D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 77 Citations 20,901 342 World Ranking 2993 National Ranking 1569

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Internal medicine
  • Enzyme

David S. Williams spends much of his time researching Cell biology, Genetics, Retina, Cilium and Mutant. He has researched Cell biology in several fields, including Retinal degeneration, Photoreceptor outer segment, Macular degeneration, MYO7A and Opsin. His work on Retinitis pigmentosa, T cell and Candidate Gene Analysis as part of his general Genetics study is frequently connected to Disease gene identification, thereby bridging the divide between different branches of science.

His biological study spans a wide range of topics, including Retinal, Anatomy and Transducin. David S. Williams has included themes like Positional cloning, Microtubule, RPGRIP1L, Senior–Løken syndrome and Myosin in his Cilium study. In his study, Gene knockdown and Respiratory enzyme is strongly linked to Molecular biology, which falls under the umbrella field of Mutant.

His most cited work include:

  • Axonopathy and transport deficits early in the pathogenesis of Alzheimer's disease. (921 citations)
  • The centrosomal protein nephrocystin-6 is mutated in Joubert syndrome and activates transcription factor ATF4 (460 citations)
  • Toxicity of Familial ALS-Linked SOD1 Mutants from Selective Recruitment to Spinal Mitochondria (424 citations)

What are the main themes of his work throughout his whole career to date?

David S. Williams mostly deals with Cell biology, Retina, Retinal, Internal medicine and Retinal pigment epithelium. His Cell biology research is multidisciplinary, incorporating perspectives in Retinal degeneration, Photoreceptor outer segment and Opsin. His Retinal degeneration study integrates concerns from other disciplines, such as Retinitis pigmentosa, MYO7A and Usher syndrome.

The study incorporates disciplines such as Mutant and Anatomy in addition to Retina. His Retinal study results in a more complete grasp of Biochemistry. The concepts of his Retinal pigment epithelium study are interwoven with issues in Phagosome and Melanosome.

He most often published in these fields:

  • Cell biology (44.61%)
  • Retina (15.79%)
  • Retinal (15.54%)

What were the highlights of his more recent work (between 2017-2021)?

  • Cell biology (44.61%)
  • Internal medicine (12.03%)
  • Nanotechnology (6.77%)

In recent papers he was focusing on the following fields of study:

David S. Williams mainly focuses on Cell biology, Internal medicine, Nanotechnology, Polymersome and Nanoparticle. David S. Williams works in the field of Cell biology, focusing on Myosin in particular. The various areas that David S. Williams examines in his Internal medicine study include Gastroenterology and Oncology.

His work in the fields of Drug delivery overlaps with other areas such as Biocompatible material. His Polymersome study incorporates themes from Vesicle, Membrane, Biophysics, Synthetic Organelles and Nanoreactor. His Nanoparticle research includes themes of Copolymer and Ethylene glycol.

Between 2017 and 2021, his most popular works were:

  • Erythrocyte membrane modified janus polymeric motors for thrombus therapy (59 citations)
  • The prognostic impact of consensus molecular subtypes (CMS) and its predictive effects for bevacizumab benefit in metastatic colorectal cancer: molecular analysis of the AGITG MAX clinical trial (57 citations)
  • Mimicking Cellular Compartmentalization in a Hierarchical Protocell through Spontaneous Spatial Organization. (37 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Enzyme

His primary areas of investigation include Polymersome, Cell biology, Biophysics, Internal medicine and Nanotechnology. His research integrates issues of Photoreceptor outer segment, Real-time polymerase chain reaction, Nicotinamide, Cytoskeletal Organization and Metabolism in his study of Cell biology. In his study, which falls under the umbrella issue of Photoreceptor outer segment, Mutant protein, Retina, Phagosome, Retinal degeneration and Rhodopsin is strongly linked to Motility.

His Biophysics research incorporates elements of Coacervate, Protocell and Cell membrane. His work in the fields of Internal medicine, such as Prospective cohort study and Colorectal cancer, overlaps with other areas such as Minimal change disease, Nephropathy and Membranous nephropathy. As a part of the same scientific study, David S. Williams usually deals with the Nanotechnology, concentrating on Function and frequently concerns with Ethylene glycol.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Axonopathy and transport deficits early in the pathogenesis of Alzheimer's disease.

Gorazd B. Stokin;Concepción Lillo;Tomás L. Falzone;Richard G. Brusch.
Science (2005)

1328 Citations

The centrosomal protein nephrocystin-6 is mutated in Joubert syndrome and activates transcription factor ATF4

John A. Sayer;John A. Sayer;Edgar A. Otto;John F. O'Toole;Gudrun Nurnberg.
Nature Genetics (2006)

709 Citations

Toxicity of Familial ALS-Linked SOD1 Mutants from Selective Recruitment to Spinal Mitochondria

Jian Liu;Concepción Lillo;P.Andreas Jonsson;Christine Vande Velde.
Neuron (2004)

611 Citations

Cadherin 23 is a component of the tip link in hair-cell stereocilia.

Jan Siemens;Concepcion Lillo;Rachel A. Dumont;Anna Reynolds.
Nature (2004)

506 Citations

Path Decomposition and Continuity of Local Time for One‐Dimensional Diffusions, I

David Williams.
Proceedings of The London Mathematical Society (1974)

464 Citations

Genetic evidence for selective transport of opsin and arrestin by kinesin-II in mammalian photoreceptors.

Joseph R Marszalek;Xinran Liu;Elizabeth A Roberts;Daniel Chui.
Cell (2000)

462 Citations

Nephrocystin-5, a ciliary IQ domain protein, is mutated in Senior-Loken syndrome and interacts with RPGR and calmodulin

Edgar A. Otto;Bart Loeys;Hemant Khanna;Jan Hellemans.
Nature Genetics (2005)

419 Citations

In-frame deletion in a novel centrosomal/ciliary protein CEP290/NPHP6 perturbs its interaction with RPGR and results in early-onset retinal degeneration in the rd16 mouse

Bo Chang;Hemant Khanna;Norman Hawes;David Jimeno.
Human Molecular Genetics (2006)

395 Citations

Candidate exome capture identifies mutation of SDCCAG8 as the cause of a retinal-renal ciliopathy

Edgar A Otto;Toby W Hurd;Rannar Airik;Moumita Chaki.
Nature Genetics (2010)

366 Citations

Serotype-dependent packaging of large genes in adeno-associated viral vectors results in effective gene delivery in mice

Mariacarmela Allocca;Monica Doria;Marco Petrillo;Pasqualina Colella.
Journal of Clinical Investigation (2008)

333 Citations

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