D-Index & Metrics Best Publications

D-Index & Metrics

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Microbiology D-index 40 Citations 6,155 93 World Ranking 3504 National Ranking 1386

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Bacteria
  • Enzyme

Carlos J. Orihuela mainly focuses on Microbiology, Streptococcus pneumoniae, Pneumolysin, Pneumococcal infections and Immunology. His research in Microbiology intersects with topics in Bacterial adhesin, Pathogenicity island, Virulence and Necroptosis. The study incorporates disciplines such as Bacteremia, Meningitis, Virology and Biofilm in addition to Streptococcus pneumoniae.

His biological study spans a wide range of topics, including Ion homeostasis, Caspase, Programmed cell death, Necrosis and Macrophage. He combines subjects such as Pyroptosis, Troponin and Cardiac function curve, Heart failure with his study of Pneumococcal infections. His Immunology research is multidisciplinary, incorporating elements of Sirolimus, Pneumococcal pneumonia and Pneumonia.

His most cited work include:

  • Rapamycin, But Not Resveratrol or Simvastatin, Extends Life Span of Genetically Heterogeneous Mice (625 citations)
  • Tissue-Specific Contributions of Pneumococcal Virulence Factors to Pathogenesis (278 citations)
  • Microarray Analysis of Pneumococcal Gene Expression during Invasive Disease (221 citations)

What are the main themes of his work throughout his whole career to date?

Carlos J. Orihuela spends much of his time researching Streptococcus pneumoniae, Microbiology, Immunology, Pneumonia and Virulence. His studies in Streptococcus pneumoniae integrate themes in fields like Meningitis and Bacteremia. His Microbiology research integrates issues from Biofilm, Bacteria, Bacterial adhesin and Immune system.

His Immunology study incorporates themes from Internal medicine, Disease and Senescence. The concepts of his Virulence study are interwoven with issues in Mutant, Pneumonia, Antibody opsonization and Streptococcaceae. His Pneumolysin study combines topics in areas such as Receptor expression and Programmed cell death, Necroptosis.

He most often published in these fields:

  • Streptococcus pneumoniae (57.14%)
  • Microbiology (57.14%)
  • Immunology (33.33%)

What were the highlights of his more recent work (between 2018-2021)?

  • Microbiology (57.14%)
  • Streptococcus pneumoniae (57.14%)
  • Pneumonia (20.63%)

In recent papers he was focusing on the following fields of study:

The scientist’s investigation covers issues in Microbiology, Streptococcus pneumoniae, Pneumonia, Immune system and Programmed cell death. His primary area of study in Microbiology is in the field of Bacterial pneumonia. His Streptococcus pneumoniae research includes themes of Meningitis, Pathogen, Serotype, Bacteremia and Virulence.

His Meningitis research incorporates themes from Immunology and Otitis. His study in Programmed cell death is interdisciplinary in nature, drawing from both Acquired immune system and Pneumolysin. Carlos J. Orihuela works mostly in the field of Pneumolysin, limiting it down to concerns involving Pathology and, occasionally, Pneumococcal pneumonia.

Between 2018 and 2021, his most popular works were:

  • Streptococcus pneumoniae: Invasion and Inflammation (34 citations)
  • Necroptotic Cell Death Promotes Adaptive Immunity Against Colonizing Pneumococci. (12 citations)
  • Interaction between Streptococcus pneumoniae and Staphylococcus aureus Generates ·OH Radicals That Rapidly Kill Staphylococcus aureus Strains. (8 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Bacteria

His primary scientific interests are in Microbiology, Streptococcus pneumoniae, Necroptosis, Programmed cell death and Cohort. His research in Microbiology is mostly focused on Pathogen. He performs integrative Streptococcus pneumoniae and Spin trapping research in his work.

Carlos J. Orihuela has included themes like Secondary infection, Inflammation, Virus, Influenza A virus and Lung injury in his Necroptosis study. Carlos J. Orihuela has researched Programmed cell death in several fields, including Acquired immune system, Pneumolysin, Antigen-presenting cell, Necrosis and Bacterial pneumonia. His work deals with themes such as Bacteremia, Serotype and Retrospective cohort study, which intersect with Cohort.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Rapamycin, But Not Resveratrol or Simvastatin, Extends Life Span of Genetically Heterogeneous Mice

Richard A. Miller;Richard A. Miller;David E. Harrison;C. M. Astle;Joseph A. Baur.
Journals of Gerontology Series A-biological Sciences and Medical Sciences (2011)

793 Citations

Tissue-Specific Contributions of Pneumococcal Virulence Factors to Pathogenesis

Carlos J. Orihuela;Geli Gao;Kevin P. Francis;Jun Yu.
The Journal of Infectious Diseases (2004)

359 Citations

Laminin receptor initiates bacterial contact with the blood brain barrier in experimental meningitis models

Carlos J. Orihuela;Jafar Mahdavi;Justin Thornton;Beth Mann.
Journal of Clinical Investigation (2009)

265 Citations

Microarray Analysis of Pneumococcal Gene Expression during Invasive Disease

Carlos J. Orihuela;Jana N. Radin;Jack E. Sublett;Geli Gao.
Infection and Immunity (2004)

260 Citations

Defective development and function of Bcl10-deficient follicular, marginal zone and B1 B cells.

Liquan Xue;Stephan W Morris;Stephan W Morris;Carlos Orihuela;Elaine Tuomanen.
Nature Immunology (2003)

214 Citations

Identification of a Candidate Streptococcus pneumoniae Core Genome and Regions of Diversity Correlated with Invasive Pneumococcal Disease

Caroline Obert;Jack Sublett;Deepak Kaushal;Ernesto Hinojosa.
Infection and Immunity (2006)

203 Citations

The Pneumococcal Serine-Rich Repeat Protein Is an Intra-Species Bacterial Adhesin That Promotes Bacterial Aggregation In Vivo and in Biofilms

Carlos J. Sanchez;Pooja Shivshankar;Kim Stol;Samuel Trakhtenbroit.
PLOS Pathogens (2010)

194 Citations

Pore-Forming Toxins Induce Macrophage Necroptosis during Acute Bacterial Pneumonia.

Norberto González-Juarbe;Norberto González-Juarbe;Ryan Paul Gilley;Cecilia Anahí Hinojosa;Cecilia Anahí Hinojosa;Kelley Margaret Bradley.
PLOS Pathogens (2015)

156 Citations

Induction of pro- and anti-inflammatory molecules in a mouse model of pneumococcal pneumonia after influenza.

Matthew W Smith;Jeffrey E Schmidt;Jerold E Rehg;Carlos J Orihuela.
Comparative Medicine (2007)

156 Citations

β-Arrestin 1 Participates in Platelet-Activating Factor Receptor-Mediated Endocytosis of Streptococcus pneumoniae

Jana N. Radin;Carlos J. Orihuela;Gopal Murti;Christopher Guglielmo.
Infection and Immunity (2005)

154 Citations

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University of Michigan–Ann Arbor

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Bill & Melinda Gates Foundation

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Kelly S. Doran

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