Ralf Dechend mostly deals with Internal medicine, Endocrinology, Angiotensin II, Renin–angiotensin system and Receptor. Ralf Dechend studied Internal medicine and Placenta that intersect with Plasma renin activity and Vitamin C. His research on Endocrinology often connects related topics like Preeclampsia.
The study incorporates disciplines such as Tumor necrosis factor alpha, Inflammation, Endothelin receptor, Valsartan and Kidney in addition to Angiotensin II. His Renin–angiotensin system research includes elements of Mineralocorticoid, Aldosterone, Muscle hypertrophy and Kinase. Ralf Dechend has researched Receptor in several fields, including Epitope and Cell biology.
Ralf Dechend mainly focuses on Internal medicine, Endocrinology, Preeclampsia, Angiotensin II and Blood pressure. In his work, Diastole is strongly intertwined with Cardiology, which is a subfield of Internal medicine. He combines subjects such as Inflammation and Ischemia with his study of Endocrinology.
He works mostly in the field of Preeclampsia, limiting it down to concerns involving Autoantibody and, occasionally, Type i receptor. Ralf Dechend interconnects Fibrosis, Reactive oxygen species, Cerivastatin and Immunology in the investigation of issues within Angiotensin II. His Blood pressure research incorporates elements of Vitamin D and neurology, Cohort and Intensive care medicine.
Ralf Dechend mainly investigates Internal medicine, Preeclampsia, Pregnancy, Endocrinology and Blood pressure. In most of his Internal medicine studies, his work intersects topics such as Cardiology. The Preeclampsia study combines topics in areas such as Autoantibody, Albuminuria, Fetus and Obstetrics.
His study in Endocrinology is interdisciplinary in nature, drawing from both Inflammation, Trophoblast and Ischemia. The various areas that Ralf Dechend examines in his Blood pressure study include Gut flora, Case-control study, Fatty acid, Vitamin D and neurology and Cohort. His Angiotensin II study frequently draws connections to adjacent fields such as Kidney.
His main research concerns Internal medicine, Preeclampsia, Endocrinology, Pregnancy and Blood pressure. Internal medicine is closely attributed to Cardiology in his research. His Preeclampsia research is multidisciplinary, incorporating elements of Receptor, Andrology, Spiral artery and Intrauterine growth restriction.
He studies Endocrinology, focusing on Renin–angiotensin system in particular. His Pregnancy research incorporates themes from Diabetes mellitus, Pravastatin, Pathophysiology and Disease. His Blood pressure research is multidisciplinary, relying on both Gut flora, Gastrointestinal Microbiome, Experimental autoimmune encephalomyelitis, Encephalomyelitis and Autoimmunity.
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Angiotensin II type 1-receptor activating antibodies in renal-allograft rejection.
Duska Dragun;Dominik N. Müller;Jan Hinrich Bräsen;Lutz Fritsche.
The New England Journal of Medicine (2005)
Salt-responsive gut commensal modulates TH17 axis and disease
Nicola Wilck;Mariana G. Matus;Sean M. Kearney;Scott W. Olesen.
NF-κB Inhibition Ameliorates Angiotensin II–Induced Inflammatory Damage in Rats
Dominik N. Muller;Ralf Dechend;Eero M. A. Mervaala;Joon-Keun Park.
AT1 Receptor Agonistic Antibodies From Preeclamptic Patients Stimulate NADPH Oxidase
Ralf Dechend;Christiane Viedt;Dominik N. Müller;Bernhard Ugele.
Oxidative Stress and Preeclampsia: Rationale for Antioxidant Clinical Trials
Maarten T. M. Raijmakers;Ralf Dechend;Lucilla Poston.
The Bcl-3 oncoprotein acts as a bridging factor between NF-kappaB/Rel and nuclear co-regulators.
R Dechend;F Hirano;K Lehmann;V Heissmeyer.
Arachidonic Acid-metabolizing Cytochrome P450 Enzymes Are Targets of ω-3 Fatty Acids
Cosima Arnold;Marija Markovic;Katrin Blossey;Gerd Wallukat.
Journal of Biological Chemistry (2010)
Immunosuppressive treatment protects against angiotensin II-induced renal damage.
Dominik N. Muller;Dominik N. Muller;Erdenechimeg Shagdarsuren;Joon-Keun Park;Ralf Dechend.
American Journal of Pathology (2002)
Aliskiren, a Human Renin Inhibitor, Ameliorates Cardiac and Renal Damage in Double-Transgenic Rats
Bernhard Pilz;Erdenechimeg Shagdarsuren;Maren Wellner;Anette Fiebeler.
Chlamydia pneumoniae infection of vascular smooth muscle and endothelial cells activates NF-kappaB and induces tissue factor and PAI-1 expression: a potential link to accelerated arteriosclerosis.
Ralf Dechend;M. Maass;J. Gieffers;R. Dietz.
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