Hong Jiang mainly focuses on Internal medicine, Endocrinology, Alzheimer's disease, Biochar and Apolipoprotein E. Internal medicine and Genetically modified mouse are commonly linked in his work. As part of the same scientific family, Hong Jiang usually focuses on Alzheimer's disease, concentrating on Neurodegeneration and intersecting with Intracellular, Neuron and Neuroinflammation.
His Biochar research includes elements of Biomass, Carbon and Adsorption. His study in Apolipoprotein E is interdisciplinary in nature, drawing from both Gene isoform, Immunology, Lipoprotein and Amyloid. His study looks at the intersection of Immunology and topics like Transgene with In vivo.
Hong Jiang spends much of his time researching Pyrolysis, Internal medicine, Biomass, Endocrinology and Biochar. His Pyrolysis research focuses on Catalysis and how it relates to Inorganic chemistry. The concepts of his Endocrinology study are interwoven with issues in Receptor and Apolipoprotein E.
His studies in Apolipoprotein E integrate themes in fields like Alzheimer's disease, Immunology and Amyloid. Hong Jiang works mostly in the field of Alzheimer's disease, limiting it down to topics relating to Transgene and, in certain cases, In vivo, as a part of the same area of interest. Hong Jiang usually deals with Biochar and limits it to topics linked to Adsorption and Aqueous solution and X-ray photoelectron spectroscopy.
His scientific interests lie mostly in Pyrolysis, Biomass, Biochar, Catalysis and Kidney. His Pyrolysis study combines topics from a wide range of disciplines, such as Environmental chemistry and Anatase. His work deals with themes such as Scientific method, Fossil fuel, Doping and Sawdust, which intersect with Biomass.
His work carried out in the field of Biochar brings together such families of science as Hydrogen, Renewable resource and Adsorption. His biological study spans a wide range of topics, including Cobalt, Radical, Carbon and Nuclear chemistry. Endocrinology and Internal medicine are the focus of his Kidney studies.
His primary areas of study are Biochar, Biomass, Catalysis, Pyrolysis and Fossil fuel. His Biochar study incorporates themes from Redox, Nanotechnology, Adsorption and Metal. In general Adsorption, his work in Sorption is often linked to Bisphenol A linking many areas of study.
His Renewable resource research extends to Biomass, which is thematically connected. The study incorporates disciplines such as Hydrogen, Carbon and Pollutant in addition to Catalysis. His Pyrolysis study frequently draws connections between adjacent fields such as Scientific method.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Human apoE Isoforms Differentially Regulate Brain Amyloid-β Peptide Clearance
Joseph M. Castellano;Jungsu Kim;Floy R. Stewart;Hong Jiang.
Science Translational Medicine (2011)
Sirt5 is a NAD-dependent protein lysine demalonylase and desuccinylase.
Jintang Du;Yeyun Zhou;Xiaoyang Su;Jiu Jiu Yu.
Science (2011)
Development of Biochar-Based Functional Materials: Toward a Sustainable Platform Carbon Material
Wu-Jun Liu;Hong Jiang;Han-Qing Yu.
Chemical Reviews (2015)
P-glycoprotein deficiency at the blood-brain barrier increases amyloid-β deposition in an Alzheimer disease mouse model
John R. Cirrito;Rashid Deane;Anne M. Fagan;Michael L. Spinner.
Journal of Clinical Investigation (2005)
SIRT6 regulates TNF-α secretion through hydrolysis of long-chain fatty acyl lysine
Hong Jiang;Saba Khan;Yi Wang;Guillaume Charron.
Nature (2013)
Trans-cellular Propagation of Tau Aggregation by Fibrillar Species
Najla Kfoury;Brandon B. Holmes;Hong Jiang;David M. Holtzman.
Journal of Biological Chemistry (2012)
ApoE4 markedly exacerbates tau-mediated neurodegeneration in a mouse model of tauopathy
Yang Shi;Kaoru Yamada;Shane Antony Liddelow;Shane Antony Liddelow;Scott T. Smith.
Nature (2017)
Anti-Tau Antibodies that Block Tau Aggregate Seeding In Vitro Markedly Decrease Pathology and Improve Cognition In Vivo
Kiran Yanamandra;Najla Kfoury;Hong Jiang;Thomas E. Mahan.
Neuron (2013)
ApoE influences amyloid-β (Aβ) clearance despite minimal apoE/Aβ association in physiological conditions
Philip B. Verghese;Joseph M. Castellano;Kanchan Garai;Yinong Wang.
Proceedings of the National Academy of Sciences of the United States of America (2013)
ABCA1 is required for normal central nervous system ApoE levels and for lipidation of astrocyte-secreted apoE.
Suzanne E. Wahrle;Hong Jiang;Maia Parsadanian;Justin Legleiter.
Journal of Biological Chemistry (2004)
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