activity.
C. L. Hall;Chao Wang;L. A. Lange;E. A. Turley.
Journal of Cell Biology (1994)
The Hyaluronan Receptor RHAMM Regulates Extracellular-regulated Kinase
Shiwen Zhang;Michael C.Y. Chang;Danuta Zylka;Stefanie Turley.
Journal of Biological Chemistry (1998)
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