What is he best known for?
The fields of study he is best known for:
His primary areas of investigation include Cell biology, Molecular biology, Lamin, Muscular dystrophy and Nuclear protein.
His studies deal with areas such as Immunology, Genetics and Cellular differentiation as well as Cell biology.
His study looks at the intersection of Lamin and topics like Emerin with Emery–Dreifuss muscular dystrophy and Cell signaling.
Nadir M. Maraldi has researched Muscular dystrophy in several fields, including Myocyte and Collagen VI, Bethlem myopathy, Ullrich congenital muscular dystrophy.
His Nuclear protein research is multidisciplinary, incorporating elements of Nuclear matrix, Cell nucleus, Internal medicine and Endocrinology.
His work carried out in the field of Skeletal muscle brings together such families of science as Degeneration and Autophagy.
His most cited work include:
- Autophagy is defective in collagen VI muscular dystrophies, and its reactivation rescues myofiber degeneration (381 citations)
- Autophagy is defective in collagen VI muscular dystrophies, and its reactivation rescues myofiber degeneration (381 citations)
- Altered pre-lamin A processing is a common mechanism leading to lipodystrophy (182 citations)
What are the main themes of his work throughout his whole career to date?
Nadir M. Maraldi mostly deals with Cell biology, Molecular biology, Lamin, Biochemistry and Chromatin.
His Cell biology research includes themes of Genetics and Nuclear protein.
His Molecular biology research incorporates elements of Collagen VI, Ullrich congenital muscular dystrophy, DNA, Phosphorylation and Phosphatidylinositol 4,5-bisphosphate.
In his study, Skeletal muscle is strongly linked to Pathology, which falls under the umbrella field of Collagen VI.
His research integrates issues of Cellular differentiation, Transcription factor, Nuclear lamina and Emerin in his study of Lamin.
His work carried out in the field of Chromatin brings together such families of science as Biophysics and Mandibuloacral dysplasia.
He most often published in these fields:
- Cell biology (41.05%)
- Molecular biology (28.42%)
- Lamin (20.53%)
What were the highlights of his more recent work (between 2010-2019)?
- Cell biology (41.05%)
- Collagen VI (16.84%)
- Lamin (20.53%)
In recent papers he was focusing on the following fields of study:
Nadir M. Maraldi mainly investigates Cell biology, Collagen VI, Lamin, Muscular dystrophy and Molecular biology.
His Cell biology research is multidisciplinary, incorporating elements of Genetics, Nuclear protein, Emerin and Cellular differentiation.
His Collagen VI research is mostly focused on the topic Ullrich congenital muscular dystrophy.
His research investigates the link between Ullrich congenital muscular dystrophy and topics such as Bethlem myopathy that cross with problems in Myopathy and Pathology.
His Lamin study combines topics from a wide range of disciplines, such as Chromatin, Phenotype, Transcription factor and Nuclear lamina.
His studies in Muscular dystrophy integrate themes in fields like Emery–Dreifuss muscular dystrophy, Cyclophilin and Anatomy.
Between 2010 and 2019, his most popular works were:
- Laminopathies and lamin‐associated signaling pathways (84 citations)
- Prelamin A mediated recruitment of SUN1 to the nuclear envelope directs nuclear positioning in human muscle (61 citations)
- The protein kinase Akt/PKB regulates both prelamin A degradation and Lmna gene expression (60 citations)
In his most recent research, the most cited papers focused on:
Nadir M. Maraldi focuses on Cell biology, Lamin, Genetics, LMNA and Chromatin.
His study in Cell biology is interdisciplinary in nature, drawing from both Multipotent Stem Cell and Cellular differentiation.
As a part of the same scientific study, Nadir M. Maraldi usually deals with the Cellular differentiation, concentrating on Myogenesis and frequently concerns with Muscular dystrophy.
His Muscular dystrophy research focuses on Muscle strength and how it relates to Ullrich congenital muscular dystrophy and Collagen VI.
The various areas that he examines in his Lamin study include Cell nucleus and Nuclear lamina.
His research investigates the connection between LMNA and topics such as Nuclear protein that intersect with problems in Cell signaling, Nuclear matrix, Signal transduction and Progeroid syndromes.
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