His main research concerns NMDA receptor, Glutamate receptor, Neuroscience, Pharmacology and Cell biology. Alain Buisson interconnects Glutamatergic, Endocrinology and MAPK/ERK pathway in the investigation of issues within NMDA receptor. His research investigates the link between Glutamate receptor and topics such as Inhibitory postsynaptic potential that cross with problems in Regulation of gene expression, Amyloid precursor protein and Calcium in biology.
In his study, AMPA receptor is strongly linked to Excitotoxicity, which falls under the umbrella field of Neuroscience. In Pharmacology, he works on issues like Biochemistry, which are connected to Brain ischemia and Middle cerebral artery. His Cell biology study combines topics from a wide range of disciplines, such as Excitatory synapse, Excitatory postsynaptic potential, Postsynaptic potential, Postsynaptic density and Long-term potentiation.
Neuroscience, Cell biology, NMDA receptor, Neuroprotection and Synaptic plasticity are his primary areas of study. His Neuroscience research includes elements of AMPA receptor, Amyloid beta, Ischemia and Amyloid precursor protein. His Cell biology research is multidisciplinary, incorporating elements of Endocrinology, Internal medicine, Postsynaptic potential and Neurotransmission.
His studies deal with areas such as Glutamate receptor, Tissue plasminogen activator, Stereochemistry and Pharmacology as well as NMDA receptor. His studies in Neuroprotection integrate themes in fields like Excitotoxicity, Programmed cell death, Plasminogen activator and Serine protease. Alain Buisson has researched Synaptic plasticity in several fields, including Long-term potentiation and Neurodegeneration.
Alain Buisson mainly focuses on Cell biology, Dendritic spine, Synaptic plasticity, Neurotransmission and Long-term potentiation. His Cell biology research is multidisciplinary, relying on both Disease, β amyloid peptide and Amyloidosis. His study focuses on the intersection of Dendritic spine and fields such as Postsynaptic potential with connections in the field of Cofilin 1, Actin cytoskeleton, Excitatory postsynaptic potential and Cofilin.
His work deals with themes such as Endocytosis, Kainate receptor, ESCRT and Signal transducing adaptor protein, which intersect with Synaptic plasticity. The various areas that he examines in his Long-term potentiation study include Microtubule-associated protein, MAP6, Protein filament and Synapse, Neuroscience. Alain Buisson has included themes like LIMK1 and Genetically modified mouse in his Neuroscience study.
His primary scientific interests are in Dendritic spine, Cell biology, Long-term potentiation, Synaptic plasticity and Cofilin 1. His Dendritic spine research is under the purview of Neuroscience. Alain Buisson combines subjects such as Autophagy and Systemic administration with his study of Neuroscience.
His research integrates issues of Cofilin and Postsynaptic density, Postsynaptic potential, Excitatory postsynaptic potential in his study of Cofilin 1. Postsynaptic potential connects with themes related to Rho-associated protein kinase in his study. His Actin research is multidisciplinary, incorporating perspectives in Microtubule-associated protein, MAP6 and Protein filament.
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The proteolytic activity of tissue-plasminogen activator enhances NMDA receptor-mediated signaling.
Olivier Nicole;Fabian Docagne;Carine Ali;Isabelle Margaill.
Nature Medicine (2001)
Ischemia-induced interleukin-6 as a potential endogenous neuroprotective cytokine against NMDA receptor-mediated excitoxicity in the brain
Carine Ali;Olivier Nicole;Fabian Docagne;Sylvain Lesne.
Journal of Cerebral Blood Flow and Metabolism (2000)
The neuroprotective effect of a nitric oxide inhibitor in a rat model of focal cerebral ischaemia.
A. Buisson;M. Plotkine;R.G. Boulu.
British Journal of Pharmacology (1992)
Neuronal viability is controlled by a functional relation between synaptic and extrasynaptic NMDA receptors
F. Léveillé;F. El gaamouch;E. Gouix;M. Lecocq.
The FASEB Journal (2008)
NMDA Receptor Activation Inhibits α-Secretase and Promotes Neuronal Amyloid-β Production
Sylvain Lesné;Carine Ali;Cecília Gabriel;Nicole Croci.
The Journal of Neuroscience (2005)
Neuroprotection Mediated by Glial Cell Line-Derived Neurotrophic Factor: Involvement of a Reduction of NMDA-Induced Calcium Influx by the Mitogen-Activated Protein Kinase Pathway
Olivier Nicole;Carine Ali;Fabian Docagne;Laurent Plawinski.
The Journal of Neuroscience (2001)
Activity-Dependent Tau Protein Translocation to Excitatory Synapse Is Disrupted by Exposure to Amyloid-Beta Oligomers
Marie Lise Frandemiche;Marie Lise Frandemiche;Sandrine De Seranno;Sandrine De Seranno;Travis Rush;Travis Rush;Eve Borel;Eve Borel.
The Journal of Neuroscience (2014)
Activation of extrasynaptic, but not synaptic, NMDA receptors modifies amyloid precursor protein expression pattern and increases amyloid-ß production
Karim Bordji;Javier Becerril-Ortega;Olivier Nicole;Alain Buisson.
The Journal of Neuroscience (2010)
Mechanisms Involved in the Neuroprotective Activity of a Nitric Oxide Synthase Inhibitor During Focal Cerebral Ischemia
A. Buisson;I. Margaill;J. Callebert;M. Plotkine.
Journal of Neurochemistry (2006)
Nitric oxide: an endogenous anticonvulsant substance.
Buisson A;Lakhmeche N;Verrecchia C;Plotkine M.
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