His main research concerns Internal medicine, Immunology, Lupus nephritis, Kidney disease and Lupus erythematosus. His Internal medicine research is multidisciplinary, incorporating elements of Gastroenterology and Endocrinology. His Immunology study integrates concerns from other disciplines, such as Biomarker, Glomerulonephritis and Allele.
His Lupus nephritis research is multidisciplinary, incorporating perspectives in Discontinuation, Treatment outcome, Systemic lupus erythematosus, Cytokine TWEAK and Nephritis. His Kidney disease study combines topics in areas such as Adverse effect and Intensive care medicine. His work carried out in the field of Proteinuria brings together such families of science as Nephropathy, Kidney metabolism, Clinical trial and Renal function.
Brad H. Rovin focuses on Internal medicine, Lupus nephritis, Immunology, Kidney and Kidney disease. The concepts of his Internal medicine study are interwoven with issues in Gastroenterology and Endocrinology. Brad H. Rovin focuses mostly in the field of Lupus nephritis, narrowing it down to topics relating to Systemic lupus erythematosus and, in certain cases, Immunosuppression.
Brad H. Rovin has included themes like Glomerulonephritis and Disease in his Immunology study. As part of one scientific family, Brad H. Rovin deals mainly with the area of Kidney, narrowing it down to issues related to the Acute kidney injury, and often Nephrectomy. His research investigates the connection with Proteinuria and areas like Renal function which intersect with concerns in Placebo.
Brad H. Rovin spends much of his time researching Internal medicine, Lupus nephritis, Kidney, Proteinuria and Immunology. His study in Internal medicine is interdisciplinary in nature, drawing from both Gastroenterology and Placebo. The study incorporates disciplines such as Glomerulonephritis, Inflammation, Adverse effect, Systemic lupus erythematosus and Nephritis in addition to Lupus nephritis.
His Kidney research incorporates elements of Kidney disease, Nephropathy, Biopsy, Pathology and Acute kidney injury. His Kidney disease research integrates issues from Immunosuppression and Intensive care medicine. His Proteinuria study incorporates themes from Nephrotic syndrome and Cohort.
Internal medicine, Lupus nephritis, Kidney disease, Proteinuria and Kidney are his primary areas of study. His biological study spans a wide range of topics, including Gastroenterology and Placebo. His Lupus nephritis research includes elements of Renal pathology, Adverse effect, Systemic lupus erythematosus, Biopsy and Cohort.
His Kidney disease study combines topics from a wide range of disciplines, such as Native kidney, Immunosuppression, Surgery and Intensive care medicine. Brad H. Rovin interconnects Acute kidney injury and Apoptosis in the investigation of issues within Kidney. The Nephrology study combines topics in areas such as Glomerulonephritis and Transplantation.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
The Influence of CCL3L1 Gene-Containing Segmental Duplications on HIV-1/AIDS Susceptibility
Enrique Gonzalez;Hemant Kulkarni;Hector Bolivar;Andrea Mangano.
Science (2005)
Kidney disease: Improving global outcomes (KDIGO) glomerulonephritis work group. KDIGO clinical practice guideline for glomerulonephritis
Daniel C. Cattran;John Feehally;H. Terence Cook;Zhi Hong Liu.
Kidney International (2012)
Mycophenolate Mofetil versus Cyclophosphamide for Induction Treatment of Lupus Nephritis
Gerald B. Appel;Gabriel Contreras;Mary Anne Dooley;Ellen M. Ginzler.
Journal of The American Society of Nephrology (2009)
Efficacy and safety of rituximab in patients with active proliferative lupus nephritis: The lupus nephritis assessment with rituximab study†
Brad H. Rovin;Richard Furie;Kevin Latinis;R. John Looney.
Arthritis & Rheumatism (2012)
A novel polymorphism in the MCP-1 gene regulatory region that influences MCP-1 expression.
Brad H. Rovin;Ling Lu;Ramesh Saxena.
Biochemical and Biophysical Research Communications (1999)
Gene Copy-Number Variation and Associated Polymorphisms of Complement Component C4 in Human Systemic Lupus Erythematosus (SLE): Low Copy Number Is a Risk Factor for and High Copy Number Is a Protective Factor against SLE Susceptibility in European Americans
Yan Yang;Erwin K. Chung;Yee Ling Wu;Stephanie L. Savelli.
American Journal of Human Genetics (2007)
Flavopiridol administered using a pharmacologically derived schedule is associated with marked clinical efficacy in refractory, genetically high-risk chronic lymphocytic leukemia.
John C. Byrd;Thomas S. Lin;James T. Dalton;Di Wu.
Blood (2007)
Activation of Nrf2/ARE pathway protects endothelial cells from oxidant injury and inhibits inflammatory gene expression.
Xi-Lin Chen;Geraldine Dodd;Suzanne Thomas;Xiaolan Zhang.
American Journal of Physiology-heart and Circulatory Physiology (2006)
Update on Lupus Nephritis
Salem Almaani;Alexa Meara;Brad H. Rovin.
Clinical Journal of The American Society of Nephrology (2017)
HIV-1 infection and AIDS dementia are influenced by a mutant MCP-1 allele linked to increased monocyte infiltration of tissues and MCP-1 levels
Enrique Gonzalez;Brad H. Rovin;Luisa Sen;Glen Cooke.
Proceedings of the National Academy of Sciences of the United States of America (2002)
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