The scientist’s investigation covers issues in Internal medicine, Mutation, Genetics, Immunology and Missense mutation. Johannes Oldenburg has researched Internal medicine in several fields, including Gastroenterology, Endocrinology and Surgery. Johannes Oldenburg regularly ties together related areas like Molecular biology in his Genetics studies.
He usually deals with Immunology and limits it to topics linked to Allele and Single-nucleotide polymorphism. His research on Missense mutation also deals with topics like
His primary areas of investigation include Internal medicine, Genetics, Haemophilia, Immunology and Haemophilia A. His Internal medicine research includes elements of Gastroenterology, Endocrinology and Surgery. His studies link Molecular biology with Genetics.
In the field of Haemophilia, his study on Haemophilia B overlaps with subjects such as In patient. Johannes Oldenburg is interested in Antibody, which is a field of Immunology. His Missense mutation study combines topics in areas such as Gene mutation, Compound heterozygosity and Genotype.
Johannes Oldenburg mainly investigates Internal medicine, Haemophilia A, Haemophilia, Pediatrics and Emicizumab. His Internal medicine study frequently draws connections between related disciplines such as Gastroenterology. The Haemophilia A study combines topics in areas such as Observational study, Quality of life and Intensive care medicine.
His Haemophilia study combines topics from a wide range of disciplines, such as Young adult, Regimen and Thrombotic microangiopathy. His Pediatrics research is multidisciplinary, relying on both Health related quality of life and MEDLINE. The study incorporates disciplines such as Mutation, Missense mutation and Cohort in addition to Von Willebrand disease.
Internal medicine, Haemophilia, Pediatrics, In patient and Haemophilia A are his primary areas of study. Johannes Oldenburg combines subjects such as Gastroenterology and Genotype with his study of Internal medicine. Within one scientific family, he focuses on topics pertaining to Young adult under Haemophilia, and may sometimes address concerns connected to Severity of illness.
As part of the same scientific family, Johannes Oldenburg usually focuses on Haemophilia A, concentrating on Quality of life and intersecting with Dosing, Factor IX and Haemophilia B. His Endogeny study also includes
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Mutations in VKORC1 cause warfarin resistance and multiple coagulation factor deficiency type 2
Simone Rost;Andreas Fregin;Vytautas Ivaskevicius;Ernst Conzelmann.
Emicizumab Prophylaxis in Hemophilia A with Inhibitors
Johannes Oldenburg;Johnny N. Mahlangu;Benjamin Kim;Christophe Schmitt.
The New England Journal of Medicine (2017)
The Genetic Basis of Resistance to Anticoagulants in Rodents
Hans-Joachim Pelz;Simone Rost;Mirja Hünerberg;Andreas Fregin.
Haemophilia A: mutation type determines risk of inhibitor formation.
Rainer Schwaab;H.-H. Brackmann;C. Meyer;J. Seehafer.
Thrombosis and Haemostasis (1995)
VKORC1 haplotypes and their impact on the inter-individual and inter-ethnical variability of oral anticoagulation
Christof Geisen;Matthias Watzka;Katja Sittinger;Michael Steffens.
Thrombosis and Haemostasis (2005)
F8 gene mutation type and inhibitor development in patients with severe hemophilia A: systematic review and meta-analysis
Samantha C. Gouw;Samantha C. Gouw;H. Marijke van den Berg;Johannes Oldenburg;Jan Astermark.
Polymorphisms in the IL10 but not in the IL1beta and IL4 genes are associated with inhibitor development in patients with hemophilia A.
Jan Astermark;Johannes Oldenburg;Anna Pavlova;Erik Berntorp.
Immune Tolerance for the Treatment of Factor VIII Inhibitors ‐ Twenty Years' ‘Bonn Protocol’
H. H. Brackmann;J. Oldenburg;R. Schwaab.
Vox Sanguinis (1996)
Genetic risk factors for inhibitors to factors VIII and IX.
J. Oldenburg;A. Pavlova.
Gender specific differences in levels of DNA methylation at selected loci from human total blood: a tendency toward higher methylation levels in males.
Osman El-Maarri;Tim Becker;Judith Junen;Syed Saadi Manzoor.
Human Genetics (2007)
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