Koichi Obata mostly deals with Dorsal root ganglion, Neuroscience, Chemistry, MAPK/ERK pathway and Neurotrophic factors. His Dorsal root ganglion research is multidisciplinary, relying on both Internal medicine, Transient receptor potential channel, Endocrinology and Nerve growth factor. His Nerve growth factor study which covers TRPM8 that intersects with Low-affinity nerve growth factor receptor and Trk receptor.
Koichi Obata studies Neuroscience, namely Neuropathic pain. His work is dedicated to discovering how MAPK/ERK pathway, Axotomy are connected with Intracellular signal transduction and other disciplines. As part of one scientific family, he deals mainly with the area of Neurotrophic factors, narrowing it down to issues related to the Nerve injury, and often Pharmacology and p38 mitogen-activated protein kinases.
His primary scientific interests are in Neuroscience, Dorsal root ganglion, Nerve injury, Endocrinology and Internal medicine. His Neuroscience research integrates issues from Sciatic nerve, Peripheral nerve injury and MAPK/ERK pathway. His work deals with themes such as Anesthesia, Intracellular signal transduction, Protein kinase A and Arthritis, which intersect with MAPK/ERK pathway.
Koichi Obata has researched Dorsal root ganglion in several fields, including Nerve growth factor, Neurotrophic factors, Transient receptor potential channel and Cell biology. His research integrates issues of Tactile Allodynia, Neuroglia, Rhizotomy and Microglia in his study of Nerve injury. His work carried out in the field of Endocrinology brings together such families of science as Neuron and Visceral pain.
Koichi Obata mainly investigates Nerve injury, Neuroscience, MAPK/ERK pathway, Dorsal root ganglion and Visceral pain. The study incorporates disciplines such as Tactile Allodynia, Neuropathic pain, Peripheral nerve injury and Microglia in addition to Nerve injury. In his research, he undertakes multidisciplinary study on Neuroscience and Interleukin 1β converting enzyme.
He studied MAPK/ERK pathway and Sensory system that intersect with Sensory loss. His Dorsal root ganglion study frequently intersects with other fields, such as Electrophysiology. When carried out as part of a general Internal medicine research project, his work on Nociceptor is frequently linked to work in Chemistry, therefore connecting diverse disciplines of study.
Koichi Obata mainly focuses on Neuroscience, Neuropathic pain, Nerve injury, Microglia and Cell biology. The concepts of his Neuroscience study are interwoven with issues in Glial cell line-derived neurotrophic factor and Neurotrophic factors. His Neuropathic pain research includes elements of Signal transduction, Astrocyte and Spinal cord.
The various areas that Koichi Obata examines in his Nerve injury study include Metabotropic receptor, Sciatic nerve, Pharmacology and MAPK/ERK pathway, p38 mitogen-activated protein kinases. By researching both Microglia and P2RX7, Koichi Obata produces research that crosses academic boundaries. His Cell biology research is multidisciplinary, incorporating elements of Tropomyosin receptor kinase A, Nerve growth factor, Electrophysiology and Dorsal root ganglion.
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Distinct expression of TRPM8, TRPA1, and TRPV1 mRNAs in rat primary afferent neurons with aδ/c‐fibers and colocalization with trk receptors
Kimiko Kobayashi;Tetsuo Fukuoka;Koichi Obata;Hiroki Yamanaka.
The Journal of Comparative Neurology (2005)
TRPA1 induced in sensory neurons contributes to cold hyperalgesia after inflammation and nerve injury
Koichi Obata;Hirokazu Katsura;Toshiyuki Mizushima;Hiroki Yamanaka.
Journal of Clinical Investigation (2005)
Sensitization of TRPA1 by PAR2 contributes to the sensation of inflammatory pain
Yi Dai;Shenglan Wang;Makoto Tominaga;Satoshi Yamamoto.
Journal of Clinical Investigation (2007)
MAPK activation in nociceptive neurons and pain hypersensitivity
Koichi Obata;Koichi Noguchi.
Life Sciences (2004)
Role of Mitogen-Activated Protein Kinase Activation in Injured and Intact Primary Afferent Neurons for Mechanical and Heat Hypersensitivity after Spinal Nerve Ligation
Koichi Obata;Hiroki Yamanaka;Kimiko Kobayashi;Yi Dai.
The Journal of Neuroscience (2004)
P2Y12 Receptor Upregulation in Activated Microglia Is a Gateway of p38 Signaling and Neuropathic Pain
Kimiko Kobayashi;Hiroki Yamanaka;Tetsuo Fukuoka;Yi Dai.
The Journal of Neuroscience (2008)
Phospholipase C and protein kinase A mediate bradykinin sensitization of TRPA1: a molecular mechanism of inflammatory pain.
Shenglan Wang;Yi Dai;Tetsuo Fukuoka;Hiroki Yamanaka.
Contribution of injured and uninjured dorsal root ganglion neurons to pain behavior and the changes in gene expression following chronic constriction injury of the sciatic nerve in rats.
Koichi Obata;Hiroki Yamanaka;Tetsuo Fukuoka;Dai Yi.
Interleukin-18-Mediated Microglia/Astrocyte Interaction in the Spinal Cord Enhances Neuropathic Pain Processing after Nerve Injury
Kan Miyoshi;Koichi Obata;Takashi Kondo;Haruki Okamura.
The Journal of Neuroscience (2008)
Differential Activation of Extracellular Signal-Regulated Protein Kinase in Primary Afferent Neurons Regulates Brain-Derived Neurotrophic Factor Expression after Peripheral Inflammation and Nerve Injury
Koichi Obata;Hiroki Yamanaka;Yi Dai;Toshiya Tachibana.
The Journal of Neuroscience (2003)
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