What is he best known for?
The fields of study he is best known for:
Michael R. Erdos mostly deals with Genetics, Genome-wide association study, Type 2 diabetes, Internal medicine and Endocrinology.
His work is connected to Lamin, LMNA, Progerin, Laminopathy and Progeria, as a part of Genetics.
His Genome-wide association study study is focused on Single-nucleotide polymorphism in general.
Michael R. Erdos has researched Single-nucleotide polymorphism in several fields, including Genotyping, Case-control study and Genetic linkage.
Diabetes mellitus covers Michael R. Erdos research in Type 2 diabetes.
The Genetic association study combines topics in areas such as CDKAL1, Obesity, FTO gene, Locus and SLC30A8.
His most cited work include:
- A genome-wide association study of type 2 diabetes in Finns detects multiple susceptibility variants. (2429 citations)
- A genome-wide association search for type 2 diabetes genes in African Americans. (1727 citations)
- New genetic loci implicated in fasting glucose homeostasis and their impact on type 2 diabetes risk (1707 citations)
What are the main themes of his work throughout his whole career to date?
Genetics, Type 2 diabetes, Progeria, Single-nucleotide polymorphism and Internal medicine are his primary areas of study.
His study in Genome-wide association study, Linkage disequilibrium, Genetic association, Gene and TCF7L2 falls within the category of Genetics.
In his study, Maturity onset diabetes of the young is strongly linked to HNF1A, which falls under the umbrella field of Type 2 diabetes.
His Progeria research incorporates elements of Mutation, Progerin, LMNA and Pathology.
His Single-nucleotide polymorphism research integrates issues from Genotyping, Polymorphism and Allele frequency.
His research in Internal medicine tackles topics such as Endocrinology which are related to areas like Allele.
He most often published in these fields:
- Genetics (57.14%)
- Type 2 diabetes (25.51%)
- Progeria (21.43%)
What were the highlights of his more recent work (between 2017-2021)?
- Progeria (21.43%)
- Genetics (57.14%)
- Pancreatic islets (8.16%)
In recent papers he was focusing on the following fields of study:
His primary areas of study are Progeria, Genetics, Pancreatic islets, Computational biology and Cancer research.
Michael R. Erdos interconnects Mutation, Progerin, LMNA and Pathology in the investigation of issues within Progeria.
His study in Genetics concentrates on DNA, Hutchinson Gilford Progeria Syndrome and Cell aging.
His study looks at the intersection of Cancer research and topics like Lamin with Muscular dystrophy and Everolimus.
His Islet research incorporates themes from Expression quantitative trait loci, Genome-wide association study, Transcriptional regulation and TCF7L2.
The study incorporates disciplines such as Regulation of gene expression, Regulatory sequence and Genetic association in addition to Genome-wide association study.
Between 2017 and 2021, his most popular works were:
- Everolimus rescues multiple cellular defects in laminopathy-patient fibroblasts. (22 citations)
- Multiomic Profiling Identifies cis-Regulatory Networks Underlying Human Pancreatic β Cell Identity and Function. (21 citations)
- In vivo base editing rescues Hutchinson-Gilford progeria syndrome in mice. (17 citations)
In his most recent research, the most cited papers focused on:
His primary areas of investigation include Progeria, Progerin, LMNA, Lamin and Mutation.
His work on Laminopathy as part of general Lamin study is frequently linked to Werner syndrome, bridging the gap between disciplines.
The concepts of his Mutation study are interwoven with issues in RNA, RNA editing, RNA splicing, Molecular biology and Genetically modified mouse.
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