What is he best known for?
The fields of study he is best known for:
- Neuron
- Internal medicine
- Central nervous system
Koichi Obata mostly deals with Dorsal root ganglion, Neuroscience, Chemistry, MAPK/ERK pathway and Neurotrophic factors.
His Dorsal root ganglion research is multidisciplinary, relying on both Internal medicine, Transient receptor potential channel, Endocrinology and Nerve growth factor.
His Nerve growth factor study which covers TRPM8 that intersects with Low-affinity nerve growth factor receptor and Trk receptor.
Koichi Obata studies Neuroscience, namely Neuropathic pain.
His work is dedicated to discovering how MAPK/ERK pathway, Axotomy are connected with Intracellular signal transduction and other disciplines.
As part of one scientific family, he deals mainly with the area of Neurotrophic factors, narrowing it down to issues related to the Nerve injury, and often Pharmacology and p38 mitogen-activated protein kinases.
His most cited work include:
- Distinct expression of TRPM8, TRPA1, and TRPV1 mRNAs in rat primary afferent neurons with aδ/c‐fibers and colocalization with trk receptors (616 citations)
- TRPA1 induced in sensory neurons contributes to cold hyperalgesia after inflammation and nerve injury (509 citations)
- Sensitization of TRPA1 by PAR2 contributes to the sensation of inflammatory pain (343 citations)
What are the main themes of his work throughout his whole career to date?
His primary scientific interests are in Neuroscience, Dorsal root ganglion, Nerve injury, Endocrinology and Internal medicine.
His Neuroscience research integrates issues from Sciatic nerve, Peripheral nerve injury and MAPK/ERK pathway.
His work deals with themes such as Anesthesia, Intracellular signal transduction, Protein kinase A and Arthritis, which intersect with MAPK/ERK pathway.
Koichi Obata has researched Dorsal root ganglion in several fields, including Nerve growth factor, Neurotrophic factors, Transient receptor potential channel and Cell biology.
His research integrates issues of Tactile Allodynia, Neuroglia, Rhizotomy and Microglia in his study of Nerve injury.
His work carried out in the field of Endocrinology brings together such families of science as Neuron and Visceral pain.
He most often published in these fields:
- Neuroscience (65.57%)
- Dorsal root ganglion (63.93%)
- Nerve injury (37.70%)
What were the highlights of his more recent work (between 2007-2010)?
- Nerve injury (37.70%)
- Neuroscience (65.57%)
- MAPK/ERK pathway (36.07%)
In recent papers he was focusing on the following fields of study:
Koichi Obata mainly investigates Nerve injury, Neuroscience, MAPK/ERK pathway, Dorsal root ganglion and Visceral pain.
The study incorporates disciplines such as Tactile Allodynia, Neuropathic pain, Peripheral nerve injury and Microglia in addition to Nerve injury.
In his research, he undertakes multidisciplinary study on Neuroscience and Interleukin 1β converting enzyme.
He studied MAPK/ERK pathway and Sensory system that intersect with Sensory loss.
His Dorsal root ganglion study frequently intersects with other fields, such as Electrophysiology.
When carried out as part of a general Internal medicine research project, his work on Nociceptor is frequently linked to work in Chemistry, therefore connecting diverse disciplines of study.
Between 2007 and 2010, his most popular works were:
- Phospholipase C and protein kinase A mediate bradykinin sensitization of TRPA1: a molecular mechanism of inflammatory pain. (209 citations)
- P2Y12 Receptor Upregulation in Activated Microglia Is a Gateway of p38 Signaling and Neuropathic Pain (202 citations)
- Interleukin-18-Mediated Microglia/Astrocyte Interaction in the Spinal Cord Enhances Neuropathic Pain Processing after Nerve Injury (187 citations)
In his most recent research, the most cited papers focused on:
- Neuron
- Signal transduction
- Internal medicine
Koichi Obata mainly focuses on Neuroscience, Neuropathic pain, Nerve injury, Microglia and Cell biology.
The concepts of his Neuroscience study are interwoven with issues in Glial cell line-derived neurotrophic factor and Neurotrophic factors.
His Neuropathic pain research includes elements of Signal transduction, Astrocyte and Spinal cord.
The various areas that Koichi Obata examines in his Nerve injury study include Metabotropic receptor, Sciatic nerve, Pharmacology and MAPK/ERK pathway, p38 mitogen-activated protein kinases.
By researching both Microglia and P2RX7, Koichi Obata produces research that crosses academic boundaries.
His Cell biology research is multidisciplinary, incorporating elements of Tropomyosin receptor kinase A, Nerve growth factor, Electrophysiology and Dorsal root ganglion.
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