What is he best known for?
The fields of study he is best known for:
- Enzyme
- Immune system
- Gene
The scientist’s investigation covers issues in Phagocytosis, Microbiology, Cell biology, Macrophage and Phagosome.
His Phagocytosis research is multidisciplinary, incorporating elements of Fimbria, Lipopolysaccharide, Oxidative phosphorylation, Fc receptor and Intracellular.
His Intracellular research is multidisciplinary, incorporating perspectives in Calcium, Extracellular, Receptor, Myeloperoxidase and Luminol.
His Microbiology research incorporates themes from Yersinia pseudotuberculosis, Virulence, Inflammation, Mycobacterium tuberculosis and Innate immune system.
His Cell biology study incorporates themes from Apoptosis, Biochemistry, Programmed cell death and Cytoskeleton.
His studies in Phagosome integrate themes in fields like Rab and Actin.
His most cited work include:
- Intra- and extracellular events in luminol-dependent chemiluminescence of polymorphonuclear leukocytes. (192 citations)
- Ca2+-dependent and Ca2+-independent phagocytosis in human neutrophils. (178 citations)
- Mycobacterium tuberculosis Promotes Apoptosis in Human Neutrophils by Activating Caspase-3 and Altering Expression of Bax/Bcl-xL Via an Oxygen-Dependent Pathway (173 citations)
What are the main themes of his work throughout his whole career to date?
Microbiology, Phagocytosis, Cell biology, Biochemistry and Immunology are his primary areas of study.
His Microbiology research includes themes of Virology, Enterobacteriaceae, Escherichia coli, Mycobacterium tuberculosis and Macrophage.
The concepts of his Phagocytosis study are interwoven with issues in Calcium and Fc receptor.
The Cell biology study combines topics in areas such as Apoptosis and Cytoskeleton.
His Biochemistry research integrates issues from Molecular biology and Biophysics.
His Immunology study combines topics in areas such as Asymptomatic, Nitric oxide and Tuberculosis.
He most often published in these fields:
- Microbiology (35.00%)
- Phagocytosis (31.87%)
- Cell biology (27.50%)
What were the highlights of his more recent work (between 2010-2021)?
- Immunology (22.50%)
- Tuberculosis (14.38%)
- Mycobacterium tuberculosis (13.13%)
In recent papers he was focusing on the following fields of study:
Olle Stendahl focuses on Immunology, Tuberculosis, Mycobacterium tuberculosis, Microbiology and Macrophage.
His work investigates the relationship between Immunology and topics such as Nitric oxide that intersect with problems in Drug resistance and Extensively drug-resistant tuberculosis.
Olle Stendahl has researched Tuberculosis in several fields, including Helminths, Vitamin D and neurology, Internal medicine, Asymptomatic and Helminthiasis.
His Phagocytosis study in the realm of Microbiology connects with subjects such as Lipophosphoglycan.
His Phagocytosis research entails a greater understanding of Cell biology.
Olle Stendahl combines subjects such as Innate immune system, Caspase 1, Transition and Multiplicity of infection with his study of Macrophage.
Between 2010 and 2021, his most popular works were:
- Human Macrophages Infected with a High Burden of ESAT-6-Expressing M. tuberculosis Undergo Caspase-1-and Cathepsin B-Independent Necrosis (99 citations)
- Mycobacterium tuberculosis-Induced Neutrophil Extracellular Traps Activate Human Macrophages (74 citations)
- Importance of phagosomal functionality for growth restriction of Mycobacterium tuberculosis in primary human macrophages. (62 citations)
In his most recent research, the most cited papers focused on:
- Enzyme
- Gene
- Immune system
Olle Stendahl spends much of his time researching Tuberculosis, Immunology, Macrophage, Microbiology and Mycobacterium tuberculosis.
His biological study spans a wide range of topics, including Internal medicine, Asymptomatic, Nitric oxide and Sputum.
Olle Stendahl interconnects Interleukin, Multiplicity of infection and Virology in the investigation of issues within Macrophage.
His study in Interleukin is interdisciplinary in nature, drawing from both Neutrophil elastase, Innate immune system, Gene and Neutrophil extracellular traps.
His Multiplicity of infection research is multidisciplinary, relying on both Pyroptosis, Cathepsin D, Phagosome, Caspase 1 and Virulence.
His Programmed cell death study integrates concerns from other disciplines, such as Necrosis, Inflammation, Cathepsin B and Tumor necrosis factor alpha.
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