World's Best Scientists 2026 revealed!

D-Index & Metrics

Biology and Biochemistry

D-Index
75
Citations
18356
World Ranking
5368
National Ranking
44

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Cancer
  • Apoptosis

His scientific interests lie mostly in Cell biology, Molecular biology, Apoptosis, Programmed cell death and Cancer research. Cell biology is often connected to Cell adhesion in his work. His Molecular biology study combines topics in areas such as Transcription factor, Immunology, Transfection, Complementary DNA and Phosphorylation.

Su Jae Lee has included themes like Oxidative stress, Mitochondrion and Intracellular in his Apoptosis study. His work carried out in the field of Programmed cell death brings together such families of science as Autophagy and Reactive oxygen species. His work deals with themes such as Cell culture, Endocrinology, Cancer cell, Internal medicine and Cyclin, which intersect with Cancer research.

His most cited work include:

  • Microplastic Size-Dependent Toxicity, Oxidative Stress Induction, and p-JNK and p-p38 Activation in the Monogonont Rotifer (Brachionus koreanus) (354 citations)
  • An orally administered multitarget tyrosine kinase inhibitor, SU11248, is a novel potent inhibitor of thyroid oncogenic RET/papillary thyroid cancer kinases. (286 citations)
  • Induction of cell cycle arrest and apoptosis in human breast cancer cells by quercetin. (268 citations)

What are the main themes of his work throughout his whole career to date?

Su Jae Lee mostly deals with Cancer research, Cell biology, Apoptosis, Molecular biology and Programmed cell death. His research in Cancer research intersects with topics in Cancer cell, Cancer stem cell, Downregulation and upregulation, Immunology and Breast cancer. His Cell biology research is multidisciplinary, incorporating perspectives in Cell cycle and Cell culture.

His research in Molecular biology focuses on subjects like Transfection, which are connected to Radioresistance. His Programmed cell death research includes themes of Autophagy, Mitochondrion and Intracellular. Su Jae Lee studied Reactive oxygen species and Oxidative stress that intersect with Glutathione.

He most often published in these fields:

  • Cancer research (40.37%)
  • Cell biology (31.11%)
  • Apoptosis (18.89%)

What were the highlights of his more recent work (between 2015-2021)?

  • Cancer research (40.37%)
  • Cancer (9.63%)
  • Cancer cell (12.59%)

In recent papers he was focusing on the following fields of study:

Cancer research, Cancer, Cancer cell, Internal medicine and Mesenchymal stem cell are his primary areas of study. The concepts of his Cancer research study are interwoven with issues in Carcinogenesis, Tumor microenvironment, Metastasis, Breast cancer and In vivo. His Cancer study also includes fields such as

  • CD44, which have a strong connection to Radiation therapy, SOX2, Radioresistance and Downregulation and upregulation,
  • In vitro that connect with fields like Programmed cell death, Inner mitochondrial membrane and Apoptosis.

His study in Cancer cell is interdisciplinary in nature, drawing from both Label free, Cancer stem cell, Disease and Constriction. His biological study spans a wide range of topics, including Glioblastoma and Oncology. His Signal transduction study is concerned with Cell biology in general.

Between 2015 and 2021, his most popular works were:

  • Microplastic Size-Dependent Toxicity, Oxidative Stress Induction, and p-JNK and p-p38 Activation in the Monogonont Rotifer (Brachionus koreanus) (354 citations)
  • Adverse effects of microplastics and oxidative stress-induced MAPK/Nrf2 pathway-mediated defense mechanisms in the marine copepod Paracyclopina nana. (171 citations)
  • Increased Serotonin Signaling Contributes to the Warburg Effect in Pancreatic Tumor Cells Under Metabolic Stress and Promotes Growth of Pancreatic Tumors in Mice (100 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Cancer
  • Enzyme

Su Jae Lee focuses on Cancer research, Internal medicine, Cancer stem cell, Oxidative stress and Oncology. He combines subjects such as Carcinogenesis, Mesenchymal stem cell, Pathology, Breast cancer and PI3K/AKT/mTOR pathway with his study of Cancer research. Su Jae Lee has researched Cancer stem cell in several fields, including Epithelial–mesenchymal transition, Targeted therapy, Homeobox protein NANOG and Stem cell marker.

His Oxidative stress research includes elements of Reactive oxygen species, Glutathione, Toxicity and Rotifer. In his work, Signal transduction is strongly intertwined with Epidermal growth factor, which is a subfield of Molecular biology. His Signal transduction study deals with the bigger picture of Cell biology.

Best Publications

  • Microplastic Size-Dependent Toxicity, Oxidative Stress Induction, and p-JNK and p-p38 Activation in the Monogonont Rotifer (Brachionus koreanus)

    Chang Bum Jeong;Chang Bum Jeong;Eun Ji Won;Eun Ji Won;Hye Min Kang;Min Chul Lee

  • Adverse effects of microplastics and oxidative stress-induced MAPK/Nrf2 pathway-mediated defense mechanisms in the marine copepod Paracyclopina nana.

