D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Neuroscience D-index 47 Citations 10,200 85 World Ranking 3678 National Ranking 121

Overview

What is he best known for?

The fields of study he is best known for:

  • Internal medicine
  • Neuron
  • Central nervous system

Yi Dai mainly investigates Dorsal root ganglion, Neuroscience, Nerve injury, Cell biology and MAPK/ERK pathway. Yi Dai has researched Dorsal root ganglion in several fields, including Anesthesia, Endocrinology, Nerve growth factor, Neurotrophic factors and Internal medicine. His work in the fields of Spinal cord overlaps with other areas such as Population.

His research investigates the connection with Nerve injury and areas like Pharmacology which intersect with concerns in Metabotropic receptor. As a part of the same scientific study, Yi Dai usually deals with the Cell biology, concentrating on Tropomyosin receptor kinase B and frequently concerns with Brain-derived neurotrophic factor. His work carried out in the field of MAPK/ERK pathway brings together such families of science as Sciatic nerve and Protein kinase A.

His most cited work include:

  • Distinct expression of TRPM8, TRPA1, and TRPV1 mRNAs in rat primary afferent neurons with aδ/c‐fibers and colocalization with trk receptors (616 citations)
  • Activating Transcription Factor 3 (ATF3) Induction by Axotomy in Sensory and Motoneurons: A Novel Neuronal Marker of Nerve Injury (605 citations)
  • TRPA1 induced in sensory neurons contributes to cold hyperalgesia after inflammation and nerve injury (509 citations)

What are the main themes of his work throughout his whole career to date?

His main research concerns Dorsal root ganglion, Neuroscience, Internal medicine, Endocrinology and Transient receptor potential channel. His Dorsal root ganglion research is multidisciplinary, relying on both Noxious stimulus, Nerve growth factor, Neurotrophic factors, Sensory neuron and Cell biology. His Cell biology research incorporates themes from Prostaglandin E and Long-term potentiation.

His study in Neuroscience is interdisciplinary in nature, drawing from both Sciatic nerve and Peripheral nerve injury. His Endocrinology study combines topics from a wide range of disciplines, such as Receptor, Anesthesia and Hyperalgesia. His Transient receptor potential channel research integrates issues from AMPK, Nociceptor, Nociception and Pharmacology.

He most often published in these fields:

  • Dorsal root ganglion (52.43%)
  • Neuroscience (42.72%)
  • Internal medicine (40.78%)

What were the highlights of his more recent work (between 2018-2021)?

  • Eosinophil (8.74%)
  • Cell biology (30.10%)
  • Inflammatory bowel disease (12.62%)

In recent papers he was focusing on the following fields of study:

Yi Dai focuses on Eosinophil, Cell biology, Inflammatory bowel disease, Dorsal root ganglion and Transient receptor potential channel. His Cell biology research is multidisciplinary, incorporating perspectives in Prostaglandin E2 and Small intestine. His work often combines Dorsal root ganglion and Allyl isothiocyanate studies.

His Transient receptor potential channel study incorporates themes from Pharmacology and Nociception. Yi Dai has included themes like Endocrinology, Central nervous system, Neuropathic pain, Prostaglandin E2 receptor and Spinal cord in his Nociception study. In his research on the topic of Spinal cord, Nerve injury and Peripheral nervous system is strongly related with Receptor.

Between 2018 and 2021, his most popular works were:

  • TRPA1-expressing lamina propria mesenchymal cells regulate colonic motility (8 citations)
  • TRPA1-expressing lamina propria mesenchymal cells regulate colonic motility (8 citations)
  • Pain matrix shift in the rat brain following persistent colonic inflammation revealed by voxel-based statistical analysis: (3 citations)

In his most recent research, the most cited papers focused on:

  • Internal medicine
  • Neuron
  • Pathology

Yi Dai mainly focuses on Inflammatory bowel disease, Inflammation, Ulcerative colitis, Abdominal pain and Voxel. His study on Inflammatory bowel disease is covered under Internal medicine. Yi Dai carries out multidisciplinary research, doing studies in Inflammation and Eosinophil.

You can notice a mix of various disciplines of study, such as Disease, Matrix, Pathology and Rat brain, in his Voxel studies.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Activating Transcription Factor 3 (ATF3) Induction by Axotomy in Sensory and Motoneurons: A Novel Neuronal Marker of Nerve Injury

Hiroaki Tsujino;Eiji Kondo;Tetsuo Fukuoka;Yi Dai.
Molecular and Cellular Neuroscience (2000)

1013 Citations

Distinct expression of TRPM8, TRPA1, and TRPV1 mRNAs in rat primary afferent neurons with aδ/c‐fibers and colocalization with trk receptors

Kimiko Kobayashi;Tetsuo Fukuoka;Koichi Obata;Hiroki Yamanaka.
The Journal of Comparative Neurology (2005)

1010 Citations

TRPA1 induced in sensory neurons contributes to cold hyperalgesia after inflammation and nerve injury

Koichi Obata;Hirokazu Katsura;Toshiyuki Mizushima;Hiroki Yamanaka.
Journal of Clinical Investigation (2005)

816 Citations

Sensitization of TRPA1 by PAR2 contributes to the sensation of inflammatory pain

Yi Dai;Shenglan Wang;Makoto Tominaga;Satoshi Yamamoto.
Journal of Clinical Investigation (2007)

559 Citations

Phosphorylation of Extracellular Signal-Regulated Kinase in Primary Afferent Neurons by Noxious Stimuli and Its Involvement in Peripheral Sensitization

Yi Dai;Koichi Iwata;Tetsuo Fukuoka;Eiji Kondo.
The Journal of Neuroscience (2002)

472 Citations

Role of Mitogen-Activated Protein Kinase Activation in Injured and Intact Primary Afferent Neurons for Mechanical and Heat Hypersensitivity after Spinal Nerve Ligation

Koichi Obata;Hiroki Yamanaka;Kimiko Kobayashi;Yi Dai.
The Journal of Neuroscience (2004)

423 Citations

Proteinase-activated receptor 2-mediated potentiation of transient receptor potential vanilloid subfamily 1 activity reveals a mechanism for proteinase-induced inflammatory pain.

Yi Dai;Tomoko Moriyama;Tomohiro Higashi;Kazuya Togashi.
The Journal of Neuroscience (2004)

417 Citations

P2Y12 Receptor Upregulation in Activated Microglia Is a Gateway of p38 Signaling and Neuropathic Pain

Kimiko Kobayashi;Hiroki Yamanaka;Tetsuo Fukuoka;Yi Dai.
The Journal of Neuroscience (2008)

346 Citations

Phospholipase C and protein kinase A mediate bradykinin sensitization of TRPA1: a molecular mechanism of inflammatory pain.

Shenglan Wang;Yi Dai;Tetsuo Fukuoka;Hiroki Yamanaka.
Brain (2008)

338 Citations

Differential Activation of Extracellular Signal-Regulated Protein Kinase in Primary Afferent Neurons Regulates Brain-Derived Neurotrophic Factor Expression after Peripheral Inflammation and Nerve Injury

Koichi Obata;Hiroki Yamanaka;Yi Dai;Toshiya Tachibana.
The Journal of Neuroscience (2003)

308 Citations

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