Microbiology, Glycoprotein, Platelet, Streptococcus gordonii and Immunology are his primary areas of study. The various areas that he examines in his Microbiology study include Embolism, Vegetation, Recombinant DNA, Staphylococcus aureus and Virulence. His work in Virulence tackles topics such as Pathogenesis which are related to areas like Streptococcus agalactiae, Group B, Meningitis and Streptococcus.
His biological study spans a wide range of topics, including Endocarditis, Immunoprecipitation, Salicylic acid, Fibrin and Cell surface receptor. His research in Streptococcus gordonii intersects with topics in Peptide sequence, Streptococcus parasanguinis and Platelet membrane glycoprotein. His Immunopathology study in the realm of Immunology interacts with subjects such as Xanthine oxidation.
His primary scientific interests are in Microbiology, Streptococcus gordonii, Glycoprotein, Bacterial adhesin and Biochemistry. His study in Microbiology is interdisciplinary in nature, drawing from both Endocarditis, Staphylococcus aureus, Streptococcus, Streptococcus mitis and Virulence. The Virulence study which covers Pathogenesis that intersects with Streptococcus agalactiae.
His work deals with themes such as Transport protein, Glycosylation, Peptide sequence, Signal peptide and Streptococcus parasanguinis, which intersect with Streptococcus gordonii. His Glycoprotein research integrates issues from Platelet, Cytoplasm and Binding protein. His work carried out in the field of Bacterial adhesin brings together such families of science as Serine, Streptococcus sanguinis, Receptor, Glycan and Cell biology.
His main research concerns Bacterial adhesin, Microbiology, Daptomycin, Streptococcus mitis and Glycoprotein. Paul M. Sullam has researched Bacterial adhesin in several fields, including Receptor, Glycan, Streptococcus gordonii and Cell biology. His Microbiology study combines topics in areas such as Endocarditis, Group B, Vaccination, Streptococcus and Antibody.
His Streptococcus research incorporates elements of Serotype and Immunology. His Glycoprotein research is multidisciplinary, relying on both Streptococcus sanguinis and Serine. Paul M. Sullam has included themes like Platelet and Acetylation in his Serine study.
Paul M. Sullam spends much of his time researching Bacterial adhesin, Microbiology, Streptococcus sanguinis, Glycan and Biochemistry. His Bacterial adhesin study integrates concerns from other disciplines, such as Binding site and Streptococcus gordonii. His work on Antimicrobial peptides is typically connected to Cardiolipin as part of general Microbiology study, connecting several disciplines of science.
His studies deal with areas such as Protein tertiary structure, Sialic acid and Virulence as well as Streptococcus sanguinis. His Glycan research is under the purview of Glycoprotein. In general Glycoprotein, his work in Platelet membrane glycoprotein is often linked to Sialic Acid Binding Immunoglobulin-like Lectins linking many areas of study.
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Two Controlled Trials of Rifabutin Prophylaxis against Mycobacterium avium Complex Infection in AIDS
Stephen D. Nightingale;D. William Cameron;Fred M. Gordin;Paul M. Sullam.
The New England Journal of Medicine (1993)
Diminished virulence of a sar-/agr- mutant of Staphylococcus aureus in the rabbit model of endocarditis.
Ambrose L. Cheung;Kelly J. Eberhardt;Edward Chung;Michael R. Yeaman.
Journal of Clinical Investigation (1994)
An accessory sec locus of Streptococcus gordonii is required for export of the surface protein GspB and for normal levels of binding to human platelets
Barbara A. Bensing;Paul M. Sullam.
Molecular Microbiology (2002)
Pattern Recognition by TREM-2: Binding of Anionic Ligands
Michael R. Daws;Paul M. Sullam;Eréne C. Niemi;Thomas T. Chen.
Journal of Immunology (2003)
Role of SraP, a Serine-Rich Surface Protein of Staphylococcus aureus, in Binding to Human Platelets
Ian R. Siboo;Henry F. Chambers;Henry F. Chambers;Paul M. Sullam;Paul M. Sullam.
Infection and Immunity (2005)
Platelet microbicidal proteins and neutrophil defensin disrupt the Staphylococcus aureus cytoplasmic membrane by distinct mechanisms of action.
M R Yeaman;A S Bayer;S P Koo;W Foss.
Journal of Clinical Investigation (1998)
Diminished platelet binding in vitro by Staphylococcus aureus is associated with reduced virulence in a rabbit model of infective endocarditis.
Paul M. Sullam;Arnold S. Bayer;Wendy M. Foss;Andambrose L. Cheung.
Infection and Immunity (1996)
The Streptococcus gordonii Surface Proteins GspB and Hsa Mediate Binding to Sialylated Carbohydrate Epitopes on the Platelet Membrane Glycoprotein Ibα
Barbara A. Bensing;José A. López;Paul M. Sullam.
Infection and Immunity (2004)
Acetylsalicylic Acid Reduces Vegetation Bacterial Density, Hematogenous Bacterial Dissemination, and Frequency of Embolic Events in Experimental Staphylococcus aureus Endocarditis Through Antiplatelet and Antibacterial Effects
Leon Iri Kupferwasser;Michael R. Yeaman;Shelley M. Shapiro;Cynthia C. Nast.
Circulation (1999)
The Pneumococcal Serine-Rich Repeat Protein Is an Intra-Species Bacterial Adhesin That Promotes Bacterial Aggregation In Vivo and in Biofilms
Carlos J. Sanchez;Pooja Shivshankar;Kim Stol;Samuel Trakhtenbroit.
PLOS Pathogens (2010)
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