His primary areas of study are Transgene, Endocrinology, Internal medicine, Molecular biology and Antioxidant. His Transgene study deals with Superoxide dismutase intersecting with Hyperoxia, Mitochondrion and Immunology. His specific area of interest is Endocrinology, where Ye-Shih Ho studies Oxidative stress.
His work carried out in the field of Molecular biology brings together such families of science as Myocarditis, Enzyme, Virus, Gene and GPX1. His Antioxidant research focuses on Knockout mouse and how it connects with Hydrogen peroxide, Gene targeting and Lung injury. His biological study spans a wide range of topics, including Ischemia and Ratón.
His primary areas of investigation include Molecular biology, Oxidative stress, Cell biology, Biochemistry and Internal medicine. His studies deal with areas such as Apoptosis, Glutathione, Wild type, Knockout mouse and Mitochondrion as well as Molecular biology. His work in Oxidative stress addresses subjects such as Lung injury, which are connected to disciplines such as TBARS.
His studies in Cell biology integrate themes in fields like Glutaredoxin and Gene. His Internal medicine research integrates issues from Endocrinology, Protein kinase B and Cardiology. The various areas that Ye-Shih Ho examines in his Endocrinology study include Vascular endothelial growth factor and Ischemia.
Ye-Shih Ho spends much of his time researching Glutaredoxin, Cell biology, Internal medicine, Oxidative stress and Endocrinology. As part of his Biochemistry and Glutathione and Glutaredoxin studies, Ye-Shih Ho is studying Glutaredoxin. His Internal medicine research incorporates elements of Protein kinase B, Knockout mouse and Pharmacology.
His research in Oxidative stress intersects with topics in Cancer research, Genetically modified mouse, Mitochondrion and Pulmonary fibrosis. The concepts of his Endocrinology study are interwoven with issues in Vascular endothelial growth factor, Downregulation and upregulation, Angiogenesis and Ischemia. Ye-Shih Ho combines subjects such as Molecular biology and Pathophysiology with his study of Immunology.
His main research concerns Oxidative stress, Reactive oxygen species, Biochemistry, GPX1 and Molecular biology. Within the field of Internal medicine and Endocrinology Ye-Shih Ho studies Oxidative stress. His Endocrinology study combines topics in areas such as Protein kinase B and Neovascularization.
His Reactive oxygen species study is focused on Cell biology in general. Ye-Shih Ho has included themes like Lipid peroxidation, Wild type and Hyperoxia in his GPX1 study. His Molecular biology research includes themes of Superoxide dismutase, Lipopolysaccharide, Immunology, Monocyte and Lung.
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Dynamic redox control of NF-kappaB through glutaredoxin-regulated S-glutathionylation of inhibitory kappaB kinase beta.
Niki L. Reynaert;Albert van der Vliet;Amy S. Guala;Toby McGovern.
Proceedings of the National Academy of Sciences of the United States of America (2006)
Overexpression of Bcl-2 attenuates apoptosis and protects against myocardial I/R injury in transgenic mice.
Zhongyi Chen;Chu Chang Chua;Ye-Shih Ho;Ronald C. Hamdy.
American Journal of Physiology-heart and Circulatory Physiology (2001)
Mice lacking catalase develop normally but show differential sensitivity to oxidant tissue injury.
Ye Shih Ho;Ye Xiong;Wanchao Ma;Abraham Spector.
Journal of Biological Chemistry (2004)
Overexpression of MnSOD protects against myocardial ischemia/reperfusion injury in transgenic mice.
Zhongyi Chen;Brian Siu;Ye-Shih Ho;Renaud Vincent.
Journal of Molecular and Cellular Cardiology (1998)
Hypoxia activates NADPH oxidase to increase [ROS]i and [Ca2+]i through the mitochondrial ROS-PKCepsilon signaling axis in pulmonary artery smooth muscle cells.
Rakesh Rathore;Yun Min Zheng;Chun Feng Niu;Qing Hua Liu.
Free Radical Biology and Medicine (2008)
Glutathione peroxidase protects mice from viral-induced myocarditis
M. A. Beck;R. S. Esworthy;Y.‐S. Ho;F.‐F. Chu.
The FASEB Journal (1998)
Overexpression of mitochondrial superoxide dismutase in mice protects the retina from diabetes-induced oxidative stress.
Renu A. Kowluru;Vibhuti Kowluru;Ye Xiong;Ye Shih Ho.
Free Radical Biology and Medicine (2006)
Transgenic mice overexpressing glutathione peroxidase are resistant to myocardial ischemia reperfusion injury.
Tetsuya Yoshida;Masazumi Watanabe;Daniel T. Engelman;Richard M. Engelman.
Journal of Molecular and Cellular Cardiology (1996)
Hemoglobin autoxidation and regulation of endogenous H2O2 levels in erythrocytes
Robert M. Johnson;Gerard Goyette;Y. Ravindranath;Ye-Shih Ho.
Free Radical Biology and Medicine (2005)
The nature of antioxidant defense mechanisms : a lesson from transgenic studies
Ye-Shih Ho;Jean-Luc Magnenat;Mary Gargano;Jin Cao.
Environmental Health Perspectives (1998)
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