Ronald E. See spends much of his time researching Neuroscience, Self-administration, Extinction, Basolateral amygdala and Addiction. His work in the fields of Amygdala, Prefrontal cortex and Nucleus accumbens overlaps with other areas such as Classical conditioning and GABA receptor. His Self-administration study improves the overall literature in Pharmacology.
His study in Extinction is interdisciplinary in nature, drawing from both Endocrinology, Drug seeking, Internal medicine, Baclofen and Receptor antagonist. His Basolateral amygdala study combines topics in areas such as Dopamine receptor D1 and Tetrodotoxin. His Addiction research includes themes of Memoria, Methamphetamine, Clinical psychology and Novel object recognition.
His scientific interests lie mostly in Self-administration, Neuroscience, Endocrinology, Internal medicine and Extinction. His Self-administration study contributes to a more complete understanding of Pharmacology. His research investigates the link between Neuroscience and topics such as Methamphetamine that cross with problems in Modafinil.
The Oxytocin, Raclopride, Putamen and Caudate nucleus research Ronald E. See does as part of his general Internal medicine study is frequently linked to other disciplines of science, such as Meth-, therefore creating a link between diverse domains of science. His Extinction research is multidisciplinary, relying on both Anesthesia, Yohimbine, Muscimol, Dopamine receptor D1 and Baclofen. The Basolateral amygdala study combines topics in areas such as Hippocampus and Tetrodotoxin.
His primary areas of study are Self-administration, Addiction, Methamphetamine, Internal medicine and Endocrinology. His Self-administration research includes elements of Glutamate receptor, Extinction, Nucleus accumbens and Yohimbine. Ronald E. See has researched Extinction in several fields, including Anesthesia, Physiology and Clinical psychology.
Ronald E. See has included themes like Cocaine seeking, Neuroplasticity and Orexin in his Addiction study. The concepts of his Methamphetamine study are interwoven with issues in Prefrontal cortex and Neuroscience. His study in the fields of Oxytocin, Striatum, Sex characteristics and Kainic acid under the domain of Internal medicine overlaps with other disciplines such as Pilocarpine.
Ronald E. See mostly deals with Self-administration, Addiction, Methamphetamine, Meth- and Pharmacology. His Self-administration study combines topics from a wide range of disciplines, such as Extinction and Endocrinology. His Methamphetamine research is multidisciplinary, incorporating perspectives in Prefrontal cortex and Neuroscience.
His primary area of study in Neuroscience is in the field of Striatum. His research in Pharmacology intersects with topics in Orexin receptor, Orexin, Nucleus accumbens and Antagonist. His Nucleus accumbens research is multidisciplinary, incorporating elements of Glutamate receptor, Social behavior and Microdialysis.
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The role of the dorsomedial prefrontal cortex, basolateral amygdala, and dorsal hippocampus in contextual reinstatement of cocaine seeking in rats.
Rita A Fuchs;K Allison Evans;Christopher C Ledford;Macon P Parker.
The neurocircuitry of addiction: an overview.
M W Feltenstein;R E See.
British Journal of Pharmacology (2009)
Selective inactivation of the dorsomedial prefrontal cortex and the basolateral amygdala attenuates conditioned-cued reinstatement of extinguished cocaine-seeking behavior in rats.
Joselyn McLaughlin;Ronald E. See.
Lesions of the basolateral amygdala abolish the ability of drug associated cues to reinstate responding during withdrawal from self-administered cocaine
William M Meil;Ronald E See.
Behavioural Brain Research (1997)
Different neural substrates mediate cocaine seeking after abstinence versus extinction training: a critical role for the dorsolateral caudate-putamen.
Rita A. Fuchs;R. Kyle Branham;Ronald E. See.
The Journal of Neuroscience (2006)
Dopamine, but not glutamate, receptor blockade in the basolateral amygdala attenuates conditioned reward in a rat model of relapse to cocaine-seeking behavior
Ronald E. See;Paul J. Kruzich;Jeffrey W. Grimm.
Differential involvement of the core and shell subregions of the nucleus accumbens in conditioned cue-induced reinstatement of cocaine seeking in rats.
Rita A. Fuchs;K. Allison Evans;Macon C. Parker;Ronald E. See.
Dissociation of Primary and Secondary Reward-Relevant Limbic Nuclei in an Animal Model of Relapse
Jeffrey W Grimm;Ronald E See.
Conditioned cued recovery of responding following prolonged withdrawal from self-administered cocaine in rats: an animal model of relapse.
W.M. Meil;R.E. See.
Behavioural Pharmacology (1996)
Neural substrates of conditioned-cued relapse to drug-seeking behavior.
Ronald E See.
Pharmacology, Biochemistry and Behavior (2002)
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