2023 - Research.com Genetics in Denmark Leader Award
2022 - Research.com Genetics and Molecular Biology in Denmark Leader Award
The scientist’s investigation covers issues in Toxin-antitoxin system, Genetics, vapBC, Microbiology and Gene. His work carried out in the field of Toxin-antitoxin system brings together such families of science as Cell biology, Transcription and Protein biosynthesis. His vapBC study combines topics from a wide range of disciplines, such as RNA and PIN domain.
As a part of the same scientific family, he mostly works in the field of RNA, focusing on Messenger RNA and, on occasion, VAPB. The concepts of his Microbiology study are interwoven with issues in Model organism, Multidrug tolerance, Phenotype, Bacteria and Operon. His biological study spans a wide range of topics, including Cell division, Locus, ParABS system and DNA replication.
His primary areas of study are Plasmid, Genetics, Escherichia coli, Cell biology and Molecular biology. His Plasmid study incorporates themes from Cell division and Nucleoid. His Escherichia coli research is multidisciplinary, relying on both Toxin, Antitoxin and Microbiology.
His research in the fields of Toxin-antitoxin system overlaps with other disciplines such as RELB. His Cell biology study integrates concerns from other disciplines, such as Amino acid, Protein biosynthesis, FtsZ, Cytoskeleton and Ribosome. His Molecular biology research focuses on subjects like RNA, which are linked to Messenger RNA.
His primary scientific interests are in Escherichia coli, Cell biology, Antitoxin, Multidrug tolerance and Stringent response. Escherichia coli is a primary field of his research addressed under Genetics. Kenn Gerdes has researched Cell biology in several fields, including Amino acid and Biosynthesis.
His Antitoxin study combines topics in areas such as Plasmid, Promoter, Operon and Molecular biology. His Toxin-antitoxin system study, which is part of a larger body of work in Plasmid, is frequently linked to Evolutionary dynamics, bridging the gap between disciplines. As part of the same scientific family, Kenn Gerdes usually focuses on Multidrug tolerance, concentrating on Translation and intersecting with Proteome, DNA replication and Metabolism.
Kenn Gerdes mainly investigates Escherichia coli, Antitoxin, Cell biology, Toxin and Multidrug tolerance. His Escherichia coli study focuses on Biochemistry and Gene. His studies in Antitoxin integrate themes in fields like Transcription, Operon, DNA replication and Enzyme.
His Cell biology research integrates issues from SeqA protein domain, Phage shock and Ribosomal protein. His research integrates issues of Derepression, Psychological repression, Molecular biology, Promoter and NAD+ kinase in his study of Toxin. His Multidrug tolerance study is concerned with Genetics in general.
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Prokaryotic toxin-antitoxin stress response loci.
Kenn Gerdes;Susanne K. Christensen;Anders Løbner-Olesen.
Nature Reviews Microbiology (2005)
Toxin–antitoxin loci are highly abundant in free-living but lost from host-associated prokaryotes
Deo Prakash Pandey;Kenn Gerdes.
Nucleic Acids Research (2005)
Unique type of plasmid maintenance function: postsegregational killing of plasmid-free cells.
Kenn Gerdes;Poul B. Rasmussen;Soren Molin.
Proceedings of the National Academy of Sciences of the United States of America (1986)
Recent functional insights into the role of (p)ppGpp in bacterial physiology
Vasili Hauryliuk;Gemma C. Atkinson;Katsuhiko S. Murakami;Tanel Tenson.
Nature Reviews Microbiology (2015)
Bacterial persistence by RNA endonucleases
Etienne Maisonneuve;Lana J. Shakespeare;Mikkel Girke Jørgensen;Kenn Gerdes.
Proceedings of the National Academy of Sciences of the United States of America (2011)
Molecular Mechanisms Underlying Bacterial Persisters
Etienne Maisonneuve;Kenn Gerdes;Kenn Gerdes.
Cell (2014)
(p)ppGpp Controls Bacterial Persistence by Stochastic Induction of Toxin-Antitoxin Activity
Etienne Maisonneuve;Manuela Castro-Camargo;Kenn Gerdes.
Cell (2013)
Mechanisms of bacterial persistence during stress and antibiotic exposure.
Alexander Harms;Etienne Maisonneuve;Kenn Gerdes.
Science (2016)
The bacterial toxin RelE displays codon-specific cleavage of mRNAs in the ribosomal A site.
Kim Pedersen;Andrey V. Zavialov;Michael Yu. Pavlov;Johan Elf.
Cell (2003)
RelE, a global inhibitor of translation, is activated during nutritional stress
Susanne K. Christensen;Marie Mikkelsen;Kim Pedersen;Kenn Gerdes.
Proceedings of the National Academy of Sciences of the United States of America (2001)
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