His main research concerns Immunology, Myeloperoxidase, Anti-neutrophil cytoplasmic antibody, Pathology and Inflammation. His Immunology study frequently links to other fields, such as Vasculitis. His Myeloperoxidase research integrates issues from Respiratory burst, Glomerulonephritis, Granulocyte and Neutrophil extravasation.
His biological study spans a wide range of topics, including Creatinine, Immunoglobulin G, Microscopic polyangiitis and Systemic vasculitis. His Inflammation study incorporates themes from Endocrinology, Endothelium, Low-density lipoprotein and Cell biology. In his study, Kidney is strongly linked to Cancer research, which falls under the umbrella field of Tumor necrosis factor alpha.
His scientific interests lie mostly in Immunology, Pathology, Internal medicine, Vasculitis and Myeloperoxidase. His work carried out in the field of Immunology brings together such families of science as Glomerulonephritis and Anti-neutrophil cytoplasmic antibody. His Glomerulonephritis research incorporates elements of Proinflammatory cytokine and Kidney disease.
His Internal medicine study combines topics from a wide range of disciplines, such as Gastroenterology and Endocrinology. His Vasculitis study combines topics in areas such as Alternative complement pathway and Pathogenesis. As part of his studies on Myeloperoxidase, Peter Heeringa frequently links adjacent subjects like Granulocyte.
Peter Heeringa mostly deals with Immunology, Pathology, Internal medicine, Vasculitis and Giant cell arteritis. Immunology is frequently linked to Granulomatosis with polyangiitis in his study. His study explores the link between Pathology and topics such as Macrophage that cross with problems in Angiogenesis.
His Internal medicine research is multidisciplinary, incorporating elements of Gastroenterology and Endocrinology. Peter Heeringa works on Vasculitis which deals in particular with Anti-neutrophil cytoplasmic antibody. The Giant cell arteritis study combines topics in areas such as Inflammation, Chemokine and Pathogenesis.
Immunology, Vasculitis, Internal medicine, Inflammation and Pathology are his primary areas of study. His research in Immunology intersects with topics in Polymyalgia rheumatica and Signal transduction, Cell biology. The study incorporates disciplines such as Neutrophil extracellular traps, Extracellular Traps, Active disease, Autoantibody and Kidney in addition to Vasculitis.
As a part of the same scientific family, Peter Heeringa mostly works in the field of Internal medicine, focusing on Endocrinology and, on occasion, Systemic inflammation, Receptor and Cytokine. Peter Heeringa regularly links together related areas like MEDLINE in his Pathology studies. The various areas that Peter Heeringa examines in his Proinflammatory cytokine study include Tumor necrosis factor alpha, Glomerulonephritis, Neutralizing antibody, Monoclonal and HMGB1.
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Antineutrophil cytoplasmic autoantibodies specific for myeloperoxidase cause glomerulonephritis and vasculitis in mice
Hong Xiao;Peter Heeringa;Peiqi Hu;Zhi Liu.
Journal of Clinical Investigation (2002)
Alternative complement pathway in the pathogenesis of disease mediated by anti-neutrophil cytoplasmic autoantibodies
Hong Xiao;Adrian Schreiber;Peter Heeringa;Ronald J. Falk.
American Journal of Pathology (2007)
Protective role of endothelial nitric oxide synthase
Ester W J A Albrecht;Coen A Stegeman;Peter Heeringa;Robert Henning.
The Journal of Pathology (2003)
Myeloperoxidase: Molecular Mechanisms of Action and Their Relevance to Human Health and Disease
Betty S. van der Veen;Menno P. J. de Winther;Peter Heeringa.
Antioxidants & Redox Signaling (2009)
Transforming growth factor-beta mediates balance between inflammation and fibrosis during plaque progression.
Esther Lutgens;Marion Gijbels;Marjan Smook;Peter Heeringa.
Arteriosclerosis, Thrombosis, and Vascular Biology (2002)
The Role of Neutrophils in the Induction of Glomerulonephritis by Anti-Myeloperoxidase Antibodies
Hong Xiao;Peter Heeringa;Zhi Liu;Dennis Huugen.
American Journal of Pathology (2005)
Complement Factor C5a Mediates Renal Ischemia-Reperfusion Injury Independent from Neutrophils
Bart de Vries;Jörg Köhl;Wouter K. G. Leclercq;Tim G. A. M. Wolfs.
Journal of Immunology (2003)
Mechanisms of Disease: pathogenesis and treatment of ANCA-associated vasculitides
Cees G M Kallenberg;Peter Heeringa;Coen A Stegeman.
Nature Reviews Rheumatology (2006)
Activation of granulocytes by anti-neutrophil cytoplasmic antibodies (ANCA): a Fc gamma RII-dependent process.
A. H. L. Mulder;P. Heeringa;E. Brouwer;P. C. Limburg.
Clinical and Experimental Immunology (2008)
Inhibition of complement factor C5 protects against anti-myeloperoxidase antibody-mediated glomerulonephritis in mice
D. Huugen;A. van Esch;H. Xiao;C.-J. Peutz-Kootstra.
Kidney International (2007)
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