What is he best known for?
The fields of study he is best known for:
- Enzyme
- Biochemistry
- Metabolism
Gábor Bánhegyi mostly deals with Biochemistry, Endoplasmic reticulum, Microsome, Glutathione and Cell biology.
His Endoplasmic reticulum research is multidisciplinary, relying on both Oxidative phosphorylation, Endocrinology and Protein folding.
His Microsome study combines topics in areas such as Dehydrogenase, Oxoglutarate dehydrogenase complex, Branched-chain alpha-keto acid dehydrogenase complex, Vesicle and Cofactor.
Gábor Bánhegyi works mostly in the field of Glutathione, limiting it down to topics relating to Oxidative folding and, in certain cases, NAD+ kinase, as a part of the same area of interest.
He combines subjects such as Apoptosis, Membrane potential and Cytosol with his study of Cell biology.
The study incorporates disciplines such as MAP1LC3B, Chaperone-mediated autophagy, BECN1 and Autolysosome in addition to Programmed cell death.
His most cited work include:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- Guidelines for the use and interpretation of assays for monitoring autophagy (3242 citations)
- Vitamin C: update on physiology and pharmacology (282 citations)
What are the main themes of his work throughout his whole career to date?
The scientist’s investigation covers issues in Biochemistry, Endoplasmic reticulum, Microsome, Cell biology and Endocrinology.
His study in the fields of Unfolded protein response under the domain of Endoplasmic reticulum overlaps with other disciplines such as Redox.
His Microsome research integrates issues from Molecular biology, Glycogen storage disease, Glucuronide and Hydrogen peroxide.
His research in Cell biology intersects with topics in Autophagy, Apoptosis and Oxidative stress.
Gábor Bánhegyi interconnects Fructose and Internal medicine in the investigation of issues within Endocrinology.
His work in Oxidative phosphorylation addresses subjects such as Oxidative folding, which are connected to disciplines such as NAD+ kinase.
He most often published in these fields:
- Biochemistry (72.52%)
- Endoplasmic reticulum (75.68%)
- Microsome (46.40%)
What were the highlights of his more recent work (between 2013-2021)?
- Cell biology (40.09%)
- Endoplasmic reticulum (75.68%)
- Autophagy (15.77%)
In recent papers he was focusing on the following fields of study:
Cell biology, Endoplasmic reticulum, Autophagy, Unfolded protein response and Biochemistry are his primary areas of study.
As a part of the same scientific study, Gábor Bánhegyi usually deals with the Cell biology, concentrating on Cell growth and frequently concerns with Membrane potential and Mitochondrial biogenesis.
His Endoplasmic reticulum research incorporates elements of Glucose transporter, Organelle and Mevalonate pathway.
His work in Autophagy covers topics such as Oxidative stress which are related to areas like Programmed cell death, Oxidative phosphorylation and KEAP1.
Gábor Bánhegyi has included themes like Cancer cell and Physiology in his Programmed cell death study.
In general Biochemistry, his work in Microsome, Mitochondrion, Dehydroascorbic acid and Amyloid beta is often linked to Biochemistry of Alzheimer's disease linking many areas of study.
Between 2013 and 2021, his most popular works were:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- On the role of 4-hydroxynonenal in health and disease. (121 citations)
- mTOR inhibition increases cell viability via autophagy induction during endoplasmic reticulum stress - An experimental and modeling study. (59 citations)
In his most recent research, the most cited papers focused on:
- Enzyme
- Metabolism
- Biochemistry
His primary scientific interests are in Cell biology, Autophagy, Endoplasmic reticulum, AMPK and PI3K/AKT/mTOR pathway.
In the subject of general Cell biology, his work in Signal transduction and Phosphorylation is often linked to Interpretation, Arama and Humanities, thereby combining diverse domains of study.
His Autophagy research is multidisciplinary, incorporating elements of Oxidative stress, Proteostasis, Programmed cell death and Physiology.
His work deals with themes such as BECN1, Autophagosome, Autolysosome, Sequestosome 1 and Computational biology, which intersect with Programmed cell death.
His Endoplasmic reticulum study integrates concerns from other disciplines, such as Intracellular, Organelle and Dehydroascorbic acid.
In his study, which falls under the umbrella issue of Unfolded protein response, ULK1 is strongly linked to Viability assay.
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