Ammon B. Peck mainly investigates Immunology, Internal medicine, Endocrinology, NOD mice and Nod. Autoimmune disease, Autoantibody, Autoimmunity, Pathogenesis and Immune system are subfields of Immunology in which his conducts study. His work carried out in the field of Internal medicine brings together such families of science as Diabetes mellitus and Stem cell.
His studies examine the connections between Diabetes mellitus and genetics, as well as such issues in In vitro, with regards to Serum free and Blood serum. His Stem cell study incorporates themes from Islet, Pancreas and Adult stem cell. His research in NOD mice intersects with topics in Salivary gland, Saliva and Insulitis.
The scientist’s investigation covers issues in Immunology, Internal medicine, Endocrinology, Molecular biology and Nod. His is involved in several facets of Immunology study, as is seen by his studies on Autoimmunity, Autoimmune disease, Immune system, Autoantibody and Antigen. His work on Oxalate expands to the thematically related Internal medicine.
Ammon B. Peck has researched Endocrinology in several fields, including Proteolytic enzymes, Stem cell and Saliva. His Molecular biology research includes elements of Antibody, Biochemistry, Gene and Lymphocyte. His work in Nod tackles topics such as Exocrine gland which are related to areas like Pathology.
His primary areas of investigation include Immunology, Disease, Autoimmune disease, Internal medicine and Autoimmunity. Ammon B. Peck usually deals with Immunology and limits it to topics linked to Salivary gland and Cytokine. His Autoimmune disease research incorporates themes from Diabetes mellitus, Nod, Receptor, Signal transduction and Pathophysiology.
He has included themes like Endocrinology and Microbiology in his Internal medicine study. The various areas that Ammon B. Peck examines in his Endocrinology study include Tears, Interleukin 17 and Saliva. His research integrates issues of Dacryoadenitis and Lacrimal gland in his study of Autoimmunity.
His main research concerns Immunology, Internal medicine, Disease, Endocrinology and Autoimmune disease. His study in Immunology is interdisciplinary in nature, drawing from both Exocrine gland and Nod. The Nod study combines topics in areas such as C57BL/6 and Gene expression.
His study connects Reactive oxygen species and Internal medicine. His Disease study combines topics in areas such as Sjogren s and Pathogenesis. His Endocrinology research incorporates elements of Respiratory burst and Oxidase test.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
A gene map of the human genome
G. D. Schuler;M. S. Boguski;E. A. Stewart;L. D. Stein.
Reversal of insulin-dependent diabetes using islets generated in vitro from pancreatic stem cells.
Vijayakumar K. Ramiya;Michael Maraist;Karl E. Arfors;Desmond A. Schatz.
Nature Medicine (2000)
Adult hematopoietic stem cells provide functional hemangioblast activity during retinal neovascularization
Maria B. Grant;W. Stratford May;Sergio Caballero;Gary A. J. Brown.
Nature Medicine (2002)
A Physical Map of 30,000 Human Genes
P. Deloukas;G. D. Schuler;G. Gyapay;E. M. Beasley.
In vitro trans-differentiation of adult hepatic stem cells into pancreatic endocrine hormone-producing cells
Lijun Yang;Shiwu Li;Heather Hatch;Kim Ahrens.
Proceedings of the National Academy of Sciences of the United States of America (2002)
Salivary gland tissue expression of interleukin-23 and interleukin-17 in Sjögren's syndrome: findings in humans and mice.
Cuong Q. Nguyen;Min H. Hu;Yi Li;Carol Stewart.
Arthritis & Rheumatism (2008)
Transfer of human serum IgG to nonobese diabetic Igμnull mice reveals a role for autoantibodies in the loss of secretory function of exocrine tissues in Sjögren’s syndrome
Christopher P. Robinson;Jason Brayer;Shigeo Yamachika;Thomas R. Esch.
Proceedings of the National Academy of Sciences of the United States of America (1998)
Insulin immunization of nonobese diabetic mice induces a protective insulitis characterized by diminished intraislet interferon-gamma transcription.
A. Muir;A. Peck;M. Clare-Salzler;Yao-Hua Song.
Journal of Clinical Investigation (1995)
Oxalobacter sp. reduces urinary oxalate excretion by promoting enteric oxalate secretion
M. Hatch;J. Cornelius;M. Allison;H. Sidhu.
Kidney International (2006)
Direct correlation between hyperoxaluria/oxalate stone disease and the absence of the gastrointestinal tract-dwelling bacterium Oxalobacter formigenes: possible prevention by gut recolonization or enzyme replacement therapy.
H Sidhu;M E Schmidt;J G Cornelius;S Thamilselvan.
Journal of The American Society of Nephrology (1999)
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