D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 65 Citations 15,166 199 World Ranking 5879 National Ranking 2792

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Cancer
  • Enzyme

James H. Resau mostly deals with Cancer research, Molecular biology, Hepatocyte growth factor, Internal medicine and Loss of heterozygosity. His work carried out in the field of Cancer research brings together such families of science as Carcinogenesis, Signal transduction, Metastasis and Immunohistochemistry. His Molecular biology study frequently draws connections between related disciplines such as Growth factor.

The study incorporates disciplines such as Cell culture, Autocrine signalling, Cell growth, Receptor tyrosine kinase and Mesenchymal stem cell in addition to Hepatocyte growth factor. His research in Internal medicine intersects with topics in Endocrinology, Oncology, Rating scale and Neurology, Neuroscience. His Loss of heterozygosity research is multidisciplinary, relying on both LRP5, Knockout mouse, Locus and Adenocarcinoma.

His most cited work include:

  • The multidrug-resistant phenotype associated with overexpression of the new ABC half-transporter, MXR (ABCG2) (460 citations)
  • Activation of Human T Lymphocytes Is Inhibited by Peroxisome Proliferator-activated Receptor γ (PPARγ) Agonists PPARγ CO-ASSOCIATION WITH TRANSCRIPTION FACTOR NFAT (345 citations)
  • Invasiveness and metastasis of NIH 3T3 cells induced by Met-hepatocyte growth factor/scatter factor autocrine stimulation (332 citations)

What are the main themes of his work throughout his whole career to date?

His primary scientific interests are in Pathology, Molecular biology, Cancer research, Internal medicine and Cell biology. His biological study deals with issues like Organ culture, which deal with fields such as Anatomy, Tissue culture and Pancreas. The concepts of his Molecular biology study are interwoven with issues in Complementary DNA, Gene, Allele, Receptor and Tumor suppressor gene.

His Cancer research research integrates issues from Cell growth, Hepatocyte growth factor, Carcinogenesis, Signal transduction and Metastasis. In his research, Loss of heterozygosity is intimately related to Adenocarcinoma, which falls under the overarching field of Carcinogenesis. His work deals with themes such as Endocrinology and Oncology, which intersect with Internal medicine.

He most often published in these fields:

  • Pathology (24.35%)
  • Molecular biology (23.83%)
  • Cancer research (21.24%)

What were the highlights of his more recent work (between 2006-2020)?

  • Cancer research (21.24%)
  • Internal medicine (18.13%)
  • Pathology (24.35%)

In recent papers he was focusing on the following fields of study:

James H. Resau focuses on Cancer research, Internal medicine, Pathology, Molecular biology and Cancer. His Cancer research research is multidisciplinary, incorporating perspectives in Cell growth, Hepatocyte growth factor, PI3K/AKT/mTOR pathway, Signal transduction and Metastasis. His Internal medicine research includes elements of Endocrinology and Oncology.

His Pathology course of study focuses on CD34 and Pericyte, Kidney cancer, Blood vessel, Kidney metabolism and Cluster of differentiation. James H. Resau interconnects Mutation, Complementary DNA, Receptor, Endogeny and Gene product in the investigation of issues within Molecular biology. His Cancer study which covers Immunology that intersects with Typhoid fever.

Between 2006 and 2020, his most popular works were:

  • Inhibition of MAPK kinase signaling pathways suppressed renal cell carcinoma growth and angiogenesis in vivo. (162 citations)
  • Plasma-based circulating MicroRNA biomarkers for Parkinson's disease. (136 citations)
  • Evidence that MIG-6 is a tumor-suppressor gene (102 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Cancer
  • Enzyme

James H. Resau mostly deals with Cancer research, Pathology, Immunohistochemistry, Internal medicine and Hepatocyte growth factor. His research integrates issues of Tumor response, Cell growth, Carcinogenesis, PI3K/AKT/mTOR pathway and Adenocarcinoma in his study of Cancer research. James H. Resau has included themes like Cell cycle and Signal transduction in his Cell growth study.

His research in Carcinogenesis focuses on subjects like Mutation, which are connected to Molecular biology. His studies deal with areas such as Cancer, Prostate and Metastasis as well as Pathology. His biological study spans a wide range of topics, including Neurology, Neuroscience and Oncology.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

The multidrug-resistant phenotype associated with overexpression of the new ABC half-transporter, MXR (ABCG2)

Thomas Litman;Mariafiorella Brangi;Eric Hudson;Patricia Fetsch.
Journal of Cell Science (2000)

698 Citations

Invasiveness and metastasis of NIH 3T3 cells induced by Met-hepatocyte growth factor/scatter factor autocrine stimulation

Sing Rong;S. Segal;M. Anver;J. H. Resau.
Proceedings of the National Academy of Sciences of the United States of America (1994)

504 Citations

Activation of Human T Lymphocytes Is Inhibited by Peroxisome Proliferator-activated Receptor γ (PPARγ) Agonists PPARγ CO-ASSOCIATION WITH TRANSCRIPTION FACTOR NFAT

Xiao Yi Yang;Li Hua Wang;Taosheng Chen;David R. Hodge.
Journal of Biological Chemistry (2000)

490 Citations

The adaptor protein Tks5/Fish is required for podosome formation and function, and for the protease-driven invasion of cancer cells.

Darren F. Seals;Eduardo F. Azucena;Ian Pass;Lia Tesfay.
Cancer Cell (2005)

459 Citations

Met and Hepatocyte Growth Factor/Scatter Factor Expression in Human Gliomas

S Koochekpour;M Jeffers;S Rulong;G Taylor.
Cancer Research (1997)

424 Citations

Decreased BMD and limb deformities in mice carrying mutations in both Lrp5 and Lrp6.

Sheri L Holmen;Troy A Giambernardi;Cassandra R Zylstra;Bree D Buckner-Berghuis.
Journal of Bone and Mineral Research (2004)

406 Citations

Differential expression and responsiveness of chemokine receptors (CXCR1–3) by human microvascular endothelial cells and umbilical vein endothelial cells

Rosalba Salcedo;James H. Resau;Douglas Halverson;Eric A. Hudson.
The FASEB Journal (2000)

351 Citations

The parafibromin tumor suppressor protein is part of a human Paf1 complex

Orit Rozenblatt-Rosen;Christina M. Hughes;Suraj J. Nannepaga;Kalai Selvi Shanmugam.
Molecular and Cellular Biology (2005)

346 Citations

Helicobacter pylori urease activity is toxic to human gastric epithelial cells

D. T. Smoot;H. L. T. Mobley;G. R. Chippendale;J. F. Lewison.
Infection and Immunity (1990)

317 Citations

C‐Met overexpression in node‐positive breast cancer identifies patients with poor clinical outcome independent of Her2/neu

Ernst Lengyel;Dieter Prechtel;James H. Resau;Katja Gauger.
International Journal of Cancer (2005)

297 Citations

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