    Chang Bum Jeong;Hye Min Kang;Min Chul Lee;Duck Hyun Kim

  • Induction of cell cycle arrest and apoptosis in human breast cancer cells by quercetin.

    Jung-A Choi;Ja-Young Kim;Jeong-Yim Lee;Chang-Mo Kang

  • An orally administered multitarget tyrosine kinase inhibitor, SU11248, is a novel potent inhibitor of thyroid oncogenic RET/papillary thyroid cancer kinases.

    Dong Wook Kim;Young Suk Jo;Hye Sook Jung;Hyo Kyun Chung

  • Update on Hsp90 inhibitors in clinical trial.

    Y. S. Kim;S. V. Alarcon;S. Lee;M.-J. Lee

  • Electrophoresis deposition of carbon nanotubes for triode-type field emission display

    W. B. Choi;Y. W. Jin;H. Y. Kim;S. J. Lee

  • Sodium selenite induces superoxide-mediated mitochondrial damage and subsequent autophagic cell death in malignant glioma cells.

    Eun Hee Kim;Seonghyang Sohn;Hyuk Jae Kwon;Seung U. Kim

  • Cell Cycle and Cancer

    Moon-Taek Park;Su-Jae Lee

  • Biological and medical applications of plasma-activated media, water and solutions

    Nagendra Kumar Kaushik;Bhagirath Ghimire;Ying Li;Manish Adhikari

  • Caspase-independent cell death by arsenic trioxide in human cervical cancer cells: reactive oxygen species-mediated poly(ADP-ribose) polymerase-1 activation signals apoptosis-inducing factor release from mitochondria.

    Young Hee Kang;Min Jung Yi;Min Jung Kim;Moon Taek Park

  • Opposing Role of Mitogen-activated Protein Kinase Subtypes, Erk-1/2 and p38, in the Regulation of Chondrogenesis of Mesenchymes

    Chun Do Oh;Sung Hee Chang;Young Mee Yoon;Su Jae Lee

  • Involvement of Autophagy in Oncogenic K-Ras-induced Malignant Cell Transformation

    Min Jung Kim;Soo Jung Woo;Chang-Hwan Yoon;Jae Seong Lee

  • A recombinant adenovirus expressing p27Kip1 induces cell cycle arrest and loss of cyclin-Cdk activity in human breast cancer cells.

    Caroline Craig;Robert Wersto;Min Kim;Ekta Ohri

  • Regulation of Cyclin D1 by Calpain Protease

    Yung Hyun Choi;Su Jae Lee;PhuongMai Nguyen;Joung Soon Jang

  • Increased Serotonin Signaling Contributes to the Warburg Effect in Pancreatic Tumor Cells Under Metabolic Stress and Promotes Growth of Pancreatic Tumors in Mice

    Shu Heng Jiang;Jun Li;Fang Yuan Dong;Jian Yu Yang

  • Claudin-1 induces epithelial-mesenchymal transition through activation of the c-Abl-ERK signaling pathway in human liver cells.

    Y. Suh;C. H. Yoon;R. K. Kim;E. J. Lim

  • Upregulation of CXCR4 is functionally crucial for maintenance of stemness in drug-resistant non-small cell lung cancer cells.

    Jung Mj;Rho Jk;Kim Ym;Jung Je

  • Dual roles of human BubR1, a mitotic checkpoint kinase, in the monitoring of chromosomal instability

    Hyun Jin Shin;Kwan Hyuck Baek;Ae Hwa Jeon;Moon Taek Park

  • Up-regulation of Nrf2-mediated heme oxygenase-1 expression by eckol, a phlorotannin compound, through activation of Erk and PI3K/Akt.

    Ki Cheon Kim;Kyoung Ah Kang;Rui Zhang;Mei Jing Piao

  • Inhibition of the 3-hydroxy-3-methylglutaryl-coenzyme A reductase pathway induces p53-independent transcriptional regulation of p21(WAF1/CIP1) in human prostate carcinoma cells

    Su Jae Lee;Mahn Joon Ha;Jeen Lee;PhuongMai Nguyen

Frequent Co-Authors

Yun-Sil Lee
Yun-Sil Lee Ewha Womans University
Jae-Seong Lee
Jae-Seong Lee Sungkyunkwan University
Jane B. Trepel
Jane B. Trepel National Institutes of Health
Jin Won Hyun
Jin Won Hyun Jeju National University
Yung Hyun Choi
Yung Hyun Choi Dong-Eui University
Gi-Young Kim
Gi-Young Kim Jeju National University
Young Tae Kim
Young Tae Kim Seoul National University Hospital
Pierre A. Henkart
Pierre A. Henkart National Institutes of Health
Wun-Jae Kim
Wun-Jae Kim Chungbuk National University
Frederick F. Lang
Frederick F. Lang The University of Texas MD Anderson Cancer Center

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