What is he best known for?
The fields of study he is best known for:
Bradley K. Yoder focuses on Cilium, Cell biology, Intraflagellar transport, Basal body and Polycystic kidney disease.
His Cilium research integrates issues from Cystic kidney disease, Kidney metabolism, Pathology, KIF3A and Organelle.
He interconnects Axoneme, Ciliary transition zone and Mutant in the investigation of issues within Cell biology.
Bradley K. Yoder combines subjects such as Sonic hedgehog, Neural tube patterning, Caenorhabditis elegans and Smoothened with his study of Intraflagellar transport.
His Basal body study deals with Microtubule intersecting with Undulipodium.
Many of his research projects under Polycystic kidney disease are closely connected to Polaris with Polaris, tying the diverse disciplines of science together.
His most cited work include:
- The Polycystic Kidney Disease Proteins, Polycystin-1, Polycystin-2, Polaris, and Cystin, Are Co-Localized in Renal Cilia (765 citations)
- Gli2 and Gli3 Localize to Cilia and Require the Intraflagellar Transport Protein Polaris for Processing and Function (734 citations)
- Comparative Genomics Identifies a Flagellar and Basal Body Proteome that Includes the BBS5 Human Disease Gene (665 citations)
What are the main themes of his work throughout his whole career to date?
His primary scientific interests are in Cilium, Cell biology, Intraflagellar transport, Pathology and Internal medicine.
Bradley K. Yoder has researched Cilium in several fields, including Ciliopathies, Basal body, Ciliopathy and Polycystic kidney disease.
His work is dedicated to discovering how Cell biology, Axoneme are connected with Ciliary transition zone and other disciplines.
The study incorporates disciplines such as Sonic hedgehog, KIF3A, Caenorhabditis elegans and Anatomy in addition to Intraflagellar transport.
His work in Internal medicine covers topics such as Endocrinology which are related to areas like Epithelium.
The Autosomal Recessive Polycystic Kidney Disease study combines topics in areas such as Molecular biology, Polycystin 2 and Gene.
He most often published in these fields:
- Cilium (59.88%)
- Cell biology (56.17%)
- Intraflagellar transport (21.60%)
What were the highlights of his more recent work (between 2018-2021)?
- Cell biology (56.17%)
- Cilium (59.88%)
- Ciliopathy (9.26%)
In recent papers he was focusing on the following fields of study:
Cell biology, Cilium, Ciliopathy, Kidney and Pathology are his primary areas of study.
His specific area of interest is Cell biology, where Bradley K. Yoder studies Ciliogenesis.
In his research, BBSome, BBS5 and Live cell imaging is intimately related to Ciliopathies, which falls under the overarching field of Cilium.
His studies deal with areas such as Retinal degeneration, Protein composition and Cell type specific as well as Ciliopathy.
His work on Cystic kidney as part of general Kidney research is often related to Macrophage proliferation, thus linking different fields of science.
In general Pathology, his work in Cystic kidney disease and Disease is often linked to Cystic liver disease linking many areas of study.
Between 2018 and 2021, his most popular works were:
- Single-Cell RNA Sequencing Identifies Candidate Renal Resident Macrophage Gene Expression Signatures across Species. (30 citations)
- Resident macrophages reprogram toward a developmental state after acute kidney injury (26 citations)
- Tissue-Resident Macrophages Promote Renal Cystic Disease. (16 citations)
In his most recent research, the most cited papers focused on:
His scientific interests lie mostly in Cell biology, Innate immune system, Kidney, Cilium and Pathogenesis.
His research in Cell biology intersects with topics in Protein composition and Ciliopathy.
The concepts of his Innate immune system study are interwoven with issues in Flow cytometry, Cell type and Cluster of differentiation.
His Kidney study integrates concerns from other disciplines, such as Cyst, Urinary system, Acute kidney injury and Kidney development.
Bradley K. Yoder performs integrative study on Cilium and Macrophage proliferation.
His research in Pathogenesis intersects with topics in Cystic kidney disease and Autosomal dominant polycystic kidney disease.
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