D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 110 Citations 61,564 400 World Ranking 3165 National Ranking 1810
Genetics D-index 110 Citations 61,640 396 World Ranking 325 National Ranking 167

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Cancer
  • DNA

His primary areas of study are Genetics, Cancer research, Gene expression profiling, Gene and Complementary DNA. His is doing research in Prostate cancer, Germline mutation, Mutation, Locus and Missense mutation, both of which are found in Genetics. He is involved in the study of Cancer research that focuses on Melanoma in particular.

His Gene expression profiling study incorporates themes from Cell cycle, Genetic heterogeneity, Motility and Regulation of gene expression. The various areas that Jeffrey M. Trent examines in his Gene study include Cancer and Binimetinib. His Complementary DNA study combines topics from a wide range of disciplines, such as Gene chip analysis, Microarray, DNA microarray, Gene expression and Molecular biology.

His most cited work include:

  • WAF1, a potential mediator of p53 tumor suppression (7441 citations)
  • Mutations of a mutS homolog in hereditary nonpolyposis colorectal cancer (2028 citations)
  • The transcriptional program in the response of human fibroblasts to serum. (1836 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of investigation include Genetics, Cancer research, Molecular biology, Gene and Cancer. His Cancer research research includes elements of Mutation, Germline mutation, Carcinogenesis and SMARCA4. His Molecular biology study which covers Chromosome microdissection that intersects with Microdissection.

Gene is represented through his Complementary DNA, Gene expression, Gene expression profiling and Genome research. In his study, which falls under the umbrella issue of Complementary DNA, DNA microarray is strongly linked to Microarray. His studies examine the connections between Cancer and genetics, as well as such issues in Bioinformatics, with regards to Genomics.

He most often published in these fields:

  • Genetics (41.90%)
  • Cancer research (25.48%)
  • Molecular biology (19.76%)

What were the highlights of his more recent work (between 2014-2021)?

  • Cancer research (25.48%)
  • Cancer (15.95%)
  • Internal medicine (10.48%)

In recent papers he was focusing on the following fields of study:

His primary scientific interests are in Cancer research, Cancer, Internal medicine, SMARCA4 and Oncology. His studies deal with areas such as Exome sequencing, Point mutation, Lung cancer, Small-cell carcinoma and Ovarian cancer as well as Cancer research. Cancer is a subfield of Genetics that Jeffrey M. Trent explores.

His Internal medicine research is multidisciplinary, relying on both Microbiome and Endocrinology. His study in Melanoma is interdisciplinary in nature, drawing from both Mutation, Computational biology and Mutant. Jeffrey M. Trent regularly links together related areas like Molecular biology in his Gene studies.

Between 2014 and 2021, his most popular works were:

  • Genomic Classification of Cutaneous Melanoma (1491 citations)
  • Toward precision medicine in glioblastoma: the promise and the challenges (127 citations)
  • Dual loss of the SWI/SNF complex ATPases SMARCA4/BRG1 and SMARCA2/BRM is highly sensitive and specific for small cell carcinoma of the ovary, hypercalcaemic type (111 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Cancer
  • DNA

Jeffrey M. Trent mostly deals with Cancer research, Oncology, Internal medicine, Cancer and Melanoma. His studies in Cancer research integrate themes in fields like Cell, Histone methyltransferase, SMARCA4 and Mutant, Point mutation. His Internal medicine study integrates concerns from other disciplines, such as Exome sequencing and Gene expression profiling.

His Cancer research includes themes of Clinical trial and Bioinformatics. His Melanoma study introduces a deeper knowledge of Genetics. He mostly deals with Germline in his studies of Genetics.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

WAF1, a potential mediator of p53 tumor suppression

Wafik S. El-Deiry;Takashi Tokino;Victor E. Velculescu;Daniel B. Levy.
Cell (1993)

10892 Citations

Mutations of a mutS homolog in hereditary nonpolyposis colorectal cancer

Fredrick S. Leach;Nicholas C. Nicolaides;Nickolas Papadopoulos;Bo Liu.
Cell (1993)

2874 Citations

Expression profiling using cdna microarrays

David J Duggan;Michael Bittner;Yidong Chen;Paul Meltzer.
Nature Genetics (1999)

2642 Citations

The transcriptional program in the response of human fibroblasts to serum.

Vishwanath R. Iyer;Michael B. Eisen;Douglas T. Ross;Greg Schuler.
Science (1999)

2630 Citations

Vascular Channel Formation by Human Melanoma Cells in Vivo and in Vitro: Vasculogenic Mimicry

Andrew J. Maniotis;Robert Folberg;Angela Hess;Elisabeth A. Seftor.
American Journal of Pathology (1999)

2365 Citations

Gene-Expression Profiles in Hereditary Breast Cancer

I Hedenfalk;D Duggan;Y Chen;M Radmacher.
The New England Journal of Medicine (2001)

2100 Citations

AIB1, a Steroid Receptor Coactivator Amplified in Breast and Ovarian Cancer

Sarah L. Anzick;Juha Kononen;Robert L. Walker;David O. Azorsa.
Science (1997)

2075 Citations

Genomic Classification of Cutaneous Melanoma

Rehan Akbani;Kadir C. Akdemir;B. Arman Aksoy;Monique Albert.
Cell (2015)

2023 Citations

High frequency of BRAF mutations in nevi.

Pamela M. Pollock;Ursula L. Harper;Katherine S. Hansen;Laura M. Yudt.
Nature Genetics (2003)

1921 Citations

Wnt5a signaling directly affects cell motility and invasion of metastatic melanoma.

Ashani T. Weeraratna;Yuan Jiang;Galen Hostetter;Kevin Rosenblatt.
Cancer Cell (2002)

1152 Citations

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National Institutes of Health

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Brown University

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