D-Index & Metrics Best Publications

D-Index & Metrics

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 43 Citations 9,171 89 World Ranking 14092 National Ranking 5942

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • Inflammation

Yoshihide Kanaoka mainly focuses on Immunology, Receptor, Prostaglandin D2, Inflammation and Molecular biology. Yoshihide Kanaoka regularly ties together related areas like Mediator in his Immunology studies. His Receptor research integrates issues from Proinflammatory cytokine, Allergic inflammation and Cell biology.

His study looks at the intersection of Proinflammatory cytokine and topics like Mast cell with Chemokine. His Inflammation research is multidisciplinary, incorporating perspectives in Interleukin 10 and Sensitization. His Molecular biology research is multidisciplinary, incorporating elements of Immune system, Zymosan, Prostaglandin-D synthase, Leukotriene C4 and In vivo.

His most cited work include:

  • Cdc25A is a novel phosphatase functioning early in the cell cycle (408 citations)
  • Cysteinyl leukotrienes and their receptors: cellular distribution and function in immune and inflammatory responses. (289 citations)
  • Neutrophil-derived leukotriene B4 is required for inflammatory arthritis (230 citations)

What are the main themes of his work throughout his whole career to date?

Yoshihide Kanaoka mainly investigates Immunology, Receptor, Leukotriene C4, Inflammation and Cell biology. His study ties his expertise on Lung together with the subject of Immunology. His work investigates the relationship between Receptor and topics such as Proinflammatory cytokine that intersect with problems in Vascular permeability.

The concepts of his Leukotriene C4 study are interwoven with issues in Eicosanoid, Biochemistry, Pharmacology and Leukotriene E4. His work focuses on many connections between Inflammation and other disciplines, such as Sensitization, that overlap with his field of interest in Immunoglobulin E. His research integrates issues of Molecular biology and Cell culture in his study of Prostaglandin.

He most often published in these fields:

  • Immunology (45.31%)
  • Receptor (38.28%)
  • Leukotriene C4 (31.25%)

What were the highlights of his more recent work (between 2014-2021)?

  • Immunology (45.31%)
  • Leukotriene C4 (31.25%)
  • Receptor (38.28%)

In recent papers he was focusing on the following fields of study:

Immunology, Leukotriene C4, Receptor, Inflammation and Cell biology are his primary areas of study. In general Immunology study, his work on Mast cell activation, Asthma, Mast cell and Proinflammatory cytokine often relates to the realm of Mite, thereby connecting several areas of interest. His Leukotriene C4 research is multidisciplinary, relying on both Chemokine, Cysteinyl leukotriene receptor 2 and Immunopathology.

The study incorporates disciplines such as Platelet activation, Platelet and Immune system in addition to Receptor. His research in Inflammation intersects with topics in Innate lymphoid cell and Leukotriene E4. His biological study spans a wide range of topics, including Brush Cell, Airway, Cytokine and Lung.

Between 2014 and 2021, his most popular works were:

  • Leukotrienes provide an NFAT-dependent signal that synergizes with IL-33 to activate ILC2s. (81 citations)
  • Aspirin-Exacerbated Respiratory Disease Involves a Cysteinyl Leukotriene-Driven IL-33-Mediated Mast Cell Activation Pathway (76 citations)
  • Leukotriene E4 elicits respiratory epithelial cell mucin release through the G-protein–coupled receptor, GPR99 (68 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Inflammation

His primary areas of investigation include Immunology, Inflammation, Leukotriene C4, Leukotriene E4 and Innate lymphoid cell. Yoshihide Kanaoka is studying Cytokine, which is a component of Immunology. Inflammation is closely attributed to Cell biology in his research.

His Cell biology research includes elements of Tuft cell, Immune system, Transcription factor and Airway. He has included themes like Receptor and Signal transduction in his Leukotriene E4 study. The Receptor study combines topics in areas such as Proinflammatory cytokine, Cellular differentiation and Vascular permeability.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Cdc25A is a novel phosphatase functioning early in the cell cycle

S Jinno;K Suto;A Nagata;M Igarashi.
The EMBO Journal (1994)

629 Citations

Cysteinyl leukotrienes and their receptors: cellular distribution and function in immune and inflammatory responses.

Yoshihide Kanaoka;Joshua A. Boyce.
Journal of Immunology (2004)

443 Citations

Pronounced eosinophilic lung inflammation and Th2 cytokine release in human lipocalin-type prostaglandin D synthase transgenic mice.

Yasushi Fujitani;Yoshihide Kanaoka;Kosuke Aritake;Nobuko Uodome.
Journal of Immunology (2002)

318 Citations

Lack of tactile pain (allodynia) in lipocalin-type prostaglandin D synthase-deficient mice

Naomi Eguchi;Toshiaki Minami;Naoki Shirafuji;Yoshihide Kanaoka.
Proceedings of the National Academy of Sciences of the United States of America (1999)

315 Citations

Cloning and crystal structure of hematopoietic prostaglandin D synthase.

Yoshihide Kanaoka;Hideo Ago;Eiji Inagaki;Toyomichi Nanayama.
Cell (1997)

312 Citations

Dectin-2 Recognition of House Dust Mite Triggers Cysteinyl Leukotriene Generation by Dendritic Cells

Nora A. Barrett;Akiko Maekawa;Opu M. Rahman;K. Frank Austen.
Journal of Immunology (2009)

301 Citations

Neutrophil-derived leukotriene B4 is required for inflammatory arthritis

Mei Chen;Bing K. Lam;Yoshihide Kanaoka;Peter A. Nigrovic.
Journal of Experimental Medicine (2006)

274 Citations

Leukotriene E4–induced pulmonary inflammation is mediated by the P2Y12 receptor

Sailaja Paruchuri;Hiroyuki Tashimo;Hiroyuki Tashimo;Chunli Feng;Akiko Maekawa;Akiko Maekawa.
Journal of Experimental Medicine (2009)

236 Citations

Dectin-2 mediates Th2 immunity through the generation of cysteinyl leukotrienes

Nora A. Barrett;Opu M. Rahman;James M. Fernandez;Matthew W. Parsons.
Journal of Experimental Medicine (2011)

220 Citations

Targeted Gene Disruption Reveals the Role of the Cysteinyl Leukotriene 2 Receptor in Increased Vascular Permeability and in Bleomycin-induced Pulmonary Fibrosis in Mice

Thomas C. Beller;Akiko Maekawa;Daniel S. Friend;Daniel S. Friend;K. Frank Austen.
Journal of Biological Chemistry (2004)

210 Citations

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