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Luísa V. Lopes

Luísa V. Lopes

D-Index & Metrics

Neuroscience

D-Index
37
Citations
5456
World Ranking
8808
National Ranking
19

Overview

Luísa V. Lopes is affiliated with the University of Lisbon in Portugal and has contributed extensively to the fields of Neuroscience and Medicine. Their research spans multiple subfields including Physiology, Neurology, Cellular and Molecular Neuroscience, Cognitive Neuroscience, and Molecular Biology.

The scientist's work covers a range of topics within neuroscience such as:

  • Alzheimer's disease research and treatments
  • Neuroinflammation and Neurodegeneration Mechanisms
  • Neuroscience and Neuropharmacology Research
  • Memory and Neural Mechanisms
  • Parkinson's Disease Mechanisms and Treatments
  • Adenosine and Purinergic Signaling
  • Mitochondrial Function and Pathology

Some of the recent papers by Luísa V. Lopes include:

  • "IL-17 triggers the onset of cognitive and synaptic deficits in early stages of Alzheimer's disease," 2021, Cell Reports
  • "Caffeine intake exerts dual genome-wide effects on hippocampal metabolism and learning-dependent transcription," 2022, Journal of Clinical Investigation
  • "Glycation modulates glutamatergic signaling and exacerbates Parkinson's disease-like phenotypes," 2022, npj Parkinson s Disease
  • "Adenosine Receptors as Neuroinflammation Modulators: Role of A1 Agonists and A2A Antagonists," 2020, Cells
  • "Validation of the Portuguese Variant of the Munich Chronotype Questionnaire (MCTQPT)," 2020, Frontiers in Physiology

Frequent co-authors who have collaborated with Luísa V. Lopes include:

  • Joana E. Coelho
  • Paula A. Pousinha
  • David Blum
  • Rui Gomes
  • Kévin Carvalho

The scientist often publishes in venues such as:

  • bioRxiv (Cold Spring Harbor Laboratory)
  • Brain
  • Advanced Biosystems
  • Cell Reports
  • Journal of Clinical Investigation

Best Publications

  • Extracellular Alpha-Synuclein Oligomers Modulate Synaptic Transmission and Impair LTP Via NMDA-Receptor Activation

    Maria José Diógenes;Raquel B. Dias;Diogo M. Rombo;Hugo Vicente Miranda

  • α-synuclein interacts with PrP C to induce cognitive impairment through mGluR5 and NMDAR2B

    Diana G Ferreira;Mariana Temido-Ferreira;Hugo Vicente Miranda;Hugo Vicente Miranda;Vânia L Batalha

  • Meningeal γδ T cell–derived IL-17 controls synaptic plasticity and short-term memory

    Miguel Ribeiro;Helena C. Brigas;Mariana Temido-Ferreira;Paula A. Pousinha

  • Adenosine A2A receptors and brain injury: Broad spectrum of neuroprotection, multifaceted actions and “fine tuning” modulation

    Jiang Fan Chen;Patricia K. Sonsalla;Felicita Pedata;Alessia Melani

  • Adenosine A2A receptor facilitation of hippocampal synaptic transmission is dependent on tonic A1 receptor inhibition

    L.V. Lopes;R.A. Cunha;B. Kull;B.B. Fredholm

  • Glycation potentiates α-synuclein-associated neurodegeneration in synucleinopathies.

    Hugo Vicente Miranda;Hugo Vicente Miranda;Éva M. Szegö;Luís M. A. Oliveira;Luís M. A. Oliveira;Carlo Breda

  • Cross Talk Between A1 and A2A Adenosine Receptors in the Hippocampus and Cortex of Young Adult and Old Rats

    Luísa V. Lopes;Rodrigo A. Cunha;J. A. Ribeiro

  • IL-17 triggers the onset of cognitive and synaptic deficits in early stages of Alzheimer’s disease

    Helena C. Brigas;Miguel Ribeiro;Joana E. Coelho;Rui Gomes;Rui Gomes

  • A2A adenosine receptor deletion is protective in a mouse model of Tauopathy.

    C Laurent;C Laurent;S Burnouf;S Burnouf;B Ferry;V L Batalha

  • Adenosine A1 and A2A receptors are co-expressed in pyramidal neurons and co-localized in glutamatergic nerve terminals of the rat hippocampus.

    N. Rebola;R.J. Rodrigues;L.V. Lopes;P.J. Richardson

  • Age-related shift in LTD is dependent on neuronal adenosine A2A receptors interplay with mGluR5 and NMDA receptors

    Mariana Temido-Ferreira;Diana G. Ferreira;Vânia L. Batalha;Inês Marques-Morgado

  • Adenosine A(2A) receptor blockade reverts hippocampal stress-induced deficits and restores corticosterone circadian oscillation

    V. L. Batalha;José M. Pêgo;B. M. Fontinha;A. R. Costenla

  • Enhancement of LTP in aged rats is dependent on endogenous BDNF

    Maria José Diógenes;Ana Rita Costenla;Luísa Vaqueiro Lopes;André Jerónimo-Santos

  • Decrease of adenosine A1 receptor density and of adenosine neuromodulation in the hippocampus of kindled rats.

    Nelson Rebola;Joana E. Coelho;Ana Rita Costenla;Luísa V. Lopes

  • Overexpression of Adenosine A2A Receptors in Rats: Effects on Depression, Locomotion, and Anxiety.

    Joana E. Coelho;Pedro Alves;Paula M. Canas;Jorge S. Valadas

  • Adenosine and related drugs in brain diseases: present and future in clinical trials.

    Luisa V. Lopes;Ana M. Sebastiao;Joaquim A. Ribeiro

  • Increase in the number, G protein coupling, and efficiency of facilitatory adenosine A2A receptors in the limbic cortex, but not striatum, of aged rats.

    Luísa V. Lopes;Rodrigo A. Cunha;J. A. Ribeiro

  • Long-term Effect of Convulsive Behavior on the Density of Adenosine A1 and A2A Receptors in the Rat Cerebral Cortex

    Nelson Rebola;Lisiane O. Porciúncula;Luísa V. Lopes;Catarina R. Oliveira

  • Binding of the prototypical adenosine A2A receptor agonist CGS 21680 to the cerebral cortex of adenosine A1 and A2A receptor knockout mice

    Luísa V Lopes;Linda Halldner;Nelson Rebola;Björn Johansson

  • Adenosine A3 receptors are located in neurons of the rat hippocampus.

    Luisa V. Lopes;Nelson Rebola;Paulo C. Pinheiro;Peter J. Richardson

  • IL-17 Triggers the Onset of Cognitive and Synaptic Deficits In Early Stages Of Alzheimer's Disease

    Helena Brigas;Miguel Ribeiro;Joana Coelho;Rui Gomes

Frequent Co-Authors

David Blum
David Blum University of Lille
Ana M. Sebastião
Ana M. Sebastião University of Lisbon
Alexandre de Mendonça
Alexandre de Mendonça University of Lisbon
Detlev Boison
Detlev Boison Rutgers, The State University of New Jersey
Brigitte Städler
Brigitte Städler Aarhus University
Olaf Riess
Olaf Riess University of Tübingen
Michael T. Heneka
Michael T. Heneka University Hospital Bonn
Michele Migliore
Michele Migliore National Academies of Sciences, Engineering, and Medicine

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