Yi Dai mainly investigates Dorsal root ganglion, Neuroscience, Nerve injury, Cell biology and MAPK/ERK pathway. Yi Dai has researched Dorsal root ganglion in several fields, including Anesthesia, Endocrinology, Nerve growth factor, Neurotrophic factors and Internal medicine. His work in the fields of Spinal cord overlaps with other areas such as Population.
His research investigates the connection with Nerve injury and areas like Pharmacology which intersect with concerns in Metabotropic receptor. As a part of the same scientific study, Yi Dai usually deals with the Cell biology, concentrating on Tropomyosin receptor kinase B and frequently concerns with Brain-derived neurotrophic factor. His work carried out in the field of MAPK/ERK pathway brings together such families of science as Sciatic nerve and Protein kinase A.
His main research concerns Dorsal root ganglion, Neuroscience, Internal medicine, Endocrinology and Transient receptor potential channel. His Dorsal root ganglion research is multidisciplinary, relying on both Noxious stimulus, Nerve growth factor, Neurotrophic factors, Sensory neuron and Cell biology. His Cell biology research incorporates themes from Prostaglandin E and Long-term potentiation.
His study in Neuroscience is interdisciplinary in nature, drawing from both Sciatic nerve and Peripheral nerve injury. His Endocrinology study combines topics from a wide range of disciplines, such as Receptor, Anesthesia and Hyperalgesia. His Transient receptor potential channel research integrates issues from AMPK, Nociceptor, Nociception and Pharmacology.
Yi Dai focuses on Eosinophil, Cell biology, Inflammatory bowel disease, Dorsal root ganglion and Transient receptor potential channel. His Cell biology research is multidisciplinary, incorporating perspectives in Prostaglandin E2 and Small intestine. His work often combines Dorsal root ganglion and Allyl isothiocyanate studies.
His Transient receptor potential channel study incorporates themes from Pharmacology and Nociception. Yi Dai has included themes like Endocrinology, Central nervous system, Neuropathic pain, Prostaglandin E2 receptor and Spinal cord in his Nociception study. In his research on the topic of Spinal cord, Nerve injury and Peripheral nervous system is strongly related with Receptor.
Yi Dai mainly focuses on Inflammatory bowel disease, Inflammation, Ulcerative colitis, Abdominal pain and Voxel. His study on Inflammatory bowel disease is covered under Internal medicine. Yi Dai carries out multidisciplinary research, doing studies in Inflammation and Eosinophil.
You can notice a mix of various disciplines of study, such as Disease, Matrix, Pathology and Rat brain, in his Voxel studies.
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Activating Transcription Factor 3 (ATF3) Induction by Axotomy in Sensory and Motoneurons: A Novel Neuronal Marker of Nerve Injury
Hiroaki Tsujino;Eiji Kondo;Tetsuo Fukuoka;Yi Dai.
Molecular and Cellular Neuroscience (2000)
Distinct expression of TRPM8, TRPA1, and TRPV1 mRNAs in rat primary afferent neurons with aδ/c‐fibers and colocalization with trk receptors
Kimiko Kobayashi;Tetsuo Fukuoka;Koichi Obata;Hiroki Yamanaka.
The Journal of Comparative Neurology (2005)
TRPA1 induced in sensory neurons contributes to cold hyperalgesia after inflammation and nerve injury
Koichi Obata;Hirokazu Katsura;Toshiyuki Mizushima;Hiroki Yamanaka.
Journal of Clinical Investigation (2005)
Sensitization of TRPA1 by PAR2 contributes to the sensation of inflammatory pain
Yi Dai;Shenglan Wang;Makoto Tominaga;Satoshi Yamamoto.
Journal of Clinical Investigation (2007)
Phosphorylation of Extracellular Signal-Regulated Kinase in Primary Afferent Neurons by Noxious Stimuli and Its Involvement in Peripheral Sensitization
Yi Dai;Koichi Iwata;Tetsuo Fukuoka;Eiji Kondo.
The Journal of Neuroscience (2002)
Role of Mitogen-Activated Protein Kinase Activation in Injured and Intact Primary Afferent Neurons for Mechanical and Heat Hypersensitivity after Spinal Nerve Ligation
Koichi Obata;Hiroki Yamanaka;Kimiko Kobayashi;Yi Dai.
The Journal of Neuroscience (2004)
Proteinase-activated receptor 2-mediated potentiation of transient receptor potential vanilloid subfamily 1 activity reveals a mechanism for proteinase-induced inflammatory pain.
Yi Dai;Tomoko Moriyama;Tomohiro Higashi;Kazuya Togashi.
The Journal of Neuroscience (2004)
P2Y12 Receptor Upregulation in Activated Microglia Is a Gateway of p38 Signaling and Neuropathic Pain
Kimiko Kobayashi;Hiroki Yamanaka;Tetsuo Fukuoka;Yi Dai.
The Journal of Neuroscience (2008)
Phospholipase C and protein kinase A mediate bradykinin sensitization of TRPA1: a molecular mechanism of inflammatory pain.
Shenglan Wang;Yi Dai;Tetsuo Fukuoka;Hiroki Yamanaka.
Differential Activation of Extracellular Signal-Regulated Protein Kinase in Primary Afferent Neurons Regulates Brain-Derived Neurotrophic Factor Expression after Peripheral Inflammation and Nerve Injury
Koichi Obata;Hiroki Yamanaka;Yi Dai;Toshiya Tachibana.
The Journal of Neuroscience (2003)
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