What is he best known for?
The fields of study he is best known for:
His primary scientific interests are in Postsynaptic density, Neuroscience, Cell biology, SHANK2 and Postsynaptic potential.
His work deals with themes such as Dendritic spine, Synapse, Cytoskeleton and Synaptogenesis, which intersect with Postsynaptic density.
His research in Neuroscience intersects with topics in Synaptic plasticity, Immunology, Neurotransmission and Presynaptic active zone.
His Cell biology research is multidisciplinary, incorporating perspectives in Calcium-activated potassium channel, Active zone, Synaptic vesicle and Actin cytoskeleton.
The various areas that Tobias M. Boeckers examines in his SHANK2 study include Biophysics, Scaffold protein, PDZ domain, Autism and Cortactin.
His study in Autism is interdisciplinary in nature, drawing from both Genetics and 22q13 deletion syndrome.
His most cited work include:
- Mutations in the gene encoding the synaptic scaffolding protein SHANK3 are associated with autism spectrum disorders. (1126 citations)
- Haploinsufficiency of TBK1 causes familial ALS and fronto-temporal dementia (448 citations)
- Autistic-like behaviours and hyperactivity in mice lacking ProSAP1/Shank2 (408 citations)
What are the main themes of his work throughout his whole career to date?
The scientist’s investigation covers issues in Cell biology, Neuroscience, Postsynaptic density, Postsynaptic potential and SHANK2.
His Cell biology study integrates concerns from other disciplines, such as Glutamatergic, Actin cytoskeleton, Cytoskeleton and Induced pluripotent stem cell.
The concepts of his Neuroscience study are interwoven with issues in Phenotype, Amyotrophic lateral sclerosis, Autism and Neurotransmission.
Tobias M. Boeckers has included themes like Dendritic spine, Synapse and Scaffold protein in his Postsynaptic density study.
His Postsynaptic potential research includes themes of Active zone, Biophysics and Synaptogenesis.
He combines subjects such as NMDA receptor and Long-term potentiation with his study of Synaptic plasticity.
He most often published in these fields:
- Cell biology (44.27%)
- Neuroscience (37.50%)
- Postsynaptic density (31.25%)
What were the highlights of his more recent work (between 2018-2021)?
- Neuroscience (37.50%)
- Cell biology (44.27%)
- Amyotrophic lateral sclerosis (10.42%)
In recent papers he was focusing on the following fields of study:
Tobias M. Boeckers spends much of his time researching Neuroscience, Cell biology, Amyotrophic lateral sclerosis, Autism and Postsynaptic potential.
His work in Forebrain, Postsynaptic density, Dendritic spine, Hippocampal formation and Central nervous system are all subfields of Neuroscience research.
His Postsynaptic density study necessitates a more in-depth grasp of Neurotransmission.
His Cell biology research integrates issues from Glutamate receptor, TANK-binding kinase 1 and Voltage-dependent calcium channel.
His Autism study combines topics from a wide range of disciplines, such as Synaptic plasticity, Phenotype, Haploinsufficiency, Knockout mouse and SHANK2.
His biological study spans a wide range of topics, including Synapse, Myogenesis, Skeletal muscle and Acetylcholine receptor.
Between 2018 and 2021, his most popular works were:
- FUS-mediated regulation of acetylcholine receptor transcription at neuromuscular junctions is compromised in amyotrophic lateral sclerosis (24 citations)
- Early Correction of N-Methyl-D-Aspartate Receptor Function Improves Autistic-like Social Behaviors in Adult Shank2-/- Mice. (22 citations)
- Altered calcium dynamics and glutamate receptor properties in iPSC-derived motor neurons from ALS patients with C9orf72, FUS, SOD1 or TDP43 mutations (13 citations)
In his most recent research, the most cited papers focused on:
Tobias M. Boeckers spends much of his time researching Cell biology, Amyotrophic lateral sclerosis, Postsynaptic potential, Autism and HOMER1.
His Cell biology research is mostly focused on the topic PI3K/AKT/mTOR pathway.
His studies deal with areas such as Myogenesis, Skeletal muscle, Motor neuron, Neuromuscular junction and Acetylcholine receptor as well as Amyotrophic lateral sclerosis.
His Postsynaptic potential study combines topics in areas such as Neuroscience and ETS transcription factor family.
His Autism research incorporates elements of Microbiome, Epithelium, Phenotype, Lipopolysaccharide and Ankyrin repeat.
The study incorporates disciplines such as Mutation, Synapse, Induced pluripotent stem cell and Synaptogenesis in addition to HOMER1.
This overview was generated by a machine learning system which analysed the scientist’s
body of work. If you have any feedback, you can
contact us
here.
