What is she best known for?
The fields of study she is best known for:
Her primary areas of study are Microtubule, Cell biology, Biochemistry, Tubulin and Detyrosination.
Her research in the fields of Kinesin overlaps with other disciplines such as Cortical dysplasia.
Her Cell biology study integrates concerns from other disciplines, such as Neuroplasticity, Neurotransmission and Cytoskeleton.
Her Nocodazole and Microtubule Depolymerization Process study in the realm of Cytoskeleton interacts with subjects such as Tubulin Modulators.
The concepts of her Tubulin study are interwoven with issues in Myosin light-chain kinase, Neurodegeneration and Enzyme.
Her Detyrosination research incorporates themes from Growth cone, Tyrosine and Tubulin—tyrosine ligase.
Her most cited work include:
- Deletion of beta 1 integrins in mice results in inner cell mass failure and peri-implantation lethality. (524 citations)
- Mutations in TUBG1 , DYNC1H1 , KIF5C and KIF2A cause malformations of cortical development and microcephaly (276 citations)
- Tubulin tyrosination is a major factor affecting the recruitment of CAP-Gly proteins at microtubule plus ends (242 citations)
What are the main themes of her work throughout her whole career to date?
Her scientific interests lie mostly in Microtubule, Cell biology, Neuroscience, Tubulin and MAP6.
Annie Andrieux works in the field of Microtubule, focusing on Kinesin in particular.
The study incorporates disciplines such as Basal body and Nocodazole in addition to Cell biology.
Her work on Microtubule Depolymerization Process as part of general Nocodazole study is frequently linked to Tubulin Modulators, therefore connecting diverse disciplines of science.
Her Neuroscience research incorporates elements of Synaptic plasticity, Schizophrenia, Internal medicine and Endocrinology.
Her work on Detyrosination as part of her general Tubulin study is frequently connected to Polyglutamylation, thereby bridging the divide between different branches of science.
She most often published in these fields:
- Microtubule (72.85%)
- Cell biology (64.24%)
- Neuroscience (34.44%)
What were the highlights of her more recent work (between 2017-2021)?
- Microtubule (72.85%)
- Cell biology (64.24%)
- Tubulin (39.07%)
In recent papers she was focusing on the following fields of study:
Her primary scientific interests are in Microtubule, Cell biology, Tubulin, Neuroscience and MAP6.
She performs multidisciplinary study in the fields of Microtubule and Paclitaxel via her papers.
She has researched Cell biology in several fields, including Tyrosine, Huntingtin, Knockout mouse and Synaptic plasticity.
Her work on Detyrosination as part of general Tubulin study is frequently linked to Polyglutamylation, bridging the gap between disciplines.
Her Neuroscience research includes elements of Schizophrenia and Cytoskeleton.
Her work in Cytoskeleton covers topics such as Actin which are related to areas like Actin cytoskeleton.
Between 2017 and 2021, her most popular works were:
- Excessive tubulin polyglutamylation causes neurodegeneration and perturbs neuronal transport (50 citations)
- Excessive tubulin polyglutamylation causes neurodegeneration and perturbs neuronal transport (50 citations)
- Cell-Intrinsic Control of Interneuron Migration Drives Cortical Morphogenesis (25 citations)
In her most recent research, the most cited papers focused on:
The scientist’s investigation covers issues in Microtubule, Cell biology, Tubulin, Biophysics and MAP6.
The Cell biology study combines topics in areas such as Neuron and Cytoskeleton.
Her studies in Cytoskeleton integrate themes in fields like Axon guidance, Axon, Microtubule plus-end and Zebrafish.
Her study in Tubulin is interdisciplinary in nature, drawing from both Degeneration, Neuronal transport, Downregulation and upregulation and Neurodegeneration.
Her biological study spans a wide range of topics, including Synaptic plasticity, Long-term potentiation, Dendritic spine and Actin.
Her Detyrosination research is multidisciplinary, relying on both Protein structure, Knockout mouse and Microcephaly.
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