His scientific interests lie mostly in Genetics, Molecular biology, Mutation, DNA and Polymerase chain reaction. In the subject of general Genetics, his work in Phenotype and Epigenetics is often linked to Gene dosage, NLRP7 and Genomic imprinting, thereby combining diverse domains of study. Within one scientific family, he focuses on topics pertaining to Digital polymerase chain reaction under Molecular biology, and may sometimes address concerns connected to TaqMan, Polymerase chain reaction optimization and Variants of PCR.
Graham R. Taylor has included themes like Optic nerve hypoplasia, Histone acetyltransferase, Lissencephaly and Cell biology in his Mutation study. Graham R. Taylor works mostly in the field of DNA, limiting it down to topics relating to Gene and, in certain cases, Immunohistochemistry, Rectum, Cancer research and Wallerian degeneration, as a part of the same area of interest. His Polymerase chain reaction research incorporates themes from RNA, Helicobacter pylori and 16S ribosomal RNA.
The scientist’s investigation covers issues in Genetics, Molecular biology, Polymerase chain reaction, Gene and DNA sequencing. His Molecular biology study combines topics in areas such as Digital polymerase chain reaction and Nested polymerase chain reaction. His Polymerase chain reaction study frequently links to other fields, such as DNA.
He is involved in the study of Gene that focuses on Mutation in particular. His studies in Mutation integrate themes in fields like Genetic heterogeneity, Colorectal cancer and Cancer research. He works mostly in the field of DNA sequencing, limiting it down to concerns involving Computational biology and, occasionally, Reference genome.
Graham R. Taylor mostly deals with Genetics, DNA sequencing, Exome sequencing, Internal medicine and Computational biology. All of his Genetics and Mutation, Genetic heterogeneity, Sequence analysis, Gene and Mutation investigations are sub-components of the entire Genetics study. His Mutation research is multidisciplinary, incorporating perspectives in Epigenetics and Conceptus.
The DNA sequencing study combines topics in areas such as Genome, Copy-number variation, Virology and Human papillomavirus. His work in the fields of Exome overlaps with other areas such as NMNAT1. His Internal medicine study incorporates themes from Gene rearrangement, Surgery, Oncology and Endocrinology.
His primary scientific interests are in Genetics, Exome sequencing, DNA sequencing, Pathology and Clinical genetic. His Genetics research is multidisciplinary, incorporating elements of Molecular biology, Persistent fetal vasculature and Optic nerve. His studies in Exome sequencing integrate themes in fields like Amplicon, Respiratory distress and Mutation.
His DNA sequencing study combines topics in areas such as Virology, Allele, Allele frequency, Deep sequencing and Single cell sequencing. Many of his research projects under Pathology are closely connected to MEGF10 with MEGF10, tying the diverse disciplines of science together. His Clinical genetic course of study focuses on Genomics and Data science.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
KRAS and BRAF Mutations in Advanced Colorectal Cancer Are Associated With Poor Prognosis but Do Not Preclude Benefit From Oxaliplatin or Irinotecan: Results From the MRC FOCUS Trial
Susan D. Richman;Matthew T. Seymour;Philip Chambers;Faye Elliott.
Journal of Clinical Oncology (2009)
PCR: a practical approach.
M. J. McPherson;P. Quirke;G. R. Taylor.
MLPA and MAPH: New techniques for detection of gene deletions
Loryn N. Sellner;Graham R. Taylor.
Human Mutation (2004)
Tissue extraction of DNA and RNA and analysis by the polymerase chain reaction.
D P Jackson;F A Lewis;G R Taylor;A W Boylston.
Journal of Clinical Pathology (1990)
Direct polymerase chain reaction test for detection of Helicobacter pylori in humans and animals.
S A Ho;J A Hoyle;F A Lewis;A D Secker.
Journal of Clinical Microbiology (1991)
Prognostic value of p53 overexpression and c-Ki-ras gene mutations in colorectal cancer
Sandra M. Bell;Sandra M. Bell;Nigel Scott;Nigel Scott;Debra Cross;Debra Cross;Peter Sagar;Peter Sagar.
A COMMON FOUNDER FOR THE 35DELG GJB2 GENE MUTATION IN CONNEXIN 26 HEARING IMPAIRMENT
L Van Laer;P Coucke;R F Mueller;G Caethoven.
Journal of Medical Genetics (2001)
Rapid detection of allele loss in colorectal tumours using microsatellites and fluorescent DNA technology.
L Cawkwell;SM Bell;FA Lewis;MF Dixon.
British Journal of Cancer (1993)
Mutations Causing Familial Biparental Hydatidiform Mole Implicate C6orf221 as a Possible Regulator of Genomic Imprinting in the Human Oocyte
David A. Parry;Clare V. Logan;Bruce E. Hayward;Michael Shires.
American Journal of Human Genetics (2011)
Genomic Deletions in MSH2 or MLH1 Are a Frequent Cause of Hereditary Non-Polyposis Colorectal Cancer: Identification of Novel and Recurrent Deletions by MLPA
C.F. Taylor;R.S. Charlton;J. Burn;E. Sheridan.
Human Mutation (2003)
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