Michael S. Glickman mainly investigates Mycobacterium tuberculosis, Microbiology, Genetics, Immunology and Tuberculosis. The Mycobacterium tuberculosis study combines topics in areas such as Virulence, Ribosomal RNA, Transcription factor, RRNA transcription and Effector. His work deals with themes such as Cord factor, Trehalose dimycolate, Stringent response, Mycolic acid and Innate immune system, which intersect with Microbiology.
His Immunology research focuses on subjects like Infectious disease, which are linked to Leprosy, Mycobacterium bovis and relapsing fever. His study looks at the intersection of Tuberculosis and topics like Gene with PCAA. His work carried out in the field of DNA ligase brings together such families of science as Homology directed repair, Circular bacterial chromosome and Cell biology.
His primary areas of investigation include Mycobacterium tuberculosis, Tuberculosis, Biochemistry, Microbiology and Cell biology. His Mycobacterium tuberculosis research includes themes of Genetics, Virology, Immunology, Cell envelope and Virulence. His study focuses on the intersection of Tuberculosis and fields such as Antibiotics with connections in the field of Isoniazid.
His Biochemistry study integrates concerns from other disciplines, such as Mycobacterium smegmatis and Mycolic acid. His Microbiology study combines topics in areas such as Transcription factor, Cord factor and Trehalose dimycolate. His work in the fields of Cell biology, such as Mediator and Effector, intersects with other areas such as Structure and function.
Tuberculosis, Mycobacterium tuberculosis, Cell biology, Cytoplasm and Microbiology are his primary areas of study. He works mostly in the field of Tuberculosis, limiting it down to topics relating to Immune system and, in certain cases, Infectious disease and Bladder cancer. As part of his studies on Mycobacterium tuberculosis, he often connects relevant subjects like Immunology.
His work on Effector and Phosphorylation as part of general Cell biology research is frequently linked to Repressor lexA and Cardiolipin, thereby connecting diverse disciplines of science. His Cytoplasm research includes elements of Cell wall and Mycolic acid. In his study, Transcription, Bacterial genetics and Rifamycin is inextricably linked to Polymerase, which falls within the broad field of Microbiology.
The scientist’s investigation covers issues in Biochemistry, Cancer, Cell, Cancer research and T cell. Many of his research projects under Biochemistry are closely connected to Chemical synthesis and Adduct with Chemical synthesis and Adduct, tying the diverse disciplines of science together. He combines subjects such as Mortality rate, Mechanical ventilation and Chemotherapy with his study of Cancer.
His research in Cancer research intersects with topics in T cell differentiation, T-cell receptor, TIGIT, Memory T cell and Effector. In his study, Mycobacterium tuberculosis is strongly linked to Innate immune system, which falls under the umbrella field of T cell. Michael S. Glickman studies Latent tuberculosis which is a part of Mycobacterium tuberculosis.
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A novel mycolic acid cyclopropane synthetase is required for cording, persistence, and virulence of Mycobacterium tuberculosis.
Michael S Glickman;Jeffery S Cox;William R Jacobs.
Molecular Cell (2000)
The mechanism of action of BCG therapy for bladder cancer—a current perspective
Gil Redelman-Sidi;Michael S. Glickman;Bernard H. Bochner.
Nature Reviews Urology (2014)
Determinants of COVID-19 disease severity in patients with cancer.
Elizabeth V. Robilotti;Elizabeth V. Robilotti;N. Esther Babady;Peter A. Mead;Peter A. Mead;Thierry Rolling.
Nature Medicine (2020)
Microbial Pathogenesis of Mycobacterium tuberculosis: Dawn of a Discipline
Michael S. Glickman;William R. Jacobs.
TOX is a critical regulator of tumour-specific T cell differentiation
Andrew C Scott;Andrew C Scott;Friederike Dündar;Paul Zumbo;Smita S Chandran.
Mycobacterium tuberculosis nuoG Is a Virulence Gene That Inhibits Apoptosis of Infected Host Cells
Kamalakannan Velmurugan;Bing Chen;Jessica L Miller;Sharon Azogue.
PLOS Pathogens (2007)
Antibody-catalyzed degradation of cocaine
Donald W. Landry;Kang Zhao;Ginger X. Q. Yang;Michael Glickman.
Mycobacterium tuberculosis controls host innate immune activation through cyclopropane modification of a glycolipid effector molecule
Vivek Rao;Nagatoshi Fujiwara;Steven A. Porcelli;Michael S. Glickman.
Journal of Experimental Medicine (2005)
Bacterial DNA repair by non-homologous end joining.
Stewart Shuman;Michael S Glickman.
Nature Reviews Microbiology (2007)
A Gamma Interferon Independent Mechanism of CD4 T Cell Mediated Control of M. tuberculosis Infection in vivo
Alena M. Gallegos;Alena M. Gallegos;Jeroen W. J. van Heijst;Miriam Samstein;Xiaodi Su.
PLOS Pathogens (2011)
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