His scientific interests lie mostly in Neuroscience, Internal medicine, Endocrinology, Gene expression and Gene. His work on Amygdala as part of general Neuroscience research is frequently linked to Poison control, bridging the gap between disciplines. His study in Serotonin, Pathological and Somatostatin is carried out as part of his studies in Internal medicine.
His Gene expression study frequently draws parallels with other fields, such as Central nervous system. Etienne Sibille has researched Gene in several fields, including Prefrontal cortex and Human brain. His Microarray research includes elements of Pathway analysis and Gene expression profiling.
Etienne Sibille mainly focuses on Neuroscience, Internal medicine, Endocrinology, Depression and Major depressive disorder. His Neuroscience study frequently draws connections to adjacent fields such as Transcriptome. His Internal medicine research is multidisciplinary, relying on both Psychiatry, Genome-wide association study and Oncology.
His Endocrinology research incorporates elements of Neuropeptide, Gene expression and Serotonin. His work on Gene expression profiling as part of general Gene expression research is often related to Poison control, thus linking different fields of science. His Major depressive disorder research is multidisciplinary, incorporating perspectives in Anterior cingulate cortex and Pathological.
Neuroscience, Internal medicine, Transcriptome, Depression and Chronic stress are his primary areas of study. Etienne Sibille interconnects Neurotrophin and GABAA receptor in the investigation of issues within Neuroscience. His research integrates issues of Venlafaxine, Endocrinology, Oncology and Schizophrenia in his study of Internal medicine.
Etienne Sibille has included themes like Gene and Green fluorescent protein in his Endocrinology study. The various areas that he examines in his Chronic stress study include Prefrontal cortex and Mood disorders. His GABAergic study combines topics from a wide range of disciplines, such as Glutamatergic, Gene expression and Interneuron.
Etienne Sibille mainly investigates Neuroscience, Depression, Transcriptome, Cognition and GABAergic. His research investigates the link between Neuroscience and topics such as Receptor that cross with problems in Mood. His Depression study integrates concerns from other disciplines, such as Senescence and Severity of illness.
Etienne Sibille works mostly in the field of Transcriptome, limiting it down to topics relating to Major depressive disorder and, in certain cases, Oncology, Internal medicine and Schizophrenia. His studies in Cognition integrate themes in fields like Physiology, Disease, Allele and Cognitive decline. His GABAergic study incorporates themes from Cortex, Pathological, Interneuron and Somatostatin.
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Increased anxiety of mice lacking the serotonin1A receptor
Christopher L. Parks;Patricia S. Robinson;Etienne Sibille;Thomas Eugene Shenk.
Proceedings of the National Academy of Sciences of the United States of America (1998)
Impaired hippocampal-dependent learning and functional abnormalities in the hippocampus in mice lacking serotonin(1A) receptors.
Zoltán Sarnyai;Etienne L. Sibille;Constantine Pavlides;Robert J. Fenster.
Proceedings of the National Academy of Sciences of the United States of America (2000)
Genetic and pharmacological disruption of neurokinin 1 receptor function decreases anxiety-related behaviors and increases serotonergic function.
Luca Santarelli;Gabriella Gobbi;Pierre C. Debs;Etienne L. Sibille.
Proceedings of the National Academy of Sciences of the United States of America (2001)
Molecular evidence for BDNF- and GABA-related dysfunctions in the amygdala of female subjects with major depression.
Jean-Philippe Guilloux;Gaelle Douillard-Guilloux;Rama Kota;Xingbin Wang.
Molecular Psychiatry (2012)
Using the Gene Ontology for Microarray Data Mining: A Comparison of Methods and Application to Age Effects in Human Prefrontal Cortex
Paul Pavlidis;Jie Qin;Victoria Arango;Victoria Arango;John J. Mann;John J. Mann.
Neurochemical Research (2004)
Genetic inactivation of the Serotonin(1A) receptor in mice results in downregulation of major GABA(A) receptor alpha subunits, reduction of GABA(A) receptor binding, and benzodiazepine-resistant anxiety.
Etienne Sibille;Constantine Pavlides;Dietmar Benke;Miklos Toth.
The Journal of Neuroscience (2000)
Neuronal tryptophan hydroxylase mRNA expression in the human dorsal and median raphe nuclei: major depression and suicide.
Helene Bach-Mizrachi;Helene Bach-Mizrachi;Mark D Underwood;Mark D Underwood;Suham A Kassir;Mihran J Bakalian.
Molecular aging in human prefrontal cortex is selective and continuous throughout adult life.
Loubna Erraji-Benchekroun;Loubna Erraji-Benchekroun;Mark D. Underwood;Mark D. Underwood;Victoria Arango;Victoria Arango;Hanga Galfalvy;Hanga Galfalvy.
Biological Psychiatry (2005)
Beyond modules and hubs: the potential of gene coexpression networks for investigating molecular mechanisms of complex brain disorders
Chris Gaiteri;Ying Ding;Beverly French;George C. Tseng.
Genes, Brain and Behavior (2014)
Brain-Derived Neurotrophic Factor Signaling and Subgenual Anterior Cingulate Cortex Dysfunction in Major Depressive Disorder
Adam Tripp;Hyunjung Oh;Jean Philippe Guilloux;Keri Martinowich.
American Journal of Psychiatry (2012)
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