The scientist’s investigation covers issues in Phagocytosis, Microbiology, Cell biology, Macrophage and Phagosome. His Phagocytosis research is multidisciplinary, incorporating elements of Fimbria, Lipopolysaccharide, Oxidative phosphorylation, Fc receptor and Intracellular. His Intracellular research is multidisciplinary, incorporating perspectives in Calcium, Extracellular, Receptor, Myeloperoxidase and Luminol.
His Microbiology research incorporates themes from Yersinia pseudotuberculosis, Virulence, Inflammation, Mycobacterium tuberculosis and Innate immune system. His Cell biology study incorporates themes from Apoptosis, Biochemistry, Programmed cell death and Cytoskeleton. His studies in Phagosome integrate themes in fields like Rab and Actin.
Microbiology, Phagocytosis, Cell biology, Biochemistry and Immunology are his primary areas of study. His Microbiology research includes themes of Virology, Enterobacteriaceae, Escherichia coli, Mycobacterium tuberculosis and Macrophage. The concepts of his Phagocytosis study are interwoven with issues in Calcium and Fc receptor.
The Cell biology study combines topics in areas such as Apoptosis and Cytoskeleton. His Biochemistry research integrates issues from Molecular biology and Biophysics. His Immunology study combines topics in areas such as Asymptomatic, Nitric oxide and Tuberculosis.
Olle Stendahl focuses on Immunology, Tuberculosis, Mycobacterium tuberculosis, Microbiology and Macrophage. His work investigates the relationship between Immunology and topics such as Nitric oxide that intersect with problems in Drug resistance and Extensively drug-resistant tuberculosis. Olle Stendahl has researched Tuberculosis in several fields, including Helminths, Vitamin D and neurology, Internal medicine, Asymptomatic and Helminthiasis.
His Phagocytosis study in the realm of Microbiology connects with subjects such as Lipophosphoglycan. His Phagocytosis research entails a greater understanding of Cell biology. Olle Stendahl combines subjects such as Innate immune system, Caspase 1, Transition and Multiplicity of infection with his study of Macrophage.
Olle Stendahl spends much of his time researching Tuberculosis, Immunology, Macrophage, Microbiology and Mycobacterium tuberculosis. His biological study spans a wide range of topics, including Internal medicine, Asymptomatic, Nitric oxide and Sputum. Olle Stendahl interconnects Interleukin, Multiplicity of infection and Virology in the investigation of issues within Macrophage.
His study in Interleukin is interdisciplinary in nature, drawing from both Neutrophil elastase, Innate immune system, Gene and Neutrophil extracellular traps. His Multiplicity of infection research is multidisciplinary, relying on both Pyroptosis, Cathepsin D, Phagosome, Caspase 1 and Virulence. His Programmed cell death study integrates concerns from other disciplines, such as Necrosis, Inflammation, Cathepsin B and Tumor necrosis factor alpha.
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Intra- and extracellular events in luminol-dependent chemiluminescence of polymorphonuclear leukocytes.
G Briheim;O Stendahl;C Dahlgren.
Infection and Immunity (1984)
Mycobacterium tuberculosis Promotes Apoptosis in Human Neutrophils by Activating Caspase-3 and Altering Expression of Bax/Bcl-xL Via an Oxygen-Dependent Pathway
Nasrin Perskvist;Min Long;Olle Stendahl;Limin Zheng.
Journal of Immunology (2002)
Ca2+-dependent and Ca2+-independent phagocytosis in human neutrophils.
Daniel Pablo Lew;T. Andersson;J. Hed;F. Di Virgilio.
Cathepsin-cleaved Bid promotes apoptosis in human neutrophils via oxidative stress-induced lysosomal membrane permeabilization.
Robert Blomgran;Limin Zheng;Olle Stendahl.
Journal of Leukocyte Biology (2007)
Yersinia pseudotuberculosis inhibits Fc receptor-mediated phagocytosis in J774 cells.
M Fällman;K Andersson;S Håkansson;K E Magnusson.
Infection and Immunity (1995)
YopH of Yersinia pseudotuberculosis interrupts early phosphotyrosine signalling associated with phagocytosis.
Kerstin Andersson;Nivia Carballeira;Karl‐Eric Magnusson;Cathrine Persson.
Molecular Microbiology (1996)
Redistribution of intracellular Ca2+ stores during phagocytosis in human neutrophils
Olle Stendahl;Karl Heinz Krause;Joachim Krischer;Petra Jerström.
Pathogen-Induced Apoptotic Neutrophils Express Heat Shock Proteins and Elicit Activation of Human Macrophages
Limin Zheng;Min He;Min He;Min Long;Robert Blomgran.
Journal of Immunology (2004)
Human Macrophages Infected with a High Burden of ESAT-6-Expressing M. tuberculosis Undergo Caspase-1-and Cathepsin B-Independent Necrosis
Amanda Welin;Daniel Eklund;Olle Stendahl;Maria Lerm;Maria Lerm.
PLOS ONE (2011)
PHAGOSOME-LYSOSOME FUSION IS A CALCIUM-INDEPENDENT EVENT IN MACROPHAGES
S Zimmerli;M Majeed;M Gustavsson;O Stendahl.
Journal of Cell Biology (1996)
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