What is she best known for?
The fields of study she is best known for:
The scientist’s investigation covers issues in Retina, Cell biology, Biochemistry, Retinal degeneration and Rhodopsin.
Her work on Retinal pigment epithelium as part of general Retina research is frequently linked to Voltage-dependent calcium channel and Calcium-binding protein, bridging the gap between disciplines.
Her Cell biology research incorporates elements of Lipofuscin and Electroretinography.
The Retinal degeneration study combines topics in areas such as Photoreceptor cell, Retinol, Visual phototransduction and Macular degeneration.
In her study, Cytotoxic T cell, Epitope, Antigen and Alternative splicing is inextricably linked to Molecular biology, which falls within the broad field of Rhodopsin.
The study incorporates disciplines such as MAP1LC3B, Sequestosome 1, Physiology and Chaperone-mediated autophagy in addition to Autophagosome.
Her most cited work include:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- Essential role of Ca2+-binding protein 4, a Cav1.4 channel regulator, in photoreceptor synaptic function. (231 citations)
- Retinopathy in Mice Induced by Disrupted All-trans-retinal Clearance (210 citations)
What are the main themes of her work throughout her whole career to date?
Retinal, Retina, Cell biology, Retinal degeneration and Visual phototransduction are her primary areas of study.
Her Retina study integrates concerns from other disciplines, such as In vivo, Recoverin, Immunology and Anatomy.
Her Cell biology research includes themes of Degeneration, Lipofuscin, Biochemistry, Induced pluripotent stem cell and Programmed cell death.
Her research links Autophagy with Programmed cell death.
Her Retinal degeneration research is multidisciplinary, incorporating perspectives in Stargardt disease, Molecular biology, ABCA4, Photoreceptor cell and Pharmacology.
The concepts of her Visual phototransduction study are interwoven with issues in Retinol dehydrogenase and Retinal Cone Photoreceptor Cells.
She most often published in these fields:
- Retinal (44.70%)
- Retina (37.88%)
- Cell biology (34.09%)
What were the highlights of her more recent work (between 2015-2021)?
- Retinal (44.70%)
- Cell biology (34.09%)
- Retina (37.88%)
In recent papers she was focusing on the following fields of study:
Her primary areas of investigation include Retinal, Cell biology, Retina, Retinal degeneration and Visual phototransduction.
Akiko Maeda has included themes like Cell therapy, In vitro, Induced pluripotent stem cell and In vivo in her Retinal study.
Her research investigates the connection between Cell biology and topics such as Retinal pigment epithelium that intersect with issues in Programmed cell death and Inflammation.
Her work deals with themes such as Chaperone-mediated autophagy, Computational biology and Autolysosome, which intersect with Programmed cell death.
Her work carried out in the field of Autolysosome brings together such families of science as MAP1LC3B, Sequestosome 1, BECN1 and Physiology.
Her Visual phototransduction study combines topics in areas such as Electroretinography, Retinol dehydrogenase, Anatomy, Photoreceptor cell and Rhodopsin.
Between 2015 and 2021, her most popular works were:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- Erratum to: Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (Autophagy, 12, 1, 1-222, 10.1080/15548627.2015.1100356 (40 citations)
- Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition) (38 citations)
In her most recent research, the most cited papers focused on:
Her primary areas of study are Cell biology, Retina, Programmed cell death, Retinal degeneration and Visual phototransduction.
She works mostly in the field of Cell biology, limiting it down to concerns involving Retinal and, occasionally, All trans retinol, Retinol dehydrogenase and Retinol.
Akiko Maeda focuses mostly in the field of Programmed cell death, narrowing it down to topics relating to Autophagy and, in certain cases, Computational biology.
Her Computational biology research integrates issues from BECN1 and Multicellular organism.
Her research integrates issues of Retinal pigment epithelium, Stargardt disease, Regulation of gene expression and ABCA4 in her study of Retinal degeneration.
Her Autolysosome study incorporates themes from MAP1LC3B, Sequestosome 1 and Physiology.
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