D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 60 Citations 13,722 124 World Ranking 7831 National Ranking 136

Overview

What is he best known for?

The fields of study Didier Picard is best known for:

  • Gene
  • Enzyme
  • Transcription factor

Estrogen-related receptor gamma and Nuclear receptor coactivator 2 are the main areas of his Nuclear receptor studies. His study deals with a combination of Estrogen-related receptor gamma and Estrogen receptor. While working on this project, Didier Picard studies both Estrogen receptor and Estrogen receptor alpha. Didier Picard connects Nuclear receptor coactivator 2 with Nuclear receptor in his study. Didier Picard undertakes multidisciplinary studies into Gene and Steroid in his work. In his works, Didier Picard performs multidisciplinary study on Steroid and Hormone. Didier Picard performs multidisciplinary study on Hormone and Hormone receptor in his works. He incorporates Hormone receptor and Steroid hormone in his research. He undertakes interdisciplinary study in the fields of Steroid hormone and Steroid hormone receptor through his works.

His most cited work include:

  • Activation of the unliganded estrogen receptor by EGF involves the MAP kinase pathway and direct phosphorylation. (911 citations)
  • Reduced levels of hsp90 compromise steroid receptor action in vivo (758 citations)
  • Heat-shock protein 90, a chaperone for folding and regulation (719 citations)

What are the main themes of his work throughout his whole career to date

In his works, Didier Picard undertakes multidisciplinary study on Gene and Gene expression. In his research, Didier Picard undertakes multidisciplinary study on Gene expression and Gene. His Cell biology study frequently intersects with other fields, such as Signal transduction. His study connects Cell biology and Signal transduction. His Biochemistry study typically links adjacent topics like Hormone. He combines Hormone and Receptor in his research. As part of his studies on Receptor, he often connects relevant subjects like Steroid hormone. His work often combines Genetics and Computational biology studies. In his research, he performs multidisciplinary study on Computational biology and Biochemistry.

Didier Picard most often published in these fields:

  • Gene (75.00%)
  • Cell biology (67.50%)
  • Biochemistry (60.83%)

What were the highlights of his more recent work (between 2017-2022)?

  • Biochemistry (86.67%)
  • Gene (73.33%)
  • Cell biology (66.67%)

In recent works Didier Picard was focusing on the following fields of study:

His study in In vitro extends to Biochemistry with its themes. In his works, he undertakes multidisciplinary study on Gene and Ubiquitin. Ubiquitin and Gene are two areas of study in which Didier Picard engages in interdisciplinary research. Cell biology connects with themes related to Mitochondrion in his study. His research on Heat shock protein frequently links to adjacent areas such as Genetics. His work on Proteostasis expands to the thematically related Genetics. Didier Picard integrates Proteostasis and Hsp90 in his studies. In his study, he carries out multidisciplinary Hsp90 and Heat shock protein research. His study on Pathology is mostly dedicated to connecting different topics, such as Chaperone (clinical).

Between 2017 and 2022, his most popular works were:

  • Methods to validate Hsp90 inhibitor specificity, to identify off-target effects, and to rethink approaches for further clinical development (78 citations)
  • Implementation of liquid chromatography–high resolution mass spectrometry methods for untargeted metabolomic analyses of biological samples: A tutorial (66 citations)
  • The Hsp70-Hsp90 co-chaperone Hop/Stip1 shifts the proteostatic balance from folding towards degradation (64 citations)

In his most recent research, the most cited works focused on:

  • Gene
  • Clinical trial
  • Cancer

Didier Picard integrates many fields, such as Computational biology and Bioinformatics, in his works. His multidisciplinary approach integrates Bioinformatics and Computational biology in his work. His research combines In vitro and Biochemistry. Didier Picard performs multidisciplinary study on In vitro and Cancer in his works. In his works, he conducts interdisciplinary research on Cancer and Disease. His Disease study frequently links to related topics such as Internal medicine. His study brings together the fields of Neurodegeneration and Internal medicine. He merges many fields, such as Neurodegeneration and Proteostasis, in his writings. His research is interdisciplinary, bridging the disciplines of Genetics and Proteostasis.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Activation of the unliganded estrogen receptor by EGF involves the MAP kinase pathway and direct phosphorylation.

Giuseppe Bunone;Pierre André Briand;Richard J. Miksicek;Didier Picard.
The EMBO Journal (1996)

1210 Citations

Heat-shock protein 90, a chaperone for folding and regulation.

Didier Picard.
Cellular and Molecular Life Sciences (2002)

1008 Citations

The G protein-coupled receptor GPR30 mediates c-fos up-regulation by 17beta-estradiol and phytoestrogens in breast cancer cells.

Marcello Maggiolini;Adele Vivacqua;Giovanna Fasanella;Anna Grazia Recchia.
Journal of Biological Chemistry (2004)

527 Citations

The glucocorticoid responses are shaped by molecular chaperones.

Iwona Grad;Didier Picard.
Molecular and Cellular Endocrinology (2007)

453 Citations

The unfolding stories of GPR30, a new membrane-bound estrogen receptor

Marcello Maggiolini;Didier Picard.
Journal of Endocrinology (2010)

438 Citations

The G protein-coupled receptor GPR30 mediates the proliferative effects induced by 17beta-estradiol and hydroxytamoxifen in endometrial cancer cells.

Adele Vivacqua;Daniela Bonofiglio;Anna Grazia Recchia;Anna Maria Musti.
Molecular Endocrinology (2006)

432 Citations

RNA interference in mammalian cells using siRNAs synthesized with T7 RNA polymerase

Olivier Donzé;Didier Picard.
Nucleic Acids Research (2002)

431 Citations

The Hsp90-binding peptidylprolyl isomerase FKBP52 potentiates glucocorticoid signaling in vivo

Daniel L. Riggs;Patricia J. Roberts;Samantha C. Chirillo;Joyce Cheung-Flynn.
The EMBO Journal (2003)

415 Citations

Signal transduction and transcriptional regulation by glucocorticoid receptor-LexA fusion proteins

Paul J. Godowski;Didier Picard;Keith R. Yamamoto.
Science (1988)

372 Citations

17beta-estradiol, genistein, and 4-hydroxytamoxifen induce the proliferation of thyroid cancer cells through the g protein-coupled receptor GPR30.

Adele Vivacqua;Daniela Bonofiglio;Lidia Albanito;Antonio Madeo.
Molecular Pharmacology (2006)

362 Citations

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