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Neuroscience

D-Index
70
Citations
14315
World Ranking
2556
National Ranking
1206

Overview

Ronald E. See is affiliated with the Medical University of South Carolina in the United States. Their research spans several areas within neuroscience and biochemistry focused on understanding brain mechanisms and signaling pathways.

Their recent publications include the 2020 paper titled "Neural Substrates and Circuits of Drug Addiction" published in Cold Spring Harbor Perspectives in Medicine, and the 2021 article "Assessment of multiple salivary biomarkers during repetitive transcranial magnetic stimulation (rTMS) treatment for major depression" in Psychiatry Research.

Frequent coauthors working alongside Ronald E. See include:

  • Matthew W. Feltenstein
  • Rita A. Fuchs
  • Branon Eusebio
  • David A. Agnew
  • Mark Heatwole

The scientist's work has been published primarily in the following venues:

  • Cold Spring Harbor Perspectives in Medicine
  • Psychiatry Research

Ronald E. See's main fields of study are:

  • Neuroscience
  • Biochemistry, Genetics and Molecular Biology

Within these fields, their subfields of study include:

  • Molecular Biology
  • Cellular and Molecular Neuroscience
  • Neurology
  • Behavioral Neuroscience
  • Cognitive Neuroscience

The main research topics addressed in their publications feature:

  • Neurotransmitter Receptor Influence on Behavior
  • Nicotinic Acetylcholine Receptors Study
  • Receptor Mechanisms and Signaling
  • Transcranial Magnetic Stimulation Studies
  • Stress Responses and Cortisol
  • Functional Brain Connectivity Studies

Best Publications

  • The role of the dorsomedial prefrontal cortex, basolateral amygdala, and dorsal hippocampus in contextual reinstatement of cocaine seeking in rats.

    Rita A Fuchs;K Allison Evans;Christopher C Ledford;Macon P Parker

  • The neurocircuitry of addiction: an overview.

    M W Feltenstein;R E See

  • Selective inactivation of the dorsomedial prefrontal cortex and the basolateral amygdala attenuates conditioned-cued reinstatement of extinguished cocaine-seeking behavior in rats.

    Joselyn McLaughlin;Ronald E. See

  • Lesions of the basolateral amygdala abolish the ability of drug associated cues to reinstate responding during withdrawal from self-administered cocaine

    William M Meil;Ronald E See

  • Different neural substrates mediate cocaine seeking after abstinence versus extinction training: a critical role for the dorsolateral caudate-putamen.

    Rita A. Fuchs;R. Kyle Branham;Ronald E. See

  • Dopamine, but not glutamate, receptor blockade in the basolateral amygdala attenuates conditioned reward in a rat model of relapse to cocaine-seeking behavior

    Ronald E. See;Paul J. Kruzich;Jeffrey W. Grimm

  • Differential involvement of the core and shell subregions of the nucleus accumbens in conditioned cue-induced reinstatement of cocaine seeking in rats.

    Rita A. Fuchs;K. Allison Evans;Macon C. Parker;Ronald E. See

  • Dissociation of Primary and Secondary Reward-Relevant Limbic Nuclei in an Animal Model of Relapse

    Jeffrey W Grimm;Ronald E See

  • Conditioned cued recovery of responding following prolonged withdrawal from self-administered cocaine in rats: an animal model of relapse.

    W.M. Meil;R.E. See

  • Orexin/hypocretin signaling at the orexin 1 receptor regulates cue-elicited cocaine-seeking.

    Rachel J. Smith;Ronald E. See;Gary Aston-Jones

  • Neural substrates of conditioned-cued relapse to drug-seeking behavior.

    Ronald E See

  • Basolateral amygdala inactivation abolishes conditioned stimulus- and heroin-induced reinstatement of extinguished heroin-seeking behavior in rats.

    Rita A. Fuchs;Ronald E. See

  • Drug Addiction, Relapse, and the Amygdala

    Ronald E. See;Rita A. Fuchs;Christopher C. Ledford;Joselyn McLAUGHLIN

  • Differential involvement of orbitofrontal cortex subregions in conditioned cue-induced and cocaine-primed reinstatement of cocaine seeking in rats

    Rita A. Fuchs;K. Allison Evans;Macon P. Parker;Ronald E. See

  • A BDNF infusion into the medial prefrontal cortex suppresses cocaine seeking in rats.

    William J. Berglind;Ronald E. See;Rita A. Fuchs;Shannon M. Ghee

  • The neural circuitry underlying reinstatement of heroin-seeking behavior in an animal model of relapse.

    J.L. Rogers;S. Ghee;R.E. See

  • Neural substrates of cocaine-cue associations that trigger relapse.

    Ronald E. See

  • Reversing cocaine-induced synaptic potentiation provides enduring protection from relapse

    Khaled Moussawi;Wenhua Zhou;Haowei Shen;Carmela M. Reichel

  • Selective inactivation of the ventral hippocampus attenuates cue-induced and cocaine-primed reinstatement of drug-seeking in rats.

    Jason L. Rogers;Ronald E. See

  • Potentiation of cocaine-primed reinstatement of drug seeking in female rats during estrus.

    Tod E. Kippin;Rita A. Fuchs;Ritu H. Mehta;Jordan M. Case

  • Differential contributions of the basolateral and central amygdala in the acquisition and expression of conditioned relapse to cocaine-seeking behavior.

    Paul J. Kruzich;Ronald E. See

Frequent Co-Authors

Jacqueline F. McGinty
Jacqueline F. McGinty Medical University of South Carolina
Peter W. Kalivas
Peter W. Kalivas Medical University of South Carolina
Gary Aston-Jones
Gary Aston-Jones Rutgers, The State University of New Jersey
Kathleen T. Brady
Kathleen T. Brady Medical University of South Carolina
Barbara A. Sorg
Barbara A. Sorg Washington State University Vancouver
Mark S. George
Mark S. George Medical University of South Carolina
John M. Roll
John M. Roll Washington State University
Xingbao Li
Xingbao Li Medical University of South Carolina
Patrick K. Randall
Patrick K. Randall Medical University of South Carolina
Karen K. Szumlinski
Karen K. Szumlinski University of California, Santa Barbara

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