Birke Bartosch spends much of her time researching Virology, Hepatitis C virus, Virus, Glycoprotein and Hepacivirus. Her studies in Virology integrate themes in fields like Monoclonal antibody, Antibody and Immunology. Her Hepatitis C virus research incorporates elements of Cell culture, Neutralization, Hypervariable region and Tropism.
Her Virus study integrates concerns from other disciplines, such as Internalization and Scavenger receptor. Her research integrates issues of Endoplasmic reticulum and Cell biology in her study of Glycoprotein. Her work in Hepacivirus tackles topics such as Infectivity which are related to areas like Hepatitis A, Genotype and Apolipoprotein C1.
Birke Bartosch mostly deals with Virology, Hepatitis C virus, Virus, Glycoprotein and Molecular biology. Her study in Virology is interdisciplinary in nature, drawing from both Antibody and Monoclonal antibody. Birke Bartosch mostly deals with Hepacivirus in her studies of Hepatitis C virus.
Her biological study spans a wide range of topics, including Recombinant virus and Antigen. Her Glycoprotein study incorporates themes from Endoplasmic reticulum, Viral entry, Lipid bilayer fusion and Liposome. Her Molecular biology research also works with subjects such as
Birke Bartosch mainly focuses on Hepatitis C virus, Cell biology, Oxidative stress, Immunology and Cancer research. Hepatitis C virus is the subject of her research, which falls under Virology. Her Virology research integrates issues from Molecular biology, Cell and Lipid droplet.
Her research investigates the link between Cell biology and topics such as Catabolism that cross with problems in Hepatocyte differentiation. Birke Bartosch has researched Oxidative stress in several fields, including Reactive oxygen species, NS5A, Biogenic Polyamines and Bioinformatics. As part of the same scientific family, Birke Bartosch usually focuses on Immunology, concentrating on Carcinogenesis and intersecting with Innate immune system, Pattern recognition receptor, Substrate-level phosphorylation and DNA.
Her primary scientific interests are in Oxidative stress, Immunology, Virus, Disease and Cancer research. Her studies deal with areas such as Mitochondrion, Reactive oxygen species, Hepatitis C and NS5A as well as Oxidative stress. Her Immunology study deals with Carcinogenesis intersecting with Liver cancer, Cell cycle, Hepatitis B and Hepatitis.
Her Virus research incorporates themes from Inflammation, Respiratory tract and Common cold. The study incorporates disciplines such as Tumor Virus, Cell migration, Virus Physiological Phenomena, Oncovirus and Lipid metabolism in addition to Cancer research. Birke Bartosch studies Hepacivirus which is a part of Hepatitis C virus.
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Infectious Hepatitis C Virus Pseudo-particles Containing Functional E1–E2 Envelope Protein Complexes
Birke Bartosch;Jean Dubuisson;François-Loïc Cosset.
Journal of Experimental Medicine (2003)
Cell entry of hepatitis C virus requires a set of co-receptors that include the CD81 tetraspanin and the SR-B1 scavenger receptor.
Birke Bartosch;Alessandra Vitelli;Christelle Granier;Caroline Goujon.
Journal of Biological Chemistry (2003)
Hepatitis C Virus NS5A Protein Triggers Oxidative Stress by Inducing NADPH Oxidases 1 and 4 and Cytochrome P450 2E1.
Olga A. Smirnova;Olga N. Ivanova;Birke Bartosch;Vladimir T. Valuev-Elliston.
Oxidative Medicine and Cellular Longevity (2016)
Rapid induction of virus-neutralizing antibodies and viral clearance in a single-source outbreak of hepatitis C
Jan M. Pestka;Mirjam B. Zeisel;Edith Bläser;Peter Schürmann.
Proceedings of the National Academy of Sciences of the United States of America (2007)
In vitro assay for neutralizing antibody to hepatitis C virus: evidence for broadly conserved neutralization epitopes.
Birke Bartosch;Jens Bukh;Jean-Christophe Meunier;Christelle Granier.
Proceedings of the National Academy of Sciences of the United States of America (2003)
An Interplay between Hypervariable Region 1 of the Hepatitis C Virus E2 Glycoprotein, the Scavenger Receptor BI, and High-Density Lipoprotein Promotes both Enhancement of Infection and Protection against Neutralizing Antibodies
Birke Bartosch;Géraldine Verney;Marlène Dreux;Peggy Donot.
Journal of Virology (2005)
Human serum facilitates hepatitis C virus infection, and neutralizing responses inversely correlate with viral replication kinetics at the acute phase of hepatitis C virus infection.
Dimitri Lavillette;Yoann Morice;Georgios Germanidis;Peggy Donot.
Journal of Virology (2005)
Characterization of host-range and cell entry properties of the major genotypes and subtypes of hepatitis C virus.
Dimitri Lavillette;Alexander W. Tarr;Cécile Voisset;Peggy Donot.
Role of N-Linked Glycans in the Functions of Hepatitis C Virus Envelope Glycoproteins
Anne Goffard;Nathalie Callens;Birke Bartosch;Czeslaw Wychowski.
Journal of Virology (2005)
Evidence for cross-genotype neutralization of hepatitis C virus pseudo-particles and enhancement of infectivity by apolipoprotein C1
Jean-Christophe Meunier;Ronald E. Engle;Kristina Faulk;Ming Zhao.
Proceedings of the National Academy of Sciences of the United States of America (2005)
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