James H. Resau mostly deals with Cancer research, Molecular biology, Hepatocyte growth factor, Internal medicine and Loss of heterozygosity. His work carried out in the field of Cancer research brings together such families of science as Carcinogenesis, Signal transduction, Metastasis and Immunohistochemistry. His Molecular biology study frequently draws connections between related disciplines such as Growth factor.
The study incorporates disciplines such as Cell culture, Autocrine signalling, Cell growth, Receptor tyrosine kinase and Mesenchymal stem cell in addition to Hepatocyte growth factor. His research in Internal medicine intersects with topics in Endocrinology, Oncology, Rating scale and Neurology, Neuroscience. His Loss of heterozygosity research is multidisciplinary, relying on both LRP5, Knockout mouse, Locus and Adenocarcinoma.
His primary scientific interests are in Pathology, Molecular biology, Cancer research, Internal medicine and Cell biology. His biological study deals with issues like Organ culture, which deal with fields such as Anatomy, Tissue culture and Pancreas. The concepts of his Molecular biology study are interwoven with issues in Complementary DNA, Gene, Allele, Receptor and Tumor suppressor gene.
His Cancer research research integrates issues from Cell growth, Hepatocyte growth factor, Carcinogenesis, Signal transduction and Metastasis. In his research, Loss of heterozygosity is intimately related to Adenocarcinoma, which falls under the overarching field of Carcinogenesis. His work deals with themes such as Endocrinology and Oncology, which intersect with Internal medicine.
James H. Resau focuses on Cancer research, Internal medicine, Pathology, Molecular biology and Cancer. His Cancer research research is multidisciplinary, incorporating perspectives in Cell growth, Hepatocyte growth factor, PI3K/AKT/mTOR pathway, Signal transduction and Metastasis. His Internal medicine research includes elements of Endocrinology and Oncology.
His Pathology course of study focuses on CD34 and Pericyte, Kidney cancer, Blood vessel, Kidney metabolism and Cluster of differentiation. James H. Resau interconnects Mutation, Complementary DNA, Receptor, Endogeny and Gene product in the investigation of issues within Molecular biology. His Cancer study which covers Immunology that intersects with Typhoid fever.
James H. Resau mostly deals with Cancer research, Pathology, Immunohistochemistry, Internal medicine and Hepatocyte growth factor. His research integrates issues of Tumor response, Cell growth, Carcinogenesis, PI3K/AKT/mTOR pathway and Adenocarcinoma in his study of Cancer research. James H. Resau has included themes like Cell cycle and Signal transduction in his Cell growth study.
His research in Carcinogenesis focuses on subjects like Mutation, which are connected to Molecular biology. His studies deal with areas such as Cancer, Prostate and Metastasis as well as Pathology. His biological study spans a wide range of topics, including Neurology, Neuroscience and Oncology.
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The multidrug-resistant phenotype associated with overexpression of the new ABC half-transporter, MXR (ABCG2)
Thomas Litman;Mariafiorella Brangi;Eric Hudson;Patricia Fetsch.
Journal of Cell Science (2000)
Invasiveness and metastasis of NIH 3T3 cells induced by Met-hepatocyte growth factor/scatter factor autocrine stimulation
Sing Rong;S. Segal;M. Anver;J. H. Resau.
Proceedings of the National Academy of Sciences of the United States of America (1994)
Activation of Human T Lymphocytes Is Inhibited by Peroxisome Proliferator-activated Receptor γ (PPARγ) Agonists PPARγ CO-ASSOCIATION WITH TRANSCRIPTION FACTOR NFAT
Xiao Yi Yang;Li Hua Wang;Taosheng Chen;David R. Hodge.
Journal of Biological Chemistry (2000)
The adaptor protein Tks5/Fish is required for podosome formation and function, and for the protease-driven invasion of cancer cells.
Darren F. Seals;Eduardo F. Azucena;Ian Pass;Lia Tesfay.
Cancer Cell (2005)
Met and Hepatocyte Growth Factor/Scatter Factor Expression in Human Gliomas
S Koochekpour;M Jeffers;S Rulong;G Taylor.
Cancer Research (1997)
Decreased BMD and limb deformities in mice carrying mutations in both Lrp5 and Lrp6.
Sheri L Holmen;Troy A Giambernardi;Cassandra R Zylstra;Bree D Buckner-Berghuis.
Journal of Bone and Mineral Research (2004)
Differential expression and responsiveness of chemokine receptors (CXCR1–3) by human microvascular endothelial cells and umbilical vein endothelial cells
Rosalba Salcedo;James H. Resau;Douglas Halverson;Eric A. Hudson.
The FASEB Journal (2000)
The parafibromin tumor suppressor protein is part of a human Paf1 complex
Orit Rozenblatt-Rosen;Christina M. Hughes;Suraj J. Nannepaga;Kalai Selvi Shanmugam.
Molecular and Cellular Biology (2005)
Helicobacter pylori urease activity is toxic to human gastric epithelial cells
D. T. Smoot;H. L. T. Mobley;G. R. Chippendale;J. F. Lewison.
Infection and Immunity (1990)
C‐Met overexpression in node‐positive breast cancer identifies patients with poor clinical outcome independent of Her2/neu
Ernst Lengyel;Dieter Prechtel;James H. Resau;Katja Gauger.
International Journal of Cancer (2005)
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