2016 - Member of the National Academy of Medicine (NAM)
Member of the Association of American Physicians
Antonello Bonci spends much of his time researching Neuroscience, Ventral tegmental area, Dopamine, Optogenetics and Nucleus accumbens. His Neuroscience study combines topics in areas such as Long-term potentiation and Glutamatergic. His Ventral tegmental area research is multidisciplinary, incorporating elements of Neurotransmission and Opioid.
His Dopamine study combines topics from a wide range of disciplines, such as PVLV, Protein kinase C and Pharmacology. Many of his research projects under Optogenetics are closely connected to Photic Stimulation with Photic Stimulation, tying the diverse disciplines of science together. As a part of the same scientific study, Antonello Bonci usually deals with the Nucleus accumbens, concentrating on Amygdala and frequently concerns with Cerebral cortex, Insula and Hyperpolarization.
Neuroscience, Ventral tegmental area, Dopamine, Nucleus accumbens and Addiction are his primary areas of study. Antonello Bonci has researched Neuroscience in several fields, including Synaptic plasticity, Long-term potentiation and Glutamatergic. His Ventral tegmental area research integrates issues from Orexin, Electrophysiology, NMDA receptor, Glutamate receptor and Substantia nigra.
Antonello Bonci works mostly in the field of Dopamine, limiting it down to topics relating to Pharmacology and, in certain cases, Receptor, Alcohol and Ethanol. His Nucleus accumbens research is multidisciplinary, relying on both Medium spiny neuron and Amygdala. Antonello Bonci combines subjects such as Transcranial magnetic stimulation, Self-administration, Neuroplasticity and Substance use with his study of Addiction.
His main research concerns Neuroscience, Dopamine, Transcranial magnetic stimulation, Addiction and Ventral tegmental area. He integrates many fields, such as Neuroscience and Microglia, in his works. His Dopamine research is multidisciplinary, incorporating perspectives in Synaptic plasticity, Receptor, Neuron, Extinction and Amygdala.
In his study, Chronic cocaine is inextricably linked to Excitatory postsynaptic potential, which falls within the broad field of Synaptic plasticity. The study incorporates disciplines such as Neuromodulation, Brain stimulation, Prefrontal cortex, Substance use and Opioid in addition to Addiction. Ventral tegmental area and Pharmacology are frequently intertwined in his study.
His scientific interests lie mostly in Neuroscience, Dopamine, Addiction, Receptor and Central nervous system. The various areas that Antonello Bonci examines in his Neuroscience study include Macrophage and Function. His research integrates issues of Self-administration and Opioid addiction, Opioid in his study of Dopamine.
Antonello Bonci interconnects Dopamine systems, κ-opioid receptor and Emotional behavior in the investigation of issues within Addiction. His Central nervous system research includes elements of Phenotype, Cell morphology and Cell signaling. His research investigates the connection between Astrocyte and topics such as Calcium imaging that intersect with issues in Neuron.
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Drugs of abuse and stress trigger a common synaptic adaptation in dopamine neurons.
Daniel Saal;Yan Dong;Antonello Bonci;Robert C Malenka.
Neuron (2003)
Single cocaine exposure in vivo induces long-term potentiation in dopamine neurons
Mark A. Ungless;Jennifer L. Whistler;Robert C. Malenka;Antonello Bonci.
Nature (2001)
Phasic Firing in Dopaminergic Neurons Is Sufficient for Behavioral Conditioning
Hsing-Chen Tsai;Feng Zhang;Antoine Roger Adamantidis;Garret D Stuber.
Science (2009)
Orexin A in the VTA is critical for the induction of synaptic plasticity and behavioral sensitization to cocaine.
Stephanie L. Borgland;Sharif A. Taha;Federica Sarti;Howard L. Fields.
Neuron (2006)
Excitatory transmission from the amygdala to nucleus accumbens facilitates reward seeking
Garret D. Stuber;Garret D. Stuber;Dennis R. Sparta;Dennis R. Sparta;Alice M. Stamatakis;Wieke A. van Leeuwen.
Nature (2011)
Long-term depression in the nucleus accumbens: a neural correlate of behavioral sensitization to cocaine.
Mark J. Thomas;Corinne Beurrier;Antonello Bonci;Robert C. Malenka.
Nature Neuroscience (2001)
Chemogenetics revealed: DREADD occupancy and activation via converted clozapine.
Juan L. Gomez;Jordi Bonaventura;Wojciech Lesniak;William B. Mathews.
Science (2017)
Synaptic and behavioral profile of multiple glutamatergic inputs to the nucleus accumbens
Jonathan P. Britt;Faiza Benaliouad;Ross A. McDevitt;Garret D. Stuber.
Neuron (2012)
Dopaminergic terminals in the nucleus accumbens but not the dorsal striatum corelease glutamate.
Garret D. Stuber;Thomas S. Hnasko;Jonathan P. Britt;Robert H. Edwards.
The Journal of Neuroscience (2010)
Rescuing cocaine-induced prefrontal cortex hypoactivity prevents compulsive cocaine seeking
Billy T. Chen;Hau-Jie Yau;Christina Hatch;Ikue Kusumoto-Yoshida.
Nature (2013)
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