Philip J. Thompson mostly deals with Immunology, Asthma, Genome-wide association study, Single-nucleotide polymorphism and Internal medicine. As part of his studies on Immunology, Philip J. Thompson frequently links adjacent subjects like Hyperphosphorylation. His study in Asthma is interdisciplinary in nature, drawing from both Allergy, Allele and Cohort.
His research in Genome-wide association study intersects with topics in Case-control study, Disease, Genetic association and Atopic dermatitis. His biological study deals with issues like Locus, which deal with fields such as Filaggrin, Immune dysregulation and Linkage disequilibrium. His studies deal with areas such as Placebo, Surgery and Physical therapy as well as Internal medicine.
His main research concerns Immunology, Asthma, Internal medicine, Endocrinology and Pathology. Philip J. Thompson interconnects Genome-wide association study and Allele in the investigation of issues within Immunology. His research integrates issues of Hay fever and Genetic association in his study of Genome-wide association study.
In his research, Haplotype and Odds ratio is intimately related to Single-nucleotide polymorphism, which falls under the overarching field of Asthma. His Internal medicine research integrates issues from Gastroenterology, Placebo, Surgery and Cardiology. Philip J. Thompson has included themes like Platelet activation and Arachidonic acid in his Endocrinology study.
His primary scientific interests are in Immunology, Genome-wide association study, Asthma, Internal medicine and Genetics. His Immunology study combines topics from a wide range of disciplines, such as Allele and Genetic architecture. The Genome-wide association study study combines topics in areas such as Human leukocyte antigen, Hay fever, Disease and Genetic association.
His Asthma study incorporates themes from Pediatrics and Environmental health. His work carried out in the field of Internal medicine brings together such families of science as Gastroenterology and Placebo. In his study, which falls under the umbrella issue of Atopy, Odds ratio is strongly linked to Risk factor.
Philip J. Thompson spends much of his time researching Genome-wide association study, Immunology, Internal medicine, Asthma and Hay fever. His Genome-wide association study study combines topics in areas such as Odds ratio, Disease and Genetic association. His Immunology study frequently draws connections between adjacent fields such as Genetic architecture.
Lung volumes, Functional residual capacity, Glucocorticoid and Reslizumab is closely connected to Gastroenterology in his research, which is encompassed under the umbrella topic of Internal medicine. Particularly relevant to Asthma care is his body of work in Asthma. His Single-nucleotide polymorphism research is multidisciplinary, incorporating elements of Quantitative trait locus, Cell activation and CLK3.
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Oral Glucocorticoid-Sparing Effect of Mepolizumab in Eosinophilic Asthma
Elisabeth H. Bel;Sally E. Wenzel;Philip J. Thompson;Charlene M. Prazma.
The New England Journal of Medicine (2014)
Der p 1 facilitates transepithelial allergen delivery by disruption of tight junctions
Hong Wan;Helen L. Winton;Christian Soeller;Euan R. Tovey.
Journal of Clinical Investigation (1999)
Sequence variants affecting eosinophil numbers associate with asthma and myocardial infarction.
Daniel F Gudbjartsson;Unnur S Bjornsdottir;Unnur S Bjornsdottir;Eva Halapi;Anna Helgadottir.
Nature Genetics (2009)
House Dust Mite Allergens Induce Proinflammatory Cytokines from Respiratory Epithelial Cells: The Cysteine Protease Allergen, Der p 1, Activates Protease-Activated Receptor (PAR)-2 and Inactivates PAR-1
Nithiananthan Asokananthan;Peter T. Graham;David J. Stewart;Anthony J. Bakker.
Journal of Immunology (2002)
Activation of Protease-Activated Receptor (PAR)-1, PAR-2, and PAR-4 Stimulates IL-6, IL-8, and Prostaglandin E2 Release from Human Respiratory Epithelial Cells
Nithiananthan Asokananthan;Peter T. Graham;Joshua Fink;Darryl A. Knight.
Journal of Immunology (2002)
Identification of IL6R and chromosome 11q13.5 as risk loci for asthma
Manuel A Ferreira;Melanie C Matheson;David L Duffy;Guy B Marks.
The Lancet (2011)
Multi-ancestry genome-wide association study of 21,000 cases and 95,000 controls identifies new risk loci for atopic dermatitis
Lavinia Paternoster;Marie Standl;Johannes Waage;Hansjoerg Baurecht.
Nature Genetics (2015)
Six minute walk distance in healthy subjects aged 55-75 years
Bernadine Camarri;Peter R. Eastwood;Peter R. Eastwood;Peter R. Eastwood;Nola M. Cecins;Nola M. Cecins;Philip J. Thompson.
Respiratory Medicine (2006)
Protease-activated receptors in human airways: upregulation of PAR-2 in respiratory epithelium from patients with asthma.
Darryl A. Knight;Sam Lim;Amelia K. Scaffidi;Nicholai Roche.
The Journal of Allergy and Clinical Immunology (2001)
The transmembrane protein occludin of epithelial tight junctions is a functional target for serine peptidases from faecal pellets of Dermatophagoides pteronyssinus.
H. Wan;H. L. Winton;C. Soeller;G. W. Taylor.
Clinical & Experimental Allergy (2001)
Profile was last updated on December 6th, 2021.
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