Carlos J. Orihuela mainly focuses on Microbiology, Streptococcus pneumoniae, Pneumolysin, Pneumococcal infections and Immunology. His research in Microbiology intersects with topics in Bacterial adhesin, Pathogenicity island, Virulence and Necroptosis. The study incorporates disciplines such as Bacteremia, Meningitis, Virology and Biofilm in addition to Streptococcus pneumoniae.
His biological study spans a wide range of topics, including Ion homeostasis, Caspase, Programmed cell death, Necrosis and Macrophage. He combines subjects such as Pyroptosis, Troponin and Cardiac function curve, Heart failure with his study of Pneumococcal infections. His Immunology research is multidisciplinary, incorporating elements of Sirolimus, Pneumococcal pneumonia and Pneumonia.
Carlos J. Orihuela spends much of his time researching Streptococcus pneumoniae, Microbiology, Immunology, Pneumonia and Virulence. His studies in Streptococcus pneumoniae integrate themes in fields like Meningitis and Bacteremia. His Microbiology research integrates issues from Biofilm, Bacteria, Bacterial adhesin and Immune system.
His Immunology study incorporates themes from Internal medicine, Disease and Senescence. The concepts of his Virulence study are interwoven with issues in Mutant, Pneumonia, Antibody opsonization and Streptococcaceae. His Pneumolysin study combines topics in areas such as Receptor expression and Programmed cell death, Necroptosis.
The scientist’s investigation covers issues in Microbiology, Streptococcus pneumoniae, Pneumonia, Immune system and Programmed cell death. His primary area of study in Microbiology is in the field of Bacterial pneumonia. His Streptococcus pneumoniae research includes themes of Meningitis, Pathogen, Serotype, Bacteremia and Virulence.
His Meningitis research incorporates themes from Immunology and Otitis. His study in Programmed cell death is interdisciplinary in nature, drawing from both Acquired immune system and Pneumolysin. Carlos J. Orihuela works mostly in the field of Pneumolysin, limiting it down to concerns involving Pathology and, occasionally, Pneumococcal pneumonia.
His primary scientific interests are in Microbiology, Streptococcus pneumoniae, Necroptosis, Programmed cell death and Cohort. His research in Microbiology is mostly focused on Pathogen. He performs integrative Streptococcus pneumoniae and Spin trapping research in his work.
Carlos J. Orihuela has included themes like Secondary infection, Inflammation, Virus, Influenza A virus and Lung injury in his Necroptosis study. Carlos J. Orihuela has researched Programmed cell death in several fields, including Acquired immune system, Pneumolysin, Antigen-presenting cell, Necrosis and Bacterial pneumonia. His work deals with themes such as Bacteremia, Serotype and Retrospective cohort study, which intersect with Cohort.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Rapamycin, But Not Resveratrol or Simvastatin, Extends Life Span of Genetically Heterogeneous Mice
Richard A. Miller;Richard A. Miller;David E. Harrison;C. M. Astle;Joseph A. Baur.
Journals of Gerontology Series A-biological Sciences and Medical Sciences (2011)
Tissue-Specific Contributions of Pneumococcal Virulence Factors to Pathogenesis
Carlos J. Orihuela;Geli Gao;Kevin P. Francis;Jun Yu.
The Journal of Infectious Diseases (2004)
Laminin receptor initiates bacterial contact with the blood brain barrier in experimental meningitis models
Carlos J. Orihuela;Jafar Mahdavi;Justin Thornton;Beth Mann.
Journal of Clinical Investigation (2009)
Microarray Analysis of Pneumococcal Gene Expression during Invasive Disease
Carlos J. Orihuela;Jana N. Radin;Jack E. Sublett;Geli Gao.
Infection and Immunity (2004)
Defective development and function of Bcl10-deficient follicular, marginal zone and B1 B cells.
Liquan Xue;Stephan W Morris;Stephan W Morris;Carlos Orihuela;Elaine Tuomanen.
Nature Immunology (2003)
Identification of a Candidate Streptococcus pneumoniae Core Genome and Regions of Diversity Correlated with Invasive Pneumococcal Disease
Caroline Obert;Jack Sublett;Deepak Kaushal;Ernesto Hinojosa.
Infection and Immunity (2006)
The Pneumococcal Serine-Rich Repeat Protein Is an Intra-Species Bacterial Adhesin That Promotes Bacterial Aggregation In Vivo and in Biofilms
Carlos J. Sanchez;Pooja Shivshankar;Kim Stol;Samuel Trakhtenbroit.
PLOS Pathogens (2010)
Pore-Forming Toxins Induce Macrophage Necroptosis during Acute Bacterial Pneumonia.
Norberto González-Juarbe;Norberto González-Juarbe;Ryan Paul Gilley;Cecilia Anahí Hinojosa;Cecilia Anahí Hinojosa;Kelley Margaret Bradley.
PLOS Pathogens (2015)
Streptococcus pneumoniae Translocates into the Myocardium and Forms Unique Microlesions That Disrupt Cardiac Function
Armand O. Brown;Beth Mann;Geli Gao;Jane S. Hankins.
PLOS Pathogens (2014)
Induction of pro- and anti-inflammatory molecules in a mouse model of pneumococcal pneumonia after influenza.
Matthew W Smith;Jeffrey E Schmidt;Jerold E Rehg;Carlos J Orihuela.
Comparative Medicine (2007)
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