Richard P. Woychik focuses on Genetics, Gene, Molecular biology, Locus and Mutant. Richard P. Woychik regularly ties together related areas like Computational biology in his Genetics studies. His Molecular biology study spans across into areas like PCDH15 and CDH23.
Richard P. Woychik interconnects Gene mapping, Regulation of gene expression and Genotype in the investigation of issues within Locus. His research in Mutant intersects with topics in Mutation and Polycystic kidney disease, Autosomal Recessive Polycystic Kidney Disease. His Gene expression research includes elements of Genetically modified mouse and Ectopic expression.
His primary areas of investigation include Genetics, Gene, Molecular biology, Mutant and Gene expression. His work on Computational biology expands to the thematically related Gene. His studies deal with areas such as Nucleic acid sequence, DNA, Recombinant DNA, Coding region and Complementary DNA as well as Molecular biology.
The various areas that Richard P. Woychik examines in his Mutant study include Transgene, Mutation, Pathology, Autosomal Recessive Polycystic Kidney Disease and Cochlea. His Gene expression research is multidisciplinary, relying on both Endocrinology, Ectopic expression, Internal medicine, Histone and Epigenetics. His Endocrinology course of study focuses on Melanocortin 1 receptor and Receptor.
Gene expression, Mitochondrion, Epigenetics, Mitochondrial DNA and Cell biology are his primary areas of study. His study with Gene expression involves better knowledge in Genetics. His work on Epigenome, Locus, Regulation of gene expression and Exon as part of his general Genetics study is frequently connected to Toxicant, thereby bridging the divide between different branches of science.
His Mitochondrial DNA study combines topics from a wide range of disciplines, such as Histone and Acetylation. His research integrates issues of Mutation, Alpha, Downregulation and upregulation and Receptor in his study of Cell biology. Many of his research projects under Biochemistry are closely connected to Citric acid cycle with Citric acid cycle, tying the diverse disciplines of science together.
The scientist’s investigation covers issues in Mitochondrial DNA, Epigenetics, Biochemistry, Cell biology and Histone. Richard P. Woychik has researched Epigenetics in several fields, including Methylation, DNA methylation and Methionine. In general Biochemistry, his work in Mitochondrion, Cell growth, Oxidative phosphorylation and Cellular respiration is often linked to Citric acid cycle linking many areas of study.
His biological study spans a wide range of topics, including Gene expression and Acetylation.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Agouti protein is an antagonist of the melanocyte-stimulating-hormone receptor.
Dongsi Lu;Derril Willard;Indravadan R. Patel;Sue Kadwell.
Nature (1994)
Molecular characterization of the mouse agouti locus
Scott J. Bultman;Edward J. Michaud;Richard P. Woychik.
Cell (1992)
The knockout mouse project
Christopher P. Austin;James F. Battey;Allan Bradley;Maja Bucan.
Nature Genetics (2004)
The Oak Ridge Polycystic Kidney (orpk) disease gene is required for left-right axis determination
N.S. Murcia;W.G. Richards;B.K. Yoder;M.L. Mucenski.
Development (2000)
Candidate gene associated with a mutation causing recessive polycystic kidney disease in mice
J. H. Moyer;M. J. Lee-Tischler;H.-Y. Kwon;J. J. Schrick.
Science (1994)
The mouse Ames waltzer hearing-loss mutant is caused by mutation of Pcdh15, a novel protocadherin gene.
Kumar N. Alagramam;Crystal L. Murcia;Heajoon Y. Kwon;Heajoon Y. Kwon;Karen S. Pawlowski.
Nature Genetics (2001)
Ectopic expression of the agouti gene in transgenic mice causes obesity, features of type II diabetes, and yellow fur
M L Klebig;J E Wilkinson;J G Geisler;R P Woychik.
Proceedings of the National Academy of Sciences of the United States of America (1995)
Differential expression of a new dominant agouti allele (Aiapy) is correlated with methylation state and is influenced by parental lineage.
E J Michaud;M J van Vugt;S J Bultman;H O Sweet.
Genes & Development (1994)
TCA Cycle and Mitochondrial Membrane Potential Are Necessary for Diverse Biological Functions
Inmaculada Martínez-Reyes;Lauren P. Diebold;Hyewon Kong;Michael Schieber.
Molecular Cell (2016)
An inherited limb deformity created by insertional mutagenesis in a transgenic mouse
R. P. Woychik;T. A. Stewart;T. A. Stewart;L. G. Davis;L. G. Davis;P. D'Eustachio.
Nature (1985)
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