His scientific interests lie mostly in Immunology, Immune system, Leptin, Autoimmunity and Internal medicine. As part of his studies on Immunology, Antonio La Cava frequently links adjacent subjects like Cell. His study in Immune system is interdisciplinary in nature, drawing from both Inflammation, Hormone, Receptor and Interferon.
His Autoimmunity research incorporates elements of Autoimmune disease and Antigen. Internal medicine connects with themes related to Endocrinology in his study. His work carried out in the field of IL-2 receptor brings together such families of science as Natural killer T cell and FOXP3.
Antonio La Cava focuses on Immunology, Immune system, Autoimmunity, Leptin and Internal medicine. His Immunology study frequently draws connections to other fields, such as Systemic lupus erythematosus. His Immune system research includes elements of Disease and Pathogenesis.
The various areas that Antonio La Cava examines in his Autoimmunity study include Myasthenia gravis, Cytokine, Immunity, Immune homeostasis and Neuroscience. He has researched Leptin in several fields, including Adipose tissue, Hormone and Receptor. His research brings together the fields of Endocrinology and Internal medicine.
Antonio La Cava spends much of his time researching Immunology, Immune system, Systemic lupus erythematosus, Autoimmunity and Inflammation. Antonio La Cava interconnects Brain ischemia and Rheumatology in the investigation of issues within Immunology. His Immune system study incorporates themes from Spleen, Cell specific, Intracerebral hemorrhage and Transforming growth factor.
His research in Autoimmunity intersects with topics in Intensive care medicine and Immunotherapy. His Inflammation study integrates concerns from other disciplines, such as Immune tolerance, Leptin and Effector. His studies examine the connections between Lupus erythematosus and genetics, as well as such issues in FOXP3, with regards to T cell.
His primary areas of study are Immune system, Immunology, Spleen, Autoimmunity and Inflammation. Antonio La Cava is interested in Immunity, which is a branch of Immune system. He has included themes like Cell specific, Intracerebral hemorrhage, Transcriptome and Immunosuppression in his Spleen study.
In his study, Acquired immune system is strongly linked to Leptin, which falls under the umbrella field of Autoimmunity. His work investigates the relationship between Acquired immune system and topics such as T cell that intersect with problems in Neuroscience and Brain ischemia. Many of his studies on Inflammation apply to Immunotherapy as well.
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The weight of leptin in immunity
Antonio La Cava;Giuseppe Matarese.
Nature Reviews Immunology (2004)
A Key Role of Leptin in the Control of Regulatory T Cell Proliferation
Veronica De Rosa;Claudio Procaccini;Gaetano Calì;Giuseppe Pirozzi.
Leptin surge precedes onset of autoimmune encephalomyelitis and correlates with development of pathogenic T cell responses
Veronica Sanna;Antonio Di Giacomo;Antonio La Cava;Robert I. Lechler.
Journal of Clinical Investigation (2003)
Organ-specific features of natural killer cells
Fu-Dong Shi;Fu-Dong Shi;Hans-Gustaf Ljunggren;Antonio La Cava;Luc Van Kaer.
Nature Reviews Immunology (2011)
Leptin increase in multiple sclerosis associates with reduced number of CD4+CD25+ regulatory T cells
Giuseppe Matarese;Pietro Biagio Carrieri;Antonio La Cava;Francesco Perna.
Proceedings of the National Academy of Sciences of the United States of America (2005)
An oscillatory switch in mTOR kinase activity sets regulatory T cell responsiveness.
Claudio Procaccini;Veronica De Rosa;Mario Galgani;Luisa Abanni.
Unraveling the multiple roles of leptin in inflammation and autoimmunity.
Antonio La Cava;Carlo Alviggi;Giuseppe Matarese.
Journal of Molecular Medicine (2004)
CD4+CD25+ Tregs and NKT cells: regulators regulating regulators
Antonio La Cava;Luc Van Kaer;Fu-Dong-Shi.
Trends in Immunology (2006)
Leptin and Inflammation
Noriko Iikuni;Queenie Lai Kwan Lam;Liwei Lu;Giuseppe Matarese.
Current Immunology Reviews (2008)
Leptin accelerates autoimmune diabetes in female NOD mice.
Giuseppe Matarese;Veronica Sanna;Robert I. Lechler;Nora Sarvetnick.
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