D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Neuroscience D-index 37 Citations 6,383 82 World Ranking 5453 National Ranking 244

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • Neuron

His main research concerns NMDA receptor, Glutamate receptor, Neuroscience, Pharmacology and Cell biology. Alain Buisson interconnects Glutamatergic, Endocrinology and MAPK/ERK pathway in the investigation of issues within NMDA receptor. His research investigates the link between Glutamate receptor and topics such as Inhibitory postsynaptic potential that cross with problems in Regulation of gene expression, Amyloid precursor protein and Calcium in biology.

In his study, AMPA receptor is strongly linked to Excitotoxicity, which falls under the umbrella field of Neuroscience. In Pharmacology, he works on issues like Biochemistry, which are connected to Brain ischemia and Middle cerebral artery. His Cell biology study combines topics from a wide range of disciplines, such as Excitatory synapse, Excitatory postsynaptic potential, Postsynaptic potential, Postsynaptic density and Long-term potentiation.

His most cited work include:

  • The proteolytic activity of tissue-plasminogen activator enhances NMDA receptor-mediated signaling. (613 citations)
  • The neuroprotective effect of a nitric oxide inhibitor in a rat model of focal cerebral ischaemia. (218 citations)
  • Neuronal viability is controlled by a functional relation between synaptic and extrasynaptic NMDA receptors (187 citations)

What are the main themes of his work throughout his whole career to date?

Neuroscience, Cell biology, NMDA receptor, Neuroprotection and Synaptic plasticity are his primary areas of study. His Neuroscience research includes elements of AMPA receptor, Amyloid beta, Ischemia and Amyloid precursor protein. His Cell biology research is multidisciplinary, incorporating elements of Endocrinology, Internal medicine, Postsynaptic potential and Neurotransmission.

His studies deal with areas such as Glutamate receptor, Tissue plasminogen activator, Stereochemistry and Pharmacology as well as NMDA receptor. His studies in Neuroprotection integrate themes in fields like Excitotoxicity, Programmed cell death, Plasminogen activator and Serine protease. Alain Buisson has researched Synaptic plasticity in several fields, including Long-term potentiation and Neurodegeneration.

He most often published in these fields:

  • Neuroscience (51.06%)
  • Cell biology (39.36%)
  • NMDA receptor (39.36%)

What were the highlights of his more recent work (between 2017-2021)?

  • Cell biology (39.36%)
  • Dendritic spine (10.64%)
  • Synaptic plasticity (28.72%)

In recent papers he was focusing on the following fields of study:

Alain Buisson mainly focuses on Cell biology, Dendritic spine, Synaptic plasticity, Neurotransmission and Long-term potentiation. His Cell biology research is multidisciplinary, relying on both Disease, β amyloid peptide and Amyloidosis. His study focuses on the intersection of Dendritic spine and fields such as Postsynaptic potential with connections in the field of Cofilin 1, Actin cytoskeleton, Excitatory postsynaptic potential and Cofilin.

His work deals with themes such as Endocytosis, Kainate receptor, ESCRT and Signal transducing adaptor protein, which intersect with Synaptic plasticity. The various areas that he examines in his Long-term potentiation study include Microtubule-associated protein, MAP6, Protein filament and Synapse, Neuroscience. Alain Buisson has included themes like LIMK1 and Genetically modified mouse in his Neuroscience study.

Between 2017 and 2021, his most popular works were:

  • Autophagy Is Required for Memory Formation and Reverses Age-Related Memory Decline. (54 citations)
  • Synaptotoxicity in Alzheimer's disease involved a dysregulation of actin cytoskeleton dynamics through cofilin 1 phosphorylation (30 citations)
  • Synaptotoxicity in Alzheimer's disease involved a dysregulation of actin cytoskeleton dynamics through cofilin 1 phosphorylation (30 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Neuron

His primary scientific interests are in Dendritic spine, Cell biology, Long-term potentiation, Synaptic plasticity and Cofilin 1. His Dendritic spine research is under the purview of Neuroscience. Alain Buisson combines subjects such as Autophagy and Systemic administration with his study of Neuroscience.

His research integrates issues of Cofilin and Postsynaptic density, Postsynaptic potential, Excitatory postsynaptic potential in his study of Cofilin 1. Postsynaptic potential connects with themes related to Rho-associated protein kinase in his study. His Actin research is multidisciplinary, incorporating perspectives in Microtubule-associated protein, MAP6 and Protein filament.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

The proteolytic activity of tissue-plasminogen activator enhances NMDA receptor-mediated signaling.

Olivier Nicole;Fabian Docagne;Carine Ali;Isabelle Margaill.
Nature Medicine (2001)

825 Citations

Ischemia-induced interleukin-6 as a potential endogenous neuroprotective cytokine against NMDA receptor-mediated excitoxicity in the brain

Carine Ali;Olivier Nicole;Fabian Docagne;Sylvain Lesne.
Journal of Cerebral Blood Flow and Metabolism (2000)

294 Citations

The neuroprotective effect of a nitric oxide inhibitor in a rat model of focal cerebral ischaemia.

A. Buisson;M. Plotkine;R.G. Boulu.
British Journal of Pharmacology (1992)

286 Citations

Neuronal viability is controlled by a functional relation between synaptic and extrasynaptic NMDA receptors

F. Léveillé;F. El gaamouch;E. Gouix;M. Lecocq.
The FASEB Journal (2008)

276 Citations

NMDA Receptor Activation Inhibits α-Secretase and Promotes Neuronal Amyloid-β Production

Sylvain Lesné;Carine Ali;Cecília Gabriel;Nicole Croci.
The Journal of Neuroscience (2005)

250 Citations

Neuroprotection Mediated by Glial Cell Line-Derived Neurotrophic Factor: Involvement of a Reduction of NMDA-Induced Calcium Influx by the Mitogen-Activated Protein Kinase Pathway

Olivier Nicole;Carine Ali;Fabian Docagne;Laurent Plawinski.
The Journal of Neuroscience (2001)

250 Citations

Activity-Dependent Tau Protein Translocation to Excitatory Synapse Is Disrupted by Exposure to Amyloid-Beta Oligomers

Marie Lise Frandemiche;Marie Lise Frandemiche;Sandrine De Seranno;Sandrine De Seranno;Travis Rush;Travis Rush;Eve Borel;Eve Borel.
The Journal of Neuroscience (2014)

234 Citations

Activation of extrasynaptic, but not synaptic, NMDA receptors modifies amyloid precursor protein expression pattern and increases amyloid-ß production

Karim Bordji;Javier Becerril-Ortega;Olivier Nicole;Alain Buisson.
The Journal of Neuroscience (2010)

233 Citations

Mechanisms Involved in the Neuroprotective Activity of a Nitric Oxide Synthase Inhibitor During Focal Cerebral Ischemia

A. Buisson;I. Margaill;J. Callebert;M. Plotkine.
Journal of Neurochemistry (2006)

230 Citations

Nitric oxide: an endogenous anticonvulsant substance.

Buisson A;Lakhmeche N;Verrecchia C;Plotkine M.
Neuroreport (1993)

229 Citations

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