Joel D. Ernst spends much of his time researching Mycobacterium tuberculosis, Immune system, Immunology, Tuberculosis and Virology. His Mycobacterium tuberculosis research includes elements of Pathogen, Microbiology and Receptor, Signal transduction, STAT1. His study focuses on the intersection of Microbiology and fields such as Macrophage with connections in the field of Intracellular parasite, Extracellular and Bacteria.
His research in Immune system is mostly focused on Interferon gamma. His Virology study combines topics in areas such as Antibody, Burden of disease and Environmental health. His work focuses on many connections between Acquired immune system and other disciplines, such as Lymph node, that overlap with his field of interest in CCL19 and Antigen.
His primary areas of investigation include Mycobacterium tuberculosis, Immunology, Tuberculosis, Immune system and Virology. The study incorporates disciplines such as Pathogen, Microbiology, Epitope, Macrophage and Antigen presentation in addition to Mycobacterium tuberculosis. His research is interdisciplinary, bridging the disciplines of Cytotoxic T cell and Immunology.
His work carried out in the field of Tuberculosis brings together such families of science as Cellular immunity, Mononuclear phagocyte system, Disease and Monocyte. His Immune system research incorporates elements of Peripheral blood mononuclear cell and Bone marrow. Joel D. Ernst has included themes like Antibody and Mycobacterium in his Virology study.
His main research concerns Mycobacterium tuberculosis, Immunology, Tuberculosis, Immune system and Virology. Joel D. Ernst works mostly in the field of Mycobacterium tuberculosis, limiting it down to topics relating to Secretion and, in certain cases, Mycobacterium africanum, Bacterial antigen, In vivo and Bacteria, as a part of the same area of interest. His Immunology study frequently draws connections to adjacent fields such as Rifapentine.
His Tuberculosis research includes themes of T cell, Bacilli, Immunity, Disease and Cytotoxic T cell. He works mostly in the field of Immune system, limiting it down to topics relating to Microbiology and, in certain cases, Leprosy. His Virology study integrates concerns from other disciplines, such as Glycolipid, Antibody and Antigen presentation.
Joel D. Ernst mainly investigates Immunology, Mycobacterium tuberculosis, Tuberculosis, Antibody and Virology. His Mononuclear phagocyte system, Serological assay and Serology study, which is part of a larger body of work in Immunology, is frequently linked to Appropriate use and Test performance, bridging the gap between disciplines. His research combines Acquired immune system and Mycobacterium tuberculosis.
His Tuberculosis study combines topics from a wide range of disciplines, such as Chronic infection, Immune system, Peripheral blood mononuclear cell, Monocyte and Bone marrow. The Autoantibody research he does as part of his general Antibody study is frequently linked to other disciplines of science, such as Zika virus, therefore creating a link between diverse domains of science. His studies deal with areas such as Cell biology, Effector, Antigen presentation and Endosome as well as Virology.
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Human T cell epitopes of Mycobacterium tuberculosis are evolutionarily hyperconserved
Iñaki Comas;Jaidip Chakravartti;Peter M Small;James Galagan.
Nature Genetics (2010)
HIV and Tuberculosis: a Deadly Human Syndemic
Candice K. Kwan;Joel D. Ernst.
Clinical Microbiology Reviews (2011)
Initiation of the adaptive immune response to Mycobacterium tuberculosis depends on antigen production in the local lymph node, not the lungs
Andrea J. Wolf;Ludovic Desvignes;Beth Linas;Niaz Banaiee.
Journal of Experimental Medicine (2008)
Macrophage receptors for Mycobacterium tuberculosis
Joel D. Ernst.
Infection and Immunity (1998)
Mycobacterium tuberculosis Infects Dendritic Cells with High Frequency and Impairs Their Function In Vivo
Andrea J. Wolf;Beth Linas;Giraldina J. Trevejo-Nuñez;Eleanor Kincaid;Eleanor Kincaid.
Journal of Immunology (2007)
The immunological life cycle of tuberculosis.
Joel D. Ernst.
Nature Reviews Immunology (2012)
Mycobacterium tuberculosis inhibits IFN-gamma transcriptional responses without inhibiting activation of STAT1.
Li Min Ting;Anne C. Kim;Ashok Cattamanchi;Joel D. Ernst.
Journal of Immunology (1999)
Chemokine receptor 2 serves an early and essential role in resistance to Mycobacterium tuberculosis
Wendy Peters;Holly M. Scott;Henry F. Chambers;JoAnne L. Flynn.
Proceedings of the National Academy of Sciences of the United States of America (2001)
Tuberculosis Pathogenesis and Immunity
Jennifer A. Philips;Joel D. Ernst.
Annual Review of Pathology-mechanisms of Disease (2012)
Modulation of Dengue Virus Infection in Human Cells by Alpha, Beta, and Gamma Interferons
Michael S. Diamond;Michael S. Diamond;T. Guy Roberts;Dianna Edgil;Betty Lu.
Journal of Virology (2000)
